Multiplication of the α-synuclein gene is not a common disease mechanism in Lewy body disease.
- Published in:
- Journal of Molecular Neuroscience, 2004, v. 24, n. 3, p. 337, doi. 10.1385/JMN:24:3:337
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- Publication type:
- Article
Works matching IS 08958696 AND DT 2004 AND VI 24 AND IP 3
Results: 8
1
2
Parkinson’s and Alzheimer’s diseases.
- Published in:
- Journal of Molecular Neuroscience, 2004, v. 24, n. 3, p. 333, doi. 10.1385/JMN:24:3:333
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- Article
3
The role of α-synuclein assembly and metabolism in the pathogenesis of Lewy body disease.
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- Journal of Molecular Neuroscience, 2004, v. 24, n. 3, p. 343, doi. 10.1385/JMN:24:3:343
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- Article
4
Iron and α-synuclein in the substantia nigra of MPTP-treated mice.
- Published in:
- Journal of Molecular Neuroscience, 2004, v. 24, n. 3, p. 401, doi. 10.1385/JMN:24:3:401
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- Article
5
New aspects of genetic contributions to Parkinson’s disease.
- Published in:
- Journal of Molecular Neuroscience, 2004, v. 24, n. 3, p. 417, doi. 10.1385/JMN:24:3:417
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- Publication type:
- Article
6
Pathological proteins in Parkinson’s disease.
- Published in:
- Journal of Molecular Neuroscience, 2004, v. 24, n. 3, p. 425, doi. 10.1385/JMN:24:3:425
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- Publication type:
- Article
7
Embryonic and adult stem cells as a source for cell therapy in Parkinson’s disease.
- Published in:
- Journal of Molecular Neuroscience, 2004, v. 24, n. 3, p. 353, doi. 10.1385/JMN:24:3:353
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- Publication type:
- Article
8
Oxidative stress, induced by 6-hydroxydopamine, reduces proteasome activities in PC12 cells.
- Published in:
- Journal of Molecular Neuroscience, 2004, v. 24, n. 3, p. 387, doi. 10.1385/JMN:24:3:387
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- Publication type:
- Article