Works matching IS 10614036 AND DT 2015 AND VI 47 AND IP 4
Results: 26
Fine mapping of type 1 diabetes susceptibility loci and evidence for colocalization of causal variants with lymphoid gene enhancers.
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- Nature Genetics, 2015, v. 47, n. 4, p. 381, doi. 10.1038/ng.3245
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Genome-wide association analysis of more than 120,000 individuals identifies 15 new susceptibility loci for breast cancer.
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- Nature Genetics, 2015, v. 47, n. 4, p. 373, doi. 10.1038/ng.3242
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Identification of common genetic variants controlling transcript isoform variation in human whole blood.
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- Nature Genetics, 2015, v. 47, n. 4, p. 345, doi. 10.1038/ng.3220
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Analysis of the genetic phylogeny of multifocal prostate cancer identifies multiple independent clonal expansions in neoplastic and morphologically normal prostate tissue.
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- Nature Genetics, 2015, v. 47, n. 4, p. 367, doi. 10.1038/ng.3221
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Mouse modeling of Alzheimer's disease.
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- Nature Genetics, 2015, v. 47, n. 4, p. 311, doi. 10.1038/ng.3267
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ALS susceptibility genes.
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- Nature Genetics, 2015, v. 47, n. 4, p. 311, doi. 10.1038/ng.3266
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Analyses of allele-specific gene expression in highly divergent mouse crosses identifies pervasive allelic imbalance.
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- Nature Genetics, 2015, v. 47, n. 4, p. 353, doi. 10.1038/ng.3222
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Transcriptional mimicry by tumor-associated stroma.
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- Nature Genetics, 2015, v. 47, n. 4, p. 307, doi. 10.1038/ng.3255
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The landscape of somatic mutations in infant MLL-rearranged acute lymphoblastic leukemias.
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- Nature Genetics, 2015, v. 47, n. 4, p. 330, doi. 10.1038/ng.3230
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The two sides of GIGANTEA.
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- Nature Genetics, 2015, v. 47, n. 4, p. 311, doi. 10.1038/ng.3264
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Epigenetic evolution of corticogenesis.
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- Nature Genetics, 2015, v. 47, n. 4, p. 311, doi. 10.1038/ng.3265
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Convergent losses of decay mechanisms and rapid turnover of symbiosis genes in mycorrhizal mutualists.
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- Nature Genetics, 2015, v. 47, n. 4, p. 410, doi. 10.1038/ng.3223
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New genomes clarify mimicry evolution.
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- Nature Genetics, 2015, v. 47, n. 4, p. 306, doi. 10.1038/ng.3260
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CRISPR screen for metastasis.
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- Nature Genetics, 2015, v. 47, n. 4, p. 311, doi. 10.1038/ng.3263
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A common variant mapping to CACNA1A is associated with susceptibility to exfoliation syndrome.
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- Nature Genetics, 2015, v. 47, n. 4, p. 387, doi. 10.1038/ng.3226
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Stromal contribution to the colorectal cancer transcriptome.
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- Nature Genetics, 2015, v. 47, n. 4, p. 312, doi. 10.1038/ng.3224
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A genetic mechanism for female-limited Batesian mimicry in Papilio butterfly.
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- Nature Genetics, 2015, v. 47, n. 4, p. 405, doi. 10.1038/ng.3241
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The genome and transcriptome of the zoonotic hookworm Ancylostoma ceylanicum identify infection-specific gene families.
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- Nature Genetics, 2015, v. 47, n. 4, p. 416, doi. 10.1038/ng.3237
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Germline gain-of-function mutations in AFF4 cause a developmental syndrome functionally linking the super elongation complex and cohesin.
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- Nature Genetics, 2015, v. 47, n. 4, p. 338, doi. 10.1038/ng.3229
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A single natural nucleotide mutation alters bacterial pathogen host tropism.
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- Nature Genetics, 2015, v. 47, n. 4, p. 361, doi. 10.1038/ng.3219
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Cause, correlation, conjecture.
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- Nature Genetics, 2015, v. 47, n. 4, p. 305, doi. 10.1038/ng.3271
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De novo loss- or gain-of-function mutations in KCNA2 cause epileptic encephalopathy.
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- Nature Genetics, 2015, v. 47, n. 4, p. 393, doi. 10.1038/ng.3239
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Symbiotic plant-fungi interactions stripped down to the root.
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- Nature Genetics, 2015, v. 47, n. 4, p. 309, doi. 10.1038/ng.3261
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Corrigendum: Common variants in ACYP2 influence susceptibility to cisplatin-induced hearing loss.
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- Nature Genetics, 2015, v. 47, n. 4, p. 423, doi. 10.1038/ng0415-423
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Recombination affects accumulation of damaging and disease-associated mutations in human populations.
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- Nature Genetics, 2015, v. 47, n. 4, p. 400, doi. 10.1038/ng.3216
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Stromal gene expression defines poor-prognosis subtypes in colorectal cancer.
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- Nature Genetics, 2015, v. 47, n. 4, p. 320, doi. 10.1038/ng.3225
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