Works matching IS 14749718 AND DT 2018 AND VI 17 AND IP 2
Results: 25
Major features of immunesenescence, including reduced thymic output, are ameliorated by high levels of physical activity in adulthood.
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- Aging Cell, 2018, v. 17, n. 2, p. 1, doi. 10.1111/acel.12750
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Properties of the vastus lateralis muscle in relation to age and physiological function in master cyclists aged 55–79 years.
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- Aging Cell, 2018, v. 17, n. 2, p. 1, doi. 10.1111/acel.12735
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Emerging roles of extracellular vesicles in cellular senescence and aging.
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- Aging Cell, 2018, v. 17, n. 2, p. 1, doi. 10.1111/acel.12734
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Smurf2 regulates stability and the autophagic–lysosomal turnover of lamin A and its disease‐associated form progerin.
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- Aging Cell, 2018, v. 17, n. 2, p. 1, doi. 10.1111/acel.12732
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Treatment with the mitochondrial‐targeted antioxidant peptide SS‐31 rescues neurovascular coupling responses and cerebrovascular endothelial function and improves cognition in aged mice.
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- Aging Cell, 2018, v. 17, n. 2, p. 1, doi. 10.1111/acel.12731
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Transcriptomics of aged <italic>Drosophila</italic> motor neurons reveals a matrix metalloproteinase that impairs motor function.
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- Aging Cell, 2018, v. 17, n. 2, p. 1, doi. 10.1111/acel.12729
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Long‐lasting pathological consequences of overexpression‐induced α‐synuclein spreading in the rat brain.
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- Aging Cell, 2018, v. 17, n. 2, p. 1, doi. 10.1111/acel.12727
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α‐Motor neurons are spared from aging while their synaptic inputs degenerate in monkeys and mice.
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- Aging Cell, 2018, v. 17, n. 2, p. 1, doi. 10.1111/acel.12726
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Skeletal muscle ex vivo mitochondrial respiration parallels decline in vivo oxidative capacity, cardiorespiratory fitness, and muscle strength: The Baltimore Longitudinal Study of Aging.
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- Aging Cell, 2018, v. 17, n. 2, p. 1, doi. 10.1111/acel.12725
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Metformin regulates metabolic and nonmetabolic pathways in skeletal muscle and subcutaneous adipose tissues of older adults.
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- Aging Cell, 2018, v. 17, n. 2, p. 1, doi. 10.1111/acel.12723
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Senescence chips for ultrahigh‐throughput isolation and removal of senescent cells.
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- Aging Cell, 2018, v. 17, n. 2, p. 1, doi. 10.1111/acel.12722
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Ablation of PPARγ in subcutaneous fat exacerbates age‐associated obesity and metabolic decline.
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- Aging Cell, 2018, v. 17, n. 2, p. 1, doi. 10.1111/acel.12721
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ATF3 represses PINK1 gene transcription in lung epithelial cells to control mitochondrial homeostasis.
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- Aging Cell, 2018, v. 17, n. 2, p. 1, doi. 10.1111/acel.12720
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Effects of 2 years of caloric restriction on oxidative status assessed by urinary F2‐isoprostanes: The CALERIE 2 randomized clinical trial.
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- Aging Cell, 2018, v. 17, n. 2, p. 1, doi. 10.1111/acel.12719
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Endothelium‐specific CYP2J2 overexpression attenuates age‐related insulin resistance.
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- Aging Cell, 2018, v. 17, n. 2, p. 1, doi. 10.1111/acel.12718
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High‐throughput serum proteomics for the identification of protein biomarkers of mortality in older men.
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- Aging Cell, 2018, v. 17, n. 2, p. 1, doi. 10.1111/acel.12717
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T‐cell Immunoglobulin and ITIM Domain Contributes to CD8<sup>+</sup> T‐cell Immunosenescence.
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- Aging Cell, 2018, v. 17, n. 2, p. 1, doi. 10.1111/acel.12716
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The mitochondrial ATP synthase is a shared drug target for aging and dementia.
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- Aging Cell, 2018, v. 17, n. 2, p. 1, doi. 10.1111/acel.12715
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Aging affects the in vivo regenerative potential of human mesoangioblasts.
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- Aging Cell, 2018, v. 17, n. 2, p. 1, doi. 10.1111/acel.12714
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UNC‐120/SRF independently controls muscle aging and lifespan in <italic>Caenorhabditis elegans</italic>.
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- Aging Cell, 2018, v. 17, n. 2, p. 1, doi. 10.1111/acel.12713
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Senescence promotes in vivo reprogramming through p16<sup>INK</sup><sup>4a</sup> and IL‐6.
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- Aging Cell, 2018, v. 17, n. 2, p. 1, doi. 10.1111/acel.12711
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Overexpression of PGC‐1α in aging muscle enhances a subset of young‐like molecular patterns.
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- Aging Cell, 2018, v. 17, n. 2, p. 1, doi. 10.1111/acel.12707
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Circulating levels of monocyte chemoattractant protein‐1 as a potential measure of biological age in mice and frailty in humans.
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- Aging Cell, 2018, v. 17, n. 2, p. 1, doi. 10.1111/acel.12706
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Plasticity of lifelong calorie‐restricted C57BL/6J mice in adapting to a medium‐fat diet intervention at old age.
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- Aging Cell, 2018, v. 17, n. 2, p. 1, doi. 10.1111/acel.12696
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Issue Information.
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- Aging Cell, 2018, v. 17, n. 2, p. 1, doi. 10.1111/acel.12668
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- Article