Eosinophilic inflammation of chronic rhinosinusitis with nasal polyps is related to OX40 ligand expression.
- Published in:
- Innate Immunity, 2015, v. 21, n. 2, p. 167, doi. 10.1177/1753425914523460
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- Publication type:
- Article
Works matching IS 17534259 AND DT 2015 AND VI 21 AND IP 2
Results: 10
1
2
Involvement of redox balance in in vitro osteoclast formation of RAW 264.7 macrophage cells in response to LPS.
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- Innate Immunity, 2015, v. 21, n. 2, p. 194, doi. 10.1177/1753425914524242
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- Publication type:
- Article
3
Therapeutic potential of adenovirus-mediated delivery of β-defensin 2 for experimental otitis media.
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- Innate Immunity, 2015, v. 21, n. 2, p. 215, doi. 10.1177/1753425914534002
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- Publication type:
- Article
4
The inflammasome adaptor ASC contributes to multiple innate immune processes in the resolution of otitis media.
- Published in:
- Innate Immunity, 2015, v. 21, n. 2, p. 203, doi. 10.1177/1753425914526074
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- Publication type:
- Article
5
Soluble human TLR2 ectodomain binds diacylglycerol from microbial lipopeptides and glycolipids.
- Published in:
- Innate Immunity, 2015, v. 21, n. 2, p. 175, doi. 10.1177/1753425914524077
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- Publication type:
- Article
6
Gene expression of TLR homologues identified by genome-wide screening of the earthworm Dendrobaena veneta.
- Published in:
- Innate Immunity, 2015, v. 21, n. 2, p. 161, doi. 10.1177/1753425914523056
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- Publication type:
- Article
7
Campylobacter infection in chickens modulates the intestinal epithelial barrier function.
- Published in:
- Innate Immunity, 2015, v. 21, n. 2, p. 151, doi. 10.1177/1753425914521648
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- Publication type:
- Article
8
Activated skin γδ T-cells regulate T-cell infiltration of the wound site after burn.
- Published in:
- Innate Immunity, 2015, v. 21, n. 2, p. 140, doi. 10.1177/1753425913519350
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- Publication type:
- Article
9
Helicobacter pylori-driven modulation of NK cell expansion, intracellular cytokine expression and cytotoxic activity.
- Published in:
- Innate Immunity, 2015, v. 21, n. 2, p. 127, doi. 10.1177/1753425913518225
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- Publication type:
- Article
10
A disturbed interaction with accessory cells upon opportunistic infection with Pseudomonas aeruginosa contributes to an impaired IFN-γ production of NK cells in the lung during sepsis-induced immunosuppression.
- Published in:
- Innate Immunity, 2015, v. 21, n. 2, p. 115, doi. 10.1177/1753425913517274
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- Publication type:
- Article