Works matching IS 03008428 AND DT 2017 AND VI 112 AND IP 4
Results: 16
Exogenous GDF11 induces cardiac and skeletal muscle dysfunction and wasting.
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- Basic Research in Cardiology, 2017, v. 112, n. 4, p. 1, doi. 10.1007/s00395-017-0639-9
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- Article
Erratum to: Changes of myocardial gene expression and protein composition in patients with dilated cardiomyopathy after immunoadsorption with subsequent immunoglobulin substitution.
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- Basic Research in Cardiology, 2017, v. 112, n. 4, p. 1, doi. 10.1007/s00395-017-0627-0
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Erratum to: Impairment of pH gradient and membrane potential mediates redox dysfunction in the mitochondria of the post-ischemic heart.
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- Basic Research in Cardiology, 2017, v. 112, n. 4, p. 1, doi. 10.1007/s00395-017-0632-3
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Differential regulation of protein phosphatase 1 (PP1) isoforms in human heart failure and atrial fibrillation.
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- Basic Research in Cardiology, 2017, v. 112, n. 4, p. 1, doi. 10.1007/s00395-017-0635-0
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Alignment of inducible vascular progenitor cells on a micro-bundle scaffold improves cardiac repair following myocardial infarction.
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- Basic Research in Cardiology, 2017, v. 112, n. 4, p. 1, doi. 10.1007/s00395-017-0631-4
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- Article
Sema3A promotes the resolution of cardiac inflammation after myocardial infarction.
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- Basic Research in Cardiology, 2017, v. 112, n. 4, p. 1, doi. 10.1007/s00395-017-0630-5
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Deleterious acute and chronic effects of bradycardic right ventricular apex pacing: consequences for arrhythmic outcome.
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- Basic Research in Cardiology, 2017, v. 112, n. 4, p. 1, doi. 10.1007/s00395-017-0636-z
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Dissecting the role of myeloid and mesenchymal fibroblasts in age-dependent cardiac fibrosis.
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- Basic Research in Cardiology, 2017, v. 112, n. 4, p. 1, doi. 10.1007/s00395-017-0623-4
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The crucial role of activin A/ALK4 pathway in the pathogenesis of Ang-II-induced atrial fibrosis and vulnerability to atrial fibrillation.
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- Basic Research in Cardiology, 2017, v. 112, n. 4, p. 1, doi. 10.1007/s00395-017-0634-1
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- Article
Impairment of pH gradient and membrane potential mediates redox dysfunction in the mitochondria of the post-ischemic heart.
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- Basic Research in Cardiology, 2017, v. 112, n. 4, p. 1, doi. 10.1007/s00395-017-0626-1
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- Article
Reduction of SR Ca leak and arrhythmogenic cellular correlates by SMP-114, a novel CaMKII inhibitor with oral bioavailability.
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- Basic Research in Cardiology, 2017, v. 112, n. 4, p. 1, doi. 10.1007/s00395-017-0637-y
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The role of spatial organization of Ca release sites in the generation of arrhythmogenic diastolic Ca release in myocytes from failing hearts.
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- Basic Research in Cardiology, 2017, v. 112, n. 4, p. 1, doi. 10.1007/s00395-017-0633-2
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Expression and regulation of type 2A protein phosphatases and alpha4 signalling in cardiac health and hypertrophy.
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- Basic Research in Cardiology, 2017, v. 112, n. 4, p. 1, doi. 10.1007/s00395-017-0625-2
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- Article
Splenic Ly6C monocytes contribute to adverse late post-ischemic left ventricular remodeling in heme oxygenase-1 deficient mice.
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- Basic Research in Cardiology, 2017, v. 112, n. 4, p. 1, doi. 10.1007/s00395-017-0629-y
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Exercise-induced circulating extracellular vesicles protect against cardiac ischemia-reperfusion injury.
- Published in:
- Basic Research in Cardiology, 2017, v. 112, n. 4, p. 1, doi. 10.1007/s00395-017-0628-z
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- Article
Fast therapeutic hypothermia prevents post-cardiac arrest syndrome through cyclophilin D-mediated mitochondrial permeability transition inhibition.
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- Basic Research in Cardiology, 2017, v. 112, n. 4, p. 1, doi. 10.1007/s00395-017-0624-3
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- Article