Works matching IS 03008428 AND DT 2024 AND VI 119 AND IP 4
Results: 10
Bone marrow cells contribute to seven different endothelial cell populations in the heart.
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- Basic Research in Cardiology, 2024, v. 119, n. 4, p. 699, doi. 10.1007/s00395-024-01065-x
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- Article
Malonate given at reperfusion prevents post-myocardial infarction heart failure by decreasing ischemia/reperfusion injury.
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- Basic Research in Cardiology, 2024, v. 119, n. 4, p. 691, doi. 10.1007/s00395-024-01063-z
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Cardioprotection in cardiovascular surgery.
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- Basic Research in Cardiology, 2024, v. 119, n. 4, p. 545, doi. 10.1007/s00395-024-01062-0
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- Article
Gasotransmitters and noble gases in cardioprotection: unraveling molecular pathways for future therapeutic strategies.
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- Basic Research in Cardiology, 2024, v. 119, n. 4, p. 509, doi. 10.1007/s00395-024-01061-1
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- Article
Mitochondrial calcium in cardiac ischemia/reperfusion injury and cardioprotection.
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- Basic Research in Cardiology, 2024, v. 119, n. 4, p. 569, doi. 10.1007/s00395-024-01060-2
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- Article
Connexin 43 modulates reverse electron transfer in cardiac mitochondria from inducible knock-out Cx43<sup>Cre−ER(T)/fl</sup> mice by altering the coenzyme Q pool.
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- Basic Research in Cardiology, 2024, v. 119, n. 4, p. 673, doi. 10.1007/s00395-024-01052-2
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Comparison of the stage-dependent mitochondrial changes in response to pressure overload between the diseased right and left ventricle in the rat.
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- Basic Research in Cardiology, 2024, v. 119, n. 4, p. 587, doi. 10.1007/s00395-024-01051-3
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- Article
Circular RNA circZFPM2 regulates cardiomyocyte hypertrophy and survival.
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- Basic Research in Cardiology, 2024, v. 119, n. 4, p. 613, doi. 10.1007/s00395-024-01048-y
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- Article
Role of inflammatory signaling pathways involving the CD40–CD40L–TRAF cascade in diabetes and hypertension—insights from animal and human studies.
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- Basic Research in Cardiology, 2024, v. 119, n. 4, p. 1, doi. 10.1007/s00395-024-01045-1
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- Article
Exercise training decreases lactylation and prevents myocardial ischemia–reperfusion injury by inhibiting YTHDF2.
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- Basic Research in Cardiology, 2024, v. 119, n. 4, p. 651, doi. 10.1007/s00395-024-01044-2
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- Article