Hypertension: Potential Player in Cardiovascular Disease Incidence in Preeclampsia.
- Published in:
- Cardiovascular Toxicology, 2022, v. 22, n. 5, p. 391, doi. 10.1007/s12012-022-09734-w
- By:
- Publication type:
- Article
Works matching IS 15307905 AND DT 2022 AND VI 22 AND IP 5
Results: 6
1
2
Exogenous H<sub>2</sub>S Ameliorates High Salt-Induced Hypertension by Alleviating Oxidative Stress and Inflammation in the Paraventricular Nucleus in Dahl S Rats.
- Published in:
- Cardiovascular Toxicology, 2022, v. 22, n. 5, p. 477, doi. 10.1007/s12012-022-09729-7
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- Publication type:
- Article
3
Overexpression of Programmed Cell Death 1 Prevents Doxorubicin-Induced Apoptosis Through Autophagy Induction in H9c2 Cardiomyocytes.
- Published in:
- Cardiovascular Toxicology, 2022, v. 22, n. 5, p. 462, doi. 10.1007/s12012-022-09726-w
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- Publication type:
- Article
4
Exploring the Pattern of Metabolic Alterations Causing Energy Imbalance via PPARα Dysregulation in Cardiac Muscle During Doxorubicin Treatment.
- Published in:
- Cardiovascular Toxicology, 2022, v. 22, n. 5, p. 436, doi. 10.1007/s12012-022-09725-x
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- Publication type:
- Article
5
Upregulation of Uric Acid Production and Caspase 3 Signalling Mediates Rohypnol-Induced Cardiorenal Damage.
- Published in:
- Cardiovascular Toxicology, 2022, v. 22, n. 5, p. 419, doi. 10.1007/s12012-022-09723-z
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- Publication type:
- Article
6
Cardiac Expression of Esophageal Cancer-Related Gene-4 is Regulated by Sp1 and is a Potential Early Target of Doxorubicin-Induced Cardiotoxicity.
- Published in:
- Cardiovascular Toxicology, 2022, v. 22, n. 5, p. 404, doi. 10.1007/s12012-022-09722-0
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- Publication type:
- Article