Works matching IS 03008428 AND DT 2024 AND VI 119 AND IP 5

Results: 9

Deficiency of the sphingosine-1-phosphate (S1P) transporter Mfsd2b protects the heart against hypertension-induced cardiac remodeling by suppressing the L-type-Ca2+ channel.

Published in:

Basic Research in Cardiology, 2024, v. 119, n. 5, p. 853, doi. 10.1007/s00395-024-01073-x

By:

Duse, Dragos Andrei;
Schröder, Nathalie Hannelore;
Srivastava, Tanu;
Benkhoff, Marcel;
Vogt, Jens;
Nowak, Melissa Kim;
Funk, Florian;
Semleit, Nina;
Wollnitzke, Philipp;
Erkens, Ralf;
Kötter, Sebastian;
Meuth, Sven Günther;
Keul, Petra;
Santos, Webster;
Polzin, Amin;
Kelm, Malte;
Krüger, Martina;
Schmitt, Joachim;
Levkau, Bodo

Publication type:

Article

β3-Adrenergic receptor overexpression in cardiomyocytes preconditions mitochondria to withstand ischemia–reperfusion injury.

Published in:

Basic Research in Cardiology, 2024, v. 119, n. 5, p. 773, doi. 10.1007/s00395-024-01072-y

By:

Fernández-Tocino, Miguel;
Pun-Garcia, Andrés;
Gómez, Mónica;
Clemente-Moragón, Agustín;
Oliver, Eduardo;
Villena-Gutierrez, Rocío;
Trigo-Anca, Sofía;
Díaz-Guerra, Anabel;
Sanz-Rosa, David;
Prados, Belén;
del Campo, Lara;
Andrés, Vicente;
Fuster, Valentín;
de la Pompa, José Luis;
Cádiz, Laura;
Ibañez, Borja

Publication type:

Article

SGLT1 contributes to glucose-mediated exacerbation of ischemia–reperfusion injury in ex vivo rat heart.

Published in:

Basic Research in Cardiology, 2024, v. 119, n. 5, p. 733, doi. 10.1007/s00395-024-01071-z

By:

Almalki, Alhanoof;
Arjun, Sapna;
Harding, Idris;
Jasem, Hussain;
Kolatsi-Joannou, Maria;
Jafree, Daniyal J.;
Pomeranz, Gideon;
Long, David A.;
Yellon, Derek M.;
Bell, Robert M.

Publication type:

Article

Recombinant human placental growth factor-2 in post-infarction left ventricular dysfunction: a randomized, placebo-controlled, preclinical study.

Published in:

Basic Research in Cardiology, 2024, v. 119, n. 5, p. 795, doi. 10.1007/s00395-024-01069-7

By:

Wu, Ming;
Pokreisz, Peter;
Claus, Piet;
Casazza, Andrea;
Gillijns, Hilde;
Caluwé, Ellen;
De Petrini, Marzia;
Belmans, Ann;
Reyns, Geert;
Collen, Desire;
Janssens, Stefan P.

Publication type:

Article

Empagliflozin prevents heart failure through inhibition of the NHE1-NO pathway, independent of SGLT2.

Published in:

Basic Research in Cardiology, 2024, v. 119, n. 5, p. 751, doi. 10.1007/s00395-024-01067-9

By:

Chen, Sha;
Wang, Qian;
Bakker, Diane;
Hu, Xin;
Zhang, Liping;
van der Made, Ingeborg;
Tebbens, Anna M.;
Kovácsházi, Csenger;
Giricz, Zoltán;
Brenner, Gábor B.;
Ferdinandy, Peter;
Schaart, Gert;
Gemmink, Anne;
Hesselink, Matthijs K. C.;
Rivaud, Mathilde R.;
Pieper, Michael P.;
Hollmann, Markus W.;
Weber, Nina C.;
Balligand, Jean-Luc;
Creemers, Esther E.

Publication type:

Article

Targeting Gαi2 in neutrophils protects from myocardial ischemia reperfusion injury.

