Neuronal overexpression of COX-2 results in dominant production of PGE<sub>2</sub> and altered fever response.
- Published in:
- NeuroMolecular Medicine, 2003, v. 3, n. 1, p. 15, doi. 10.1385/NMM:3:1:15
- By:
- Publication type:
- Article
Works matching IS 15351084 AND DT 2003 AND VI 3 AND IP 1
Results: 5
1
2
Presenilin-1 mutation sensitizes oligodendrocytes to glutamate and amyloid toxicities, and exacerbates white matter damage and memory impairment in mice.
- Published in:
- NeuroMolecular Medicine, 2003, v. 3, n. 1, p. 53, doi. 10.1385/NMM:3:1:53
- By:
- Publication type:
- Article
3
NF2 Tumor suppressor gene.
- Published in:
- NeuroMolecular Medicine, 2003, v. 3, n. 1, p. 41, doi. 10.1385/NMM:3:1:41
- By:
- Publication type:
- Article
4
Autoantibodies to amyloid β-peptide (Aβ) are increased in Alzheimer’s disease patients and Aβ antibodies can enhance Aβ neurotoxicity.
- Published in:
- NeuroMolecular Medicine, 2003, v. 3, n. 1, p. 29, doi. 10.1385/NMM:3:1:29
- By:
- Publication type:
- Article
5
Parkin’s substrates and the pathways leading to neuronal damage.
- Published in:
- NeuroMolecular Medicine, 2003, v. 3, n. 1, p. 1, doi. 10.1385/NMM:3:1:1
- By:
- Publication type:
- Article