Works matching IS 10233830 AND DT 2023 AND VI 72 AND IP 3
Results: 20
IL-33 induces histidine decarboxylase, especially in c-kit<sup>+</sup> cells and mast cells, and roles of histamine include negative regulation of IL-33-induced eosinophilia.
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- Inflammation Research, 2023, v. 72, n. 3, p. 651, doi. 10.1007/s00011-023-01699-y
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- Article
Exocytic machineries differentially control mediator release from allergen-triggered RBL-2H3 cells.
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- Inflammation Research, 2023, v. 72, n. 3, p. 639, doi. 10.1007/s00011-023-01698-z
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The potential role of N6-methyladenosine modification of LncRNAs in contributing to the pathogenesis of chronic glomerulonephritis.
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- Inflammation Research, 2023, v. 72, n. 3, p. 623, doi. 10.1007/s00011-023-01695-2
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Uncovering the role of transient receptor potential channels in pterygium: a machine learning approach.
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- Inflammation Research, 2023, v. 72, n. 3, p. 589, doi. 10.1007/s00011-023-01693-4
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The molecular mechanisms of remodeling in asthma, COPD and IPF with a special emphasis on the complex role of Wnt5A.
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- Inflammation Research, 2023, v. 72, n. 3, p. 577, doi. 10.1007/s00011-023-01692-5
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γ-Glutamylcysteine rescues mice from TNBS-driven inflammatory bowel disease through regulating macrophages polarization.
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- Inflammation Research, 2023, v. 72, n. 3, p. 603, doi. 10.1007/s00011-023-01691-6
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Eosinophilia and elevated IgE serum levels: a red flag: when your diagnosis is not a common atopic eczema or common allergy.
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- Inflammation Research, 2023, v. 72, n. 3, p. 541, doi. 10.1007/s00011-023-01690-7
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circFLNA promotes intestinal injury during abdominal sepsis through Fas-mediated apoptosis pathway by sponging miR-766-3p.
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- Inflammation Research, 2023, v. 72, n. 3, p. 509, doi. 10.1007/s00011-023-01688-1
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Inflammation, coagulation, and cellular injury in heat-induced shock.
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- Inflammation Research, 2023, v. 72, n. 3, p. 463, doi. 10.1007/s00011-022-01687-8
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Inhibition of gp130 alleviates LPS-induced lung injury by attenuating apoptosis and inflammation through JAK1/STAT3 signaling pathway.
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- Inflammation Research, 2023, v. 72, n. 3, p. 493, doi. 10.1007/s00011-022-01686-9
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TCR repertoire landscape reveals macrophage-mediated clone deletion in endotoxin tolerance.
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- Inflammation Research, 2023, v. 72, n. 3, p. 531, doi. 10.1007/s00011-022-01685-w
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New frontiers in inflammation: from translational research to clinic (information about the 15th World Congress on Inflammation (WCI2022)).
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- Inflammation Research, 2023, v. 72, n. 3, p. 425, doi. 10.1007/s00011-022-01684-x
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Evaluation of urinary cysteinyl leukotrienes as biomarkers of severity and putative therapeutic targets in COVID-19 patients.
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- Inflammation Research, 2023, v. 72, n. 3, p. 475, doi. 10.1007/s00011-022-01682-z
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- Article
m6A-related bioinformatics analysis and functional characterization reveals that METTL3-mediated NPC1L1 mRNA hypermethylation facilitates progression of atherosclerosis via inactivation of the MAPK pathway.
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- Inflammation Research, 2023, v. 72, n. 3, p. 429, doi. 10.1007/s00011-022-01681-0
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Loss of pleckstrin homology domain and leucine-rich repeat protein phosphatase 2 has protective effects on high glucose-injured retinal ganglion cells via the effect on the Akt–GSK–3β–Nrf2 pathway.
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- Inflammation Research, 2023, v. 72, n. 3, p. 373, doi. 10.1007/s00011-022-01680-1
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Periostin regulation and cartilage degradation early after anterior cruciate ligament reconstruction.
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- Inflammation Research, 2023, v. 72, n. 3, p. 387, doi. 10.1007/s00011-022-01678-9
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Tissue-resident immune cells in the pathogenesis of multiple sclerosis.
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- Inflammation Research, 2023, v. 72, n. 3, p. 363, doi. 10.1007/s00011-022-01677-w
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Role of α-synuclein in microglia: autophagy and phagocytosis balance neuroinflammation in Parkinson's disease.
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- Inflammation Research, 2023, v. 72, n. 3, p. 443, doi. 10.1007/s00011-022-01676-x
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ADAM10-a "multitasker" in sepsis: focus on its posttranslational target.
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- Inflammation Research, 2023, v. 72, n. 3, p. 395, doi. 10.1007/s00011-022-01673-0
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Human bone marrow mesenchymal stem cell-derived extracellular vesicles reduce inflammation and pyroptosis in acute kidney injury via miR-223-3p/HDAC2/SNRK.
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- Inflammation Research, 2023, v. 72, n. 3, p. 553, doi. 10.1007/s00011-022-01653-4
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