IL-33 in obesity: where do we go from here?
- Published in:
- Inflammation Research, 2019, v. 68, n. 3, p. 185, doi. 10.1007/s00011-019-01214-2
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- Publication type:
- Article
Works matching IS 10233830 AND DT 2019 AND VI 68 AND IP 3
Results: 8
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MiR-19a mediates the negative regulation of the NF-κB pathway in lipopolysaccharide-induced endometritis by targeting TBK1.
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- Inflammation Research, 2019, v. 68, n. 3, p. 231, doi. 10.1007/s00011-019-01213-3
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- Article
3
Giulio Gabbiani and the discovery of myofibroblasts.
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- Inflammation Research, 2019, v. 68, n. 3, p. 241, doi. 10.1007/s00011-018-01211-x
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- Article
4
Malt1 inactivation attenuates experimental colitis through the regulation of Th17 and Th1/17 cells.
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- Inflammation Research, 2019, v. 68, n. 3, p. 223, doi. 10.1007/s00011-018-1207-y
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- Article
5
miR-196a2 (rs11614913) polymorphism is associated with coronary artery disease, but not with in-stent coronary restenosis.
- Published in:
- Inflammation Research, 2019, v. 68, n. 3, p. 215, doi. 10.1007/s00011-018-1206-z
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- Article
6
Immunological modulation following bone marrow-derived mesenchymal stromal cells and Th17 lymphocyte co-cultures.
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- Inflammation Research, 2019, v. 68, n. 3, p. 203, doi. 10.1007/s00011-018-1205-0
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- Article
7
An association study in PTPN22 suggests that is a risk factor to Takayasu's arteritis.
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- Inflammation Research, 2019, v. 68, n. 3, p. 195, doi. 10.1007/s00011-018-1204-1
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- Article
8
Promotion of microtubule acetylation plays an important role in degranulation of antigen-activated mast cells.
- Published in:
- Inflammation Research, 2019, v. 68, n. 3, p. 181, doi. 10.1007/s00011-018-1203-2
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- Publication type:
- Article