Published in:

Basic Research in Cardiology, 2024, v. 119, n. 5, p. 717, doi. 10.1007/s00395-024-01057-x

By:

Köhler, David;
Leiss, Veronika;
Beichert, Lukas;
Killinger, Simon;
Grothe, Daniela;
Kushwaha, Ragini;
Schröter, Agnes;
Roslan, Anna;
Eggstein, Claudia;
Focken, Jule;
Granja, Tiago;
Devanathan, Vasudharani;
Schittek, Birgit;
Lukowski, Robert;
Weigelin, Bettina;
Rosenberger, Peter;
Nürnberg, Bernd;
Beer-Hammer, Sandra

Publication type:

Article

Chronic kidney disease activates the HDAC6-inflammatory axis in the heart and contributes to myocardial remodeling in mice: inhibition of HDAC6 alleviates chronic kidney disease-induced myocardial remodeling.

Published in:

Basic Research in Cardiology, 2024, v. 119, n. 5, p. 831, doi. 10.1007/s00395-024-01056-y

By:

Kundu, Sourav;
Gairola, Shobhit;
Verma, Smriti;
Mugale, Madhav Nilakanth;
Sahu, Bidya Dhar

Publication type:

Article

Vasodilator reactive oxygen species ameliorate perturbed myocardial oxygen delivery in exercising swine with multiple comorbidities.

Published in:

Basic Research in Cardiology, 2024, v. 119, n. 5, p. 869, doi. 10.1007/s00395-024-01055-z

By:

van Drie, R. W. A.;
van de Wouw, J.;
Zandbergen, L. M.;
Dehairs, J.;
Swinnen, J. V.;
Mulder, M. T.;
Verhaar, M. C.;
MaassenVanDenBrink, A.;
Duncker, D. J.;
Sorop, O.;
Merkus, D.

Publication type:

Article

Increased [18F]FDG uptake in the infarcted myocardial area displayed by combined PET/CMR correlates with snRNA-seq-detected inflammatory cell invasion.

Published in:

Basic Research in Cardiology, 2024, v. 119, n. 5, p. 807, doi. 10.1007/s00395-024-01064-y

By:

Lukovic, Dominika;
Gyöngyösi, Mariann;
Pavo, Imre J.;
Mester-Tonczar, Julia;
Einzinger, Patrick;
Zlabinger, Katrin;
Kastner, Nina;
Spannbauer, Andreas;
Traxler, Denise;
Pavo, Noemi;
Goliasch, Georg;
Pils, Dietmar;
Jakab, Andras;
Szankai, Zsuzsanna;
Michel-Behnke, Ina;
Zhang, Lu;
Devaux, Yvan;
Graf, Senta;
Beitzke, Dietrich;
Winkler, Johannes

Publication type:

Article

Works matching IS 03008428 AND DT 2024 AND VI 119 AND IP 5

Deficiency of the sphingosine-1-phosphate (S1P) transporter Mfsd2b protects the heart against hypertension-induced cardiac remodeling by suppressing the L-type-Ca<sup>2+</sup> channel.

β3-Adrenergic receptor overexpression in cardiomyocytes preconditions mitochondria to withstand ischemia–reperfusion injury.

SGLT1 contributes to glucose-mediated exacerbation of ischemia–reperfusion injury in ex vivo rat heart.

Recombinant human placental growth factor-2 in post-infarction left ventricular dysfunction: a randomized, placebo-controlled, preclinical study.

Empagliflozin prevents heart failure through inhibition of the NHE1-NO pathway, independent of SGLT2.

Targeting Gα<sub>i2</sub> in neutrophils protects from myocardial ischemia reperfusion injury.

Chronic kidney disease activates the HDAC6-inflammatory axis in the heart and contributes to myocardial remodeling in mice: inhibition of HDAC6 alleviates chronic kidney disease-induced myocardial remodeling.

Vasodilator reactive oxygen species ameliorate perturbed myocardial oxygen delivery in exercising swine with multiple comorbidities.

Increased [<sup>18</sup>F]FDG uptake in the infarcted myocardial area displayed by combined PET/CMR correlates with snRNA-seq-detected inflammatory cell invasion.