Introduction to the Special Issue in Honor of Graham L Collingridge.
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- Neurochemical Research, 2019, v. 44, n. 3, p. 513, doi. 10.1007/s11064-019-02751-2
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Works matching IS 03643190 AND DT 2019 AND VI 44 AND IP 3
Results: 21
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The Barnes Maze Task Reveals Specific Impairment of Spatial Learning Strategy in the Intrahippocampal Kainic Acid Model for Temporal Lobe Epilepsy.
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- Neurochemical Research, 2019, v. 44, n. 3, p. 600, doi. 10.1007/s11064-018-2610-z
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NMDA Receptor Dependent Long-term Potentiation in Chronic Pain.
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- Neurochemical Research, 2019, v. 44, n. 3, p. 531, doi. 10.1007/s11064-018-2614-8
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4
Gβγ SNARE Interactions and Their Behavioral Effects.
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- Neurochemical Research, 2019, v. 44, n. 3, p. 636, doi. 10.1007/s11064-018-2531-x
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5
Cannabidiol Reverses Deficits in Hippocampal LTP in a Model of Alzheimer's Disease.
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- Neurochemical Research, 2019, v. 44, n. 3, p. 703, doi. 10.1007/s11064-018-2513-z
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6
Scalable Measurements of Intrinsic Excitability in Human iPS Cell-Derived Excitatory Neurons Using All-Optical Electrophysiology.
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- Neurochemical Research, 2019, v. 44, n. 3, p. 714, doi. 10.1007/s11064-018-2694-5
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7
Muscarinic Receptor Modulation of the Cerebellar Interpositus Nucleus In Vitro.
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- Neurochemical Research, 2019, v. 44, n. 3, p. 627, doi. 10.1007/s11064-018-2613-9
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8
Susceptibility to Aβo and TBOA of LTD and Extrasynaptic NMDAR-Dependent Tonic Current in the Aged Rat Hippocampus.
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- Neurochemical Research, 2019, v. 44, n. 3, p. 692, doi. 10.1007/s11064-018-2677-6
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9
Assembly and Trafficking of Homomeric and Heteromeric Kainate Receptors with Impaired Ligand Binding Sites.
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- Neurochemical Research, 2019, v. 44, n. 3, p. 585, doi. 10.1007/s11064-018-2654-0
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10
The 1980s: D-AP5, LTP and a Decade of NMDA Receptor Discoveries.
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- Neurochemical Research, 2019, v. 44, n. 3, p. 516, doi. 10.1007/s11064-018-2640-6
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11
AMPA-Type Glutamate Receptor Conductance Changes and Plasticity: Still a Lot of Noise.
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- Neurochemical Research, 2019, v. 44, n. 3, p. 539, doi. 10.1007/s11064-018-2491-1
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Muscarinic Receptor-Dependent Long Term Depression in the Perirhinal Cortex and Recognition Memory are Impaired in the rTg4510 Mouse Model of Tauopathy.
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- Neurochemical Research, 2019, v. 44, n. 3, p. 617, doi. 10.1007/s11064-018-2487-x
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13
PDGF Modulates Synaptic Excitability and Short-Latency Afferent Inhibition in Multiple Sclerosis.
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- Neurochemical Research, 2019, v. 44, n. 3, p. 726, doi. 10.1007/s11064-018-2484-0
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14
Disturbance of Metabotropic Glutamate Receptor-Mediated Long-Term Depression (mGlu-LTD) of Excitatory Synaptic Transmission in the Rat Hippocampus After Prenatal Immune Challenge.
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- Neurochemical Research, 2019, v. 44, n. 3, p. 609, doi. 10.1007/s11064-018-2476-0
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15
Exciting Times: New Advances Towards Understanding the Regulation and Roles of Kainate Receptors.
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- Neurochemical Research, 2019, v. 44, n. 3, p. 572, doi. 10.1007/s11064-017-2450-2
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16
The AMPA Receptor Subunit GluA1 is Required for CA1 Hippocampal Long-Term Potentiation but is not Essential for Synaptic Transmission.
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- Neurochemical Research, 2019, v. 44, n. 3, p. 549, doi. 10.1007/s11064-017-2425-3
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17
Regulation of Neurotransmitter Release by Amyloid Precursor Protein Through Synapsin Phosphorylation.
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- Neurochemical Research, 2019, v. 44, n. 3, p. 683, doi. 10.1007/s11064-017-2418-2
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18
Decreased Neuron Number and Synaptic Plasticity in SIRT3-Knockout Mice with Poor Remote Memory.
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- Neurochemical Research, 2019, v. 44, n. 3, p. 676, doi. 10.1007/s11064-017-2417-3
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19
GluA4 Dependent Plasticity Mechanisms Contribute to Developmental Synchronization of the CA3-CA1 Circuitry in the Hippocampus.
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- Neurochemical Research, 2019, v. 44, n. 3, p. 562, doi. 10.1007/s11064-017-2392-8
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The Purine Salvage Pathway and the Restoration of Cerebral ATP: Implications for Brain Slice Physiology and Brain Injury.
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- Neurochemical Research, 2019, v. 44, n. 3, p. 661, doi. 10.1007/s11064-017-2386-6
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21
Leptin Regulation of Synaptic Function at Hippocampal TA-CA1 and SC-CA1 Synapses: Implications for Health and Disease.
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- Neurochemical Research, 2019, v. 44, n. 3, p. 650, doi. 10.1007/s11064-017-2362-1
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