Works matching IS 03008428 AND DT 2023 AND VI 118 AND IP 1
Results: 50
Translational control of Ybx1 expression regulates cardiac function in response to pressure overload in vivo.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-00996-1
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Retraction Note to: NR4A1 aggravates the cardiac microvascular ischemia reperfusion injury through suppressing FUNDC1‑mediated mitophagy and promoting Mff‑required mitochondrial fission by CK2α.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-00994-3
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No robust reduction of infarct size and no-reflow by metoprolol pretreatment in adult Göttingen minipigs.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-00993-4
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Deficiency of mitochondrial calcium uniporter abrogates iron overload-induced cardiac dysfunction by reducing ferroptosis.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-00990-7
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Integration of epigenetic regulatory mechanisms in heart failure.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-00986-3
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Oxygen-sensing pathways below autoregulatory threshold act to sustain myocardial oxygen delivery during reductions in perfusion pressure.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-00985-4
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Membrane remodelling triggers maturation of excitation–contraction coupling in 3D-shaped human-induced pluripotent stem cell-derived cardiomyocytes.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-00984-5
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Multi-omic analysis of the cardiac cellulome defines a vascular contribution to cardiac diastolic dysfunction in obese female mice.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-00983-6
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Ploidy-stratified single cardiomyocyte transcriptomics map Zinc Finger E-Box Binding Homeobox 1 to underly cardiomyocyte proliferation before birth.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-00979-2
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Single-cell RNA sequencing to identify cellular heterogeneity and targets in cardiovascular diseases: from bench to bedside.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-022-00972-1
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Activation of the integrated stress response rewires cardiac metabolism in Barth syndrome.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-01017-x
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Targeting mitochondrial shape: at the heart of cardioprotection.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-01019-9
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The role of glycolytic metabolic pathways in cardiovascular disease and potential therapeutic approaches.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-01018-w
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Activation of the integrated stress response rewires cardiac metabolism in Barth syndrome.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-01017-x
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- Article
Musashi-2 causes cardiac hypertrophy and heart failure by inducing mitochondrial dysfunction through destabilizing Cluh and Smyd1 mRNA.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-01016-y
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Succinate dehydrogenase is essential for epigenetic and metabolic homeostasis in hearts.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-01015-z
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Splenic monocytes mediate inflammatory response and exacerbate myocardial ischemia/reperfusion injury in a mitochondrial cell-free DNA-TLR9-NLRP3-dependent fashion.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-01014-0
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Outcomes of hypothalamic oxytocin neuron-driven cardioprotection after acute myocardial infarction.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-01013-1
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Mitophagy for cardioprotection.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-01009-x
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Recent advances on the role of monoamine oxidases in cardiac pathophysiology.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-01012-2
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Novel GSDMD inhibitor GI-Y1 protects heart against pyroptosis and ischemia/reperfusion injury by blocking pyroptotic pore formation.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-01010-4
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Giant mitochondria in cardiomyocytes: cellular architecture in health and disease.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-01011-3
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The roles of intracellular proteolysis in cardiac ischemia–reperfusion injury.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-01007-z
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Ketone body 3-hydroxybutyrate elevates cardiac output through peripheral vasorelaxation and enhanced cardiac contractility.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-01008-y
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Myeloperoxidase is a critical mediator of anthracycline-induced cardiomyopathy.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-01006-0
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Cardiac fibroblast GSK-3α aggravates ischemic cardiac injury by promoting fibrosis, inflammation, and impairing angiogenesis.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-01005-1
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Identification of the specific molecular and functional signatures of pre-beta-HDL: relevance to cardiovascular disease.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-01004-2
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Preventing mitochondrial reverse electron transport as a strategy for cardioprotection.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-01002-4
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Perspective: mitochondrial STAT3 in cardioprotection.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-01003-3
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Rivaroxaban attenuates neutrophil maturation in the bone marrow niche.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-01001-5
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Matrix metalloproteinase-2 proteolyzes mitofusin-2 and impairs mitochondrial function during myocardial ischemia–reperfusion injury.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-00999-y
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The heterocellular heart: identities, interactions, and implications for cardiology.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-01000-6
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Endothelial ILK induces cardioprotection by preventing coronary microvascular dysfunction and endothelial-to-mesenchymal transition.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-00997-0
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A tale of pigs, beta-blockers and genetic variants.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-00998-z
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Three-vessel coronary infusion of cardiosphere-derived cells for the treatment of heart failure with preserved ejection fraction in a pre-clinical pig model.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-00995-2
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The RISK pathway leading to mitochondria and cardioprotection: how everything started.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-00992-5
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SMYD1a protects the heart from ischemic injury by regulating OPA1-mediated cristae remodeling and supercomplex formation.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-00991-6
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In-ovo echocardiography for application in cardiovascular research.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-00989-0
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Matrix metalloproteinases in coronary artery disease and myocardial infarction.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-00987-2
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Multilineage contribution of CD34<sup>+</sup> cells in cardiac remodeling after ischemia/reperfusion injury.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-00981-8
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Sexual dimorphism in bidirectional SR-mitochondria crosstalk in ventricular cardiomyocytes.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-00988-1
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Electrophysiological and calcium-handling development during long-term culture of human-induced pluripotent stem cell-derived cardiomyocytes.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-022-00973-0
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A cardiologist’s guide to machine learning in cardiovascular disease prognosis prediction.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-00982-7
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Indirect epigenetic testing identifies a diagnostic signature of cardiomyocyte DNA methylation in heart failure.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-022-00954-3
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Toll-like receptor-3 contributes to the development of aortic valve stenosis.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-00980-9
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The endothelial-enriched lncRNA LINC00607 mediates angiogenic function.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-00978-3
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Monoamine oxidase A-dependent ROS formation modulates human cardiomyocyte differentiation through AKT and WNT activation.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-00977-4
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Reduction in mitochondrial ROS improves oxidative phosphorylation and provides resilience to coronary endothelium in non-reperfused myocardial infarction.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-022-00976-x
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Anti-angiogenic effect of exo-LncRNA TUG1 in myocardial infarction and modulation by remote ischemic conditioning.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-022-00975-y
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Imatinib attenuates reperfusion injury in a rat model of acute myocardial infarction.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-022-00974-z
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