Works matching IS 19450877 AND DT 2014 AND VI 7 AND IP 351

Results: 3

Type I interferon signaling contributes to the bias that Toll-like receptor 4 exhibits for signaling mediated by the adaptor protein TRIF.
Published in:
Science Signaling, 2014, v. 7, n. 351, p. 1, doi. 10.1126/scisignal.2005442
By:
- Kolb, Joseph P.;
- Casella, Carolyn R.;
- SenGupta, Shuvasree;
- Chilton, Paula M.;
- Mitchell, Thomas C.
Publication type:
Article
Resistance to dual blockade of the kinases PI3K and mTOR in KRAS-mutant colorectal cancer models results in combined sensitivity to inhibition of the receptor tyrosine kinase EGFR.
Published in:
Science Signaling, 2014, v. 7, n. 351, p. 1, doi. 10.1126/scisignal.2005516
By:
- Belmont, Peter J.;
- Ping Jiang;
- McKee, Trevor D.;
- Tao Xie;
- Isaacson, Jason;
- Baryla, Nicole E.;
- Roper, Jatin;
- Sinnamon, Mark J.;
- Lee, Nathan V.;
- Kan, Julie L. C.;
- Guicherit, Oivin;
- Wouters, Bradly G.;
- O'Brien, Catherine A.;
- Shields, David;
- Olson, Peter;
- VanArsdale, Todd;
- Weinrich, Scott L.;
- Rejto, Paul;
- Christensen, James G.;
- Fantin, Valeria R.
Publication type:
Article
Inflammatory stimuli induce inhibitory S-nitrosylation of the deacetylase SIRT1 to increase acetylation and activation of p53 and p65.
Published in:
Science Signaling, 2014, v. 7, n. 351, p. 1, doi. 10.1126/scisignal.2005375
By:
- Shohei Shinozaki;
- Kyungho Chang;
- Michihiro Sakai;
- Nobuyuki Shimizu;
- Marina Yamada;
- Tomokazu Tanaka;
- Harumasa Nakazawa;
- Fumito Ichinose;
- Yoshitsugu Yamada;
- Akihito Ishigami;
- Hideki Ito;
- Yasuyoshi Ouchi;
- Starr, Marlene E.;
- Hiroshi Saito;
- Kentaro Shimokado;
- Jonathan S. Stamler;
- Masao Kaneki
Publication type:
Article

Works matching IS 19450877 AND DT 2014 AND VI 7 AND IP 351

Type I interferon signaling contributes to the bias that Toll-like receptor 4 exhibits for signaling mediated by the adaptor protein TRIF.

Resistance to dual blockade of the kinases PI3K and mTOR in KRAS-mutant colorectal cancer models results in combined sensitivity to inhibition of the receptor tyrosine kinase EGFR.

Inflammatory stimuli induce inhibitory S-nitrosylation of the deacetylase SIRT1 to increase acetylation and activation of p53 and p65.