Works matching IS 09509232 AND DT 2000 AND VI 19 AND IP 29
Results: 11
A Drosophila analogue of v-Cbl is a dominant-negative oncoprotein in vivo.
- Published in:
- Oncogene, 2000, v. 19, n. 29, p. 3299, doi. 10.1038/sj.onc.1203624
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- Article
STAT3-mediated differentiation and survival of myeloid cells in response to granulocyte colony-stimulating factor: role for the cyclin-dependent kinase inhibitor p27<sup>Kip1</sup>.
- Published in:
- Oncogene, 2000, v. 19, n. 29, p. 3290, doi. 10.1038/sj.onc.1203627
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- Article
Normal and c-Myc-promoted human keratinocyte differentiation both occur via a novel cell cycle involving cellular growth and endoreplication.
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- Oncogene, 2000, v. 19, n. 29, p. 3278, doi. 10.1038/sj.onc.1203630
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- Article
Mlx, a new Max-like bHLHZip family member: the center stage of a novel transcription factors regulatory pathway?
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- Oncogene, 2000, v. 19, n. 29, p. 3266, doi. 10.1038/sj.onc.1203634
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- Article
A genetic screen to identify genes that rescue the slow growth phenotype of c-myc null fibroblasts.
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- Oncogene, 2000, v. 19, n. 29, p. 3330, doi. 10.1038/sj.onc.1203639
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- Article
Differential transcriptional regulation of the monocyte-chemoattractant protein-1 (MCP-1) gene in tumorigenic and non-tumorigenic HPV 18 positive cells: The role of the chromatin structure and AP-1 composition.
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- Oncogene, 2000, v. 19, n. 29, p. 3235, doi. 10.1038/sj.onc.1203643
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- Article
Substrate specificity of the p53-associated 3′-5′ exonuclease.
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- Oncogene, 2000, v. 19, n. 29, p. 3321, doi. 10.1038/sj.onc.1203649
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- Article
Transformation and Stat activation by derivatives of FGFR1, FGFR3, and FGFR4.
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- Oncogene, 2000, v. 19, n. 29, p. 3309, doi. 10.1038/sj.onc.1203650
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- Article
Caveolin-1 is regulated by c-myc and suppresses c-myc-induced apoptosis.
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- Oncogene, 2000, v. 19, n. 29, p. 3256, doi. 10.1038/sj.onc.1203654
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- Article
Cooperative transformation of 32D cells by the combined expression of IRS-1 and V-Ha-Ras.
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- Oncogene, 2000, v. 19, n. 29, p. 3245, doi. 10.1038/sj.onc.1203664
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- Article
Survivin initiates cell cycle entry by the competitive interaction with Cdk4/p16<sup>INK4a</sup> and Cdk2/Cyclin E complex activation.
- Published in:
- Oncogene, 2000, v. 19, n. 29, p. 3225, doi. 10.1038/sj.onc.1203665
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- Article