Works matching IS 21686149 AND DT 2016 AND VI 73 AND IP 10
Results: 25
Status Epilepticus and Brain Atrophy: Shrinkage Is a Growing Problem.
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- JAMA Neurology, 2016, v. 73, n. 10, p. 1182, doi. 10.1001/jamaneurol.2016.2639
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Spectrum of Movement Disorders in Mitochondrial Disorders: Reply.
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- JAMA Neurology, 2016, v. 73, n. 10, p. 1254, doi. 10.1001/jamaneurol.2016.2578
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Spectrum of Movement Disorders in Mitochondrial Disorders.
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- JAMA Neurology, 2016, v. 73, n. 10, p. 1253, doi. 10.1001/jamaneurol.2016.2581
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CORRECTION.
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- JAMA Neurology, 2016, v. 73, n. 10, p. 1260
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- Article
Comparing Painful Stimulation vs Rest in Studies of Pain.
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- JAMA Neurology, 2016, v. 73, n. 10, p. 1258, doi. 10.1001/jamaneurol.2016.2989
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Factors Associated With Neurological Outcome After Childhood Stroke.
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- JAMA Neurology, 2016, v. 73, n. 10, p. 1257, doi. 10.1001/jamaneurol.2016.2951
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The Dark Matter of Cerebral Microbleeds: Implications for Thrombolysis and Other Clinical Scenarios.
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- JAMA Neurology, 2016, v. 73, n. 10, p. 1255, doi. 10.1001/jamaneurol.2016.2948
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Association of Alzheimer Disease Susceptibility Variants and Gene Expression in the Human Brain.
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- JAMA Neurology, 2016, v. 73, n. 10, p. 1255, doi. 10.1001/jamaneurol.2016.2796
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Bortezomib Treatment for Patients With Anti-N-Methyl-D-Aspartate Receptor Encephalitis.
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- JAMA Neurology, 2016, v. 73, n. 10, p. 1251, doi. 10.1001/jamaneurol.2016.2588
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Characteristic Neuroimaging Abnormalities of Korsakoff Syndrome.
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- JAMA Neurology, 2016, v. 73, n. 10, p. 1248, doi. 10.1001/jamaneurol.2016.1843
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Bilateral Infarction of the Corpus Callosum in a Patient With a Single Pericallosal Artery.
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- JAMA Neurology, 2016, v. 73, n. 10, p. 1246, doi. 10.1001/jamaneurol.2016.1230
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New Reasons to Pursue the Therapeutic Potential of Synthetic Nucleic Acids for Neurological Diseases.
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- JAMA Neurology, 2016, v. 73, n. 10, p. 1175, doi. 10.1001/jamaneurol.2016.2571
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A Young Man With Recurrent Coma and Refractory Status Epilepticus.
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- JAMA Neurology, 2016, v. 73, n. 10, p. 1243, doi. 10.1001/jamaneurol.2016.1788
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The Role of Cardiovascular Risk Factors and Stroke in Familial Alzheimer Disease.
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- JAMA Neurology, 2016, v. 73, n. 10, p. 1231, doi. 10.1001/jamaneurol.2016.2539
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Safety and Outcome of Intra-Arterial Treatment for Basilar Artery Occlusion.
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- JAMA Neurology, 2016, v. 73, n. 10, p. 1225, doi. 10.1001/jamaneurol.2016.1408
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Association of GBA Mutations and the E326K Polymorphism With Motor and Cognitive Progression in Parkinson Disease.
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- JAMA Neurology, 2016, v. 73, n. 10, p. 1217, doi. 10.1001/jamaneurol.2016.2245
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Association of Serum Docosahexaenoic Acid With Cerebral Amyloidosis.
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- JAMA Neurology, 2016, v. 73, n. 10, p. 1208, doi. 10.1001/jamaneurol.2016.1924
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Synthetic Nucleic Acids and Treatment of Neurological Diseases.
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- JAMA Neurology, 2016, v. 73, n. 10, p. 1238, doi. 10.1001/jamaneurol.2016.2089
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Progressive Brain Atrophy in Super-refractory Status Epilepticus.
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- JAMA Neurology, 2016, v. 73, n. 10, p. 1201, doi. 10.1001/jamaneurol.2016.1572
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Heterogeneity in Suspected Non-Alzheimer Disease Pathophysiology Among Clinically Normal Older Individuals.
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- JAMA Neurology, 2016, v. 73, n. 10, p. 1185, doi. 10.1001/jamaneurol.2016.2237
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Do ω-3 Fatty Acids Regulate Cerebral β Amyloid?
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- JAMA Neurology, 2016, v. 73, n. 10, p. 1183, doi. 10.1001/jamaneurol.2016.2534
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Brain Atrophy on Magnetic Resonance Imaging as a Biomarker of Neurodegeneration.
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- JAMA Neurology, 2016, v. 73, n. 10, p. 1179, doi. 10.1001/jamaneurol.2016.2843
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Cause of Suspected Non-Alzheimer Disease Pathophysiology.
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- JAMA Neurology, 2016, v. 73, n. 10, p. 1177, doi. 10.1001/jamaneurol.2016.2842
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Longitudinal α-Amyloid Deposition and Hippocampal Volume in Preclinical Alzheimer Disease and Suspected Non-Alzheimer Disease Pathophysiology.
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- JAMA Neurology, 2016, v. 73, n. 10, p. 1192, doi. 10.1001/jamaneurol.2016.2642
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The Roadmap to Value-Based Care.
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- JAMA Neurology, 2016, v. 73, n. 10, p. 1173, doi. 10.1001/jamaneurol.2016.1299
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