Works matching IS 03008428 AND DT 2016 AND VI 111 AND IP 2
Results: 15
Myocardial mechanical dysfunction following endotoxemia: role of changes in energy substrate metabolism.
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- Basic Research in Cardiology, 2016, v. 111, n. 2, p. 1, doi. 10.1007/s00395-016-0544-7
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- Article
δ-Opioid receptor (DOR) signaling and reactive oxygen species (ROS) mediate intermittent hypoxia induced protection of canine myocardium.
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- Basic Research in Cardiology, 2016, v. 111, n. 2, p. 1, doi. 10.1007/s00395-016-0538-5
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- Article
Studies on the role of apoptosis after transient myocardial ischemia: genetic deletion of the executioner caspases-3 and -7 does not limit infarct size and ventricular remodeling.
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- Basic Research in Cardiology, 2016, v. 111, n. 2, p. 1, doi. 10.1007/s00395-016-0537-6
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- Article
Dual-specificity phosphatase 14 protects the heart from aortic banding-induced cardiac hypertrophy and dysfunction through inactivation of TAK1-P38MAPK/-JNK1/2 signaling pathway.
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- Basic Research in Cardiology, 2016, v. 111, n. 2, p. 1, doi. 10.1007/s00395-016-0536-7
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- Article
Cells and extracellular matrix interplay in cardiac valve disease: because age matters.
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- Basic Research in Cardiology, 2016, v. 111, n. 2, p. 1, doi. 10.1007/s00395-016-0534-9
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Cardiac expression of the CREM repressor isoform CREM-IbΔCX in mice leads to arrhythmogenic alterations in ventricular cardiomyocytes.
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- Basic Research in Cardiology, 2016, v. 111, n. 2, p. 15, doi. 10.1007/s00395-016-0532-y
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- Article
SIRT6 protects cardiomyocytes against ischemia/reperfusion injury by augmenting FoxO3α-dependent antioxidant defense mechanisms.
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- Basic Research in Cardiology, 2016, v. 111, n. 2, p. N.PAG, doi. 10.1007/s00395-016-0531-z
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- Article
N-myc downstream-regulated gene 4, up-regulated by tumor necrosis factor-α and nuclear factor kappa B, aggravates cardiac ischemia/reperfusion injury by inhibiting reperfusion injury salvage kinase pathway.
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- Basic Research in Cardiology, 2016, v. 111, n. 2, p. 1, doi. 10.1007/s00395-015-0519-0
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Differential regulation of TRPV1 channels by H<sub>2</sub>O<sub>2</sub>: implications for diabetic microvascular dysfunction.
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- Basic Research in Cardiology, 2016, v. 111, n. 2, p. 1, doi. 10.1007/s00395-016-0539-4
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- Article
Atheroprotection through SYK inhibition fails in established disease when local macrophage proliferation dominates lesion progression.
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- Basic Research in Cardiology, 2016, v. 111, n. 2, p. 1, doi. 10.1007/s00395-016-0535-8
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- Article
Inhibition of ceramide de novo synthesis as a postischemic strategy to reduce myocardial reperfusion injury.
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- Basic Research in Cardiology, 2016, v. 111, n. 2, p. 1, doi. 10.1007/s00395-016-0533-x
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- Article
Human iPS cell model of type 3 long QT syndrome recapitulates drug-based phenotype correction.
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- Basic Research in Cardiology, 2016, v. 111, n. 2, p. 14, doi. 10.1007/s00395-016-0530-0
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- Article
Nitric oxide treatments as adjuncts to reperfusion in acute myocardial infarction: a systematic review of experimental and clinical studies.
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- Basic Research in Cardiology, 2016, v. 111, n. 2, p. 23, doi. 10.1007/s00395-016-0540-y
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- Article
Endothelial actions of atrial natriuretic peptide prevent pulmonary hypertension in mice.
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- Basic Research in Cardiology, 2016, v. 111, n. 2, p. 22, doi. 10.1007/s00395-016-0541-x
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- Article
The remote ischemic preconditioning algorithm: effect of number of cycles, cycle duration and effector organ mass on efficacy of protection.
- Published in:
- Basic Research in Cardiology, 2016, v. 111, n. 2, p. 1, doi. 10.1007/s00395-016-0529-6
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- Article