Works matching IS 14749718 AND DT 2020 AND VI 19 AND IP 3
Results: 24
Increased miR‐34c mediates synaptic deficits by targeting synaptotagmin 1 through ROS‐JNK‐p53 pathway in Alzheimer's Disease.
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- Aging Cell, 2020, v. 19, n. 3, p. 1, doi. 10.1111/acel.13125
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Transcriptome analysis reveals the difference between "healthy" and "common" aging and their connection with age‐related diseases.
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- Aging Cell, 2020, v. 19, n. 3, p. 1, doi. 10.1111/acel.13121
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Sexual dimorphism in the nutritional requirement for adult lifespan in Drosophila melanogaster.
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- Aging Cell, 2020, v. 19, n. 3, p. 1, doi. 10.1111/acel.13120
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miR‐181a negatively modulates synaptic plasticity in hippocampal cultures and its inhibition rescues memory deficits in a mouse model of Alzheimer's disease.
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- Aging Cell, 2020, v. 19, n. 3, p. 1, doi. 10.1111/acel.13118
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Inhibition of USP7 activity selectively eliminates senescent cells in part via restoration of p53 activity.
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- Aging Cell, 2020, v. 19, n. 3, p. 1, doi. 10.1111/acel.13117
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A lncRNA survey finds increases in neuroprotective LINC‐PINT in Parkinson's disease substantia nigra.
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- Aging Cell, 2020, v. 19, n. 3, p. 1, doi. 10.1111/acel.13115
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TRPV1 activation alleviates cognitive and synaptic plasticity impairments through inhibiting AMPAR endocytosis in APP23/PS45 mouse model of Alzheimer's disease.
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- Aging Cell, 2020, v. 19, n. 3, p. 1, doi. 10.1111/acel.13113
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Age‐related changes to macrophages are detrimental to fracture healing in mice.
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- Aging Cell, 2020, v. 19, n. 3, p. 1, doi. 10.1111/acel.13112
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Melatonin suppresses senescence‐derived mitochondrial dysfunction in mesenchymal stem cells via the HSPA1L–mitophagy pathway.
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- Aging Cell, 2020, v. 19, n. 3, p. 1, doi. 10.1111/acel.13111
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Mobilization‐based transplantation of young‐donor hematopoietic stem cells extends lifespan in mice.
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- Aging Cell, 2020, v. 19, n. 3, p. 1, doi. 10.1111/acel.13110
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"Amyloid‐beta accumulation cycle" as a prevention and/or therapy target for Alzheimer's disease.
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- Aging Cell, 2020, v. 19, n. 3, p. 1, doi. 10.1111/acel.13109
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Heterochromatin loss as a determinant of progerin‐induced DNA damage in Hutchinson–Gilford Progeria.
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- Aging Cell, 2020, v. 19, n. 3, p. 1, doi. 10.1111/acel.13108
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Transplanting cells from old but not young donors causes physical dysfunction in older recipients.
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- Aging Cell, 2020, v. 19, n. 3, p. 1, doi. 10.1111/acel.13106
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Physical fitness in community‐dwelling older adults is linked to dietary intake, gut microbiota, and metabolomic signatures.
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- Aging Cell, 2020, v. 19, n. 3, p. 1, doi. 10.1111/acel.13105
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A high‐fat diet reverses metabolic disorders and premature aging by modulating insulin and IGF1 signaling in SIRT6 knockout mice.
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- Aging Cell, 2020, v. 19, n. 3, p. 1, doi. 10.1111/acel.13104
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Cellular senescence contributes to age‐dependent changes in circulating extracellular vesicle cargo and function.
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- Aging Cell, 2020, v. 19, n. 3, p. 1, doi. 10.1111/acel.13103
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LSD1 contributes to programmed oocyte death by regulating the transcription of autophagy adaptor SQSTM1/p62.
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- Aging Cell, 2020, v. 19, n. 3, p. 1, doi. 10.1111/acel.13102
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Parkinson's disease, aging and adult neurogenesis: Wnt/β‐catenin signalling as the key to unlock the mystery of endogenous brain repair.
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- Aging Cell, 2020, v. 19, n. 3, p. 1, doi. 10.1111/acel.13101
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Corrigendum.
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- Aging Cell, 2020, v. 19, n. 3, p. 1, doi. 10.1111/acel.13098
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- Article
Mitochondrial function in skeletal myofibers is controlled by a TRF2‐SIRT3 axis over lifetime.
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- Aging Cell, 2020, v. 19, n. 3, p. 1, doi. 10.1111/acel.13097
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Tissue specificity of senescent cell accumulation during physiologic and accelerated aging of mice.
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- Aging Cell, 2020, v. 19, n. 3, p. 1, doi. 10.1111/acel.13094
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Loss of tenomodulin expression is a risk factor for age‐related intervertebral disc degeneration.
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- Aging Cell, 2020, v. 19, n. 3, p. 1, doi. 10.1111/acel.13091
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Deficiency in the DNA repair protein ERCC1 triggers a link between senescence and apoptosis in human fibroblasts and mouse skin.
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- Aging Cell, 2020, v. 19, n. 3, p. 1, doi. 10.1111/acel.13072
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Issue Information.
- Published in:
- Aging Cell, 2020, v. 19, n. 3, p. 1, doi. 10.1111/acel.12974
- Publication type:
- Article