Works matching IS 25233548 AND DT 2021 AND VI 41 AND IP 9
Results: 10
Front Cover: Nuclear Aurora kinase A triggers programmed death‐ligand 1‐mediated immune suppression by activating MYC transcription in triple‐negative breast cancer.
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- Cancer Communications, 2021, v. 41, n. 9, p. i, doi. 10.1002/cac2.12219
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CD70‐targeting CAR‐T cells have potential activity against CD19‐negative B‐cell Lymphoma.
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- Cancer Communications, 2021, v. 41, n. 9, p. 925, doi. 10.1002/cac2.12201
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Outcomes and toxicities of immune checkpoint inhibitors in colorectal cancer: a real‐world retrospective analysis.
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- Cancer Communications, 2021, v. 41, n. 9, p. 921, doi. 10.1002/cac2.12199
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Advancing to the era of cancer immunotherapy.
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- Cancer Communications, 2021, v. 41, n. 9, p. 803, doi. 10.1002/cac2.12178
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- Article
Issue Information.
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- Cancer Communications, 2021, v. 41, n. 9, p. 801, doi. 10.1002/cac2.12060
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- Article
Improving antitumor immunity using antiangiogenic agents: Mechanistic insights, current progress, and clinical challenges.
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- Cancer Communications, 2021, v. 41, n. 9, p. 830, doi. 10.1002/cac2.12183
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- Article
KDELR2 promotes breast cancer proliferation via HDAC3‐mediated cell cycle progression.
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- Cancer Communications, 2021, v. 41, n. 9, p. 904, doi. 10.1002/cac2.12180
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- Article
Bevacizumab biosimilar LY01008 compared with bevacizumab (Avastin) as first‐line treatment for Chinese patients with unresectable, metastatic, or recurrent non‐squamous non–small‐cell lung cancer: A multicenter, randomized, double‐blinded, phase III trial
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- Cancer Communications, 2021, v. 41, n. 9, p. 889, doi. 10.1002/cac2.12179
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- Article
Nuclear Aurora kinase A triggers programmed death‐ligand 1‐mediated immune suppression by activating MYC transcription in triple‐negative breast cancer.
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- Cancer Communications, 2021, v. 41, n. 9, p. 851, doi. 10.1002/cac2.12190
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- Article
Intra‐heterogeneity in transcription and chemoresistant property of leukemia‐initiating cells in murine Setd2<sup>−/−</sup> acute myeloid leukemia.
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- Cancer Communications, 2021, v. 41, n. 9, p. 867, doi. 10.1002/cac2.12189
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- Article