Works matching IS 10788956 AND DT 2015 AND VI 21 AND IP 10
Results: 28
A dormant danger: New therapies target a ubiquitous pathogen known as cytomegalovirus.
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- Nature Medicine, 2015, v. 21, n. 10, p. 1104, doi. 10.1038/nm1015-1104
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Biomedical briefing.
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- Nature Medicine, 2015, v. 21, n. 10, p. 1112, doi. 10.1038/nm1015-1112
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- Article
Endogenous antigen processing drives the primary CD4<sup>+</sup> T cell response to influenza.
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- Nature Medicine, 2015, v. 21, n. 10, p. 1216, doi. 10.1038/nm.3958
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Remodeling the susceptibility to stress-induced depression.
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- Nature Medicine, 2015, v. 21, n. 10, p. 1125, doi. 10.1038/nm.3970
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Chikungunya is moving fast, but so are researchers in the field.
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- Nature Medicine, 2015, v. 21, n. 10, p. 1107, doi. 10.1038/nm1015-1107
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CD47 blockade as another immune checkpoint therapy for cancer.
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- Nature Medicine, 2015, v. 21, n. 10, p. 1122, doi. 10.1038/nm.3965
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CD47 blockade triggers T cell-mediated destruction of immunogenic tumors.
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- Nature Medicine, 2015, v. 21, n. 10, p. 1209, doi. 10.1038/nm.3931
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Bugging out over Chagas: Bioluminescent protozoans and old drugs might help unravel kissing-bug disease.
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- Nature Medicine, 2015, v. 21, n. 10, p. 1108, doi. 10.1038/nm1015-1108
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The histone lysine methyltransferase KMT2D sustains a gene expression program that represses B cell lymphoma development.
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- Nature Medicine, 2015, v. 21, n. 10, p. 1199, doi. 10.1038/nm.3943
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ACF chromatin-remodeling complex mediates stress-induced depressive-like behavior.
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- Nature Medicine, 2015, v. 21, n. 10, p. 1146, doi. 10.1038/nm.3939
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The Canadian MD/PhD training program needs reinstated support.
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- Nature Medicine, 2015, v. 21, n. 10, p. 1111, doi. 10.1038/nm1015-1111
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HIV-1 infections with multiple founders are associated with higher viral loads than infections with single founders.
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- Nature Medicine, 2015, v. 21, n. 10, p. 1139, doi. 10.1038/nm.3932
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The association between sterilizing activity and drug distribution into tuberculosis lesions.
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- Nature Medicine, 2015, v. 21, n. 10, p. 1223, doi. 10.1038/nm.3937
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Critical role of acetylation in tau-mediated neurodegeneration and cognitive deficits.
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- Nature Medicine, 2015, v. 21, n. 10, p. 1154, doi. 10.1038/nm.3951
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Characterization of extracellular DDX4- or Ddx4-positive ovarian cells.
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- Nature Medicine, 2015, v. 21, n. 10, p. 1114, doi. 10.1038/nm.3966
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Antigen-presenting cells look within during influenza infection.
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- Nature Medicine, 2015, v. 21, n. 10, p. 1123, doi. 10.1038/nm.3971
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A closer look at the 21<sup>st</sup> Century Cures Act.
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- Nature Medicine, 2015, v. 21, n. 10, p. 1103, doi. 10.1038/nm.3976
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- Article
Early life dynamics of the human gut virome and bacterial microbiome in infants.
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- Nature Medicine, 2015, v. 21, n. 10, p. 1228, doi. 10.1038/nm.3950
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Clinical translation of a high-performance neural prosthesis.
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- Nature Medicine, 2015, v. 21, n. 10, p. 1142, doi. 10.1038/nm.3953
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A detour in the quest for oogonial stem cells: methods matter.
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- Nature Medicine, 2015, v. 21, n. 10, p. 1126, doi. 10.1038/nm.3969
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Reply to Adult human and mouse ovaries lack DDX4-expressing functional oogonial stem cells.
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- Nature Medicine, 2015, v. 21, n. 10, p. 1118, doi. 10.1038/nm.3964
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MERS vaccines advance, but will humans or camels get the jab?
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- Nature Medicine, 2015, v. 21, n. 10, p. 1106, doi. 10.1038/nm1015-1106
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Minimal PU.1 reduction induces a preleukemic state and promotes development of acute myeloid leukemia.
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- Nature Medicine, 2015, v. 21, n. 10, p. 1172, doi. 10.1038/nm.3936
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Natural and therapy-induced immunosurveillance in breast cancer.
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- Nature Medicine, 2015, v. 21, n. 10, p. 1128, doi. 10.1038/nm.3944
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Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based therapy for pancreatic ductal adenocarcinoma.
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- Nature Medicine, 2015, v. 21, n. 10, p. 1163, doi. 10.1038/nm.3952
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Adult human and mouse ovaries lack DDX4-expressing functional oogonial stem cells.
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- Nature Medicine, 2015, v. 21, n. 10, p. 1116, doi. 10.1038/nm.3775
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Disruption of KMT2D perturbs germinal center B cell development and promotes lymphomagenesis.
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- Nature Medicine, 2015, v. 21, n. 10, p. 1190, doi. 10.1038/nm.3940
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Metabolic reprogramming induces resistance to anti-NOTCH1 therapies in T cell acute lymphoblastic leukemia.
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- Nature Medicine, 2015, v. 21, n. 10, p. 1182, doi. 10.1038/nm.3955
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