Works matching IS 09196544 AND DT 2001 AND VI 21 AND IP 1


Results: 17
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    Formation of advanced glycation end-product-modified superoxide dismutase-1 (SOD1) is one of the mechanisms responsible for inclusions common to familial amyotrophic lateral sclerosis patients with SOD1 gene mutation, and transgenic mice expressing human SOD1 gene mutation.

    Published in:
    Neuropathology, 2001, v. 21, n. 1, p. 67, doi. 10.1046/j.1440-1789.2001.00359.x
    By:
    • Kato, S.;
    • Nakashima, K.;
    • Horiuchi, S.;
    • Nagai, R.;
    • Cleveland, D.W.;
    • Liu, J.;
    • Hirano, A.;
    • Takikawa, M.;
    • Kato, M.;
    • Nakano, I.;
    • Sakoda, S.;
    • Asayama, K.;
    • Ohama, E.
    Publication type:
    Article
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