Works matching IS 03008428 AND DT 2011 AND VI 106 AND IP 6
Results: 37
Chronic exercise modulates RAS components and improves balance between pro- and anti-inflammatory cytokines in the brain of SHR.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1069, doi. 10.1007/s00395-011-0231-7
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G-CSF therapy reduces myocardial repolarization reserve in the presence of increased arteriogenesis, angiogenesis and connexin 43 expression in an experimental model of pacing-induced heart failure.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 995, doi. 10.1007/s00395-011-0230-8
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Cyclic nucleotide phosphodiesterase 1A: a key regulator of cardiac fibroblast activation and extracellular matrix remodeling in the heart.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1023, doi. 10.1007/s00395-011-0228-2
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Differential effects of heart rate reduction with ivabradine in two models of endothelial dysfunction and oxidative stress.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1147, doi. 10.1007/s00395-011-0227-3
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Conditional transgenic expression of TIR-domain-containing adaptor-inducing interferon-β (TRIF) in the adult mouse heart is protective in acute viral myocarditis.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1159, doi. 10.1007/s00395-011-0226-4
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The role of mitochondrial permeability transition in reperfusion-induced cardiomyocyte death depends on the duration of ischemia.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1259, doi. 10.1007/s00395-011-0225-5
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Estradiol inhibits hyaluronic acid synthase 1 expression in human vascular smooth muscle cells.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1099, doi. 10.1007/s00395-011-0217-5
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The potential effects of anti-diabetic medications on myocardial ischemia–reperfusion injury.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 925, doi. 10.1007/s00395-011-0216-6
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A novel role for mitochondrial sphingosine-1-phosphate produced by sphingosine kinase-2 in PTP-mediated cell survival during cardioprotection.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1341, doi. 10.1007/s00395-011-0223-7
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Presence of endothelial colony-forming cells is associated with reduced microvascular obstruction limiting infarct size and left ventricular remodelling in patients with acute myocardial infarction.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1397, doi. 10.1007/s00395-011-0220-x
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EphB signaling inhibits gap junctional intercellular communication and synchronized contraction in cultured cardiomyocytes.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1057, doi. 10.1007/s00395-011-0219-3
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NF-κB in the paraventricular nucleus modulates neurotransmitters and contributes to sympathoexcitation in heart failure.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1087, doi. 10.1007/s00395-011-0215-7
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Chronic akt activation accentuates aging-induced cardiac hypertrophy and myocardial contractile dysfunction: role of autophagy.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1173, doi. 10.1007/s00395-011-0222-8
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Mesenchymal stromal cells mediate a switch to alternatively activated monocytes/macrophages after acute myocardial infarction.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1299, doi. 10.1007/s00395-011-0221-9
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Determination of the myocardial area at risk with pre-versus post-reperfusion imaging techniques in the pig model.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1247, doi. 10.1007/s00395-011-0214-8
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Mitochondrial biogenesis and PGC-1α deacetylation by chronic treadmill exercise: differential response in cardiac and skeletal muscle.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1221, doi. 10.1007/s00395-011-0213-9
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Interferon-gamma induced adipose tissue inflammation is linked to endothelial dysfunction in type 2 diabetic mice.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1135, doi. 10.1007/s00395-011-0212-x
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Carbon monoxide exposure enhances arrhythmia after cardiac stress: involvement of oxidative stress.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1235, doi. 10.1007/s00395-011-0211-y
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RISK and SAFE signaling pathway interactions in remote limb ischemic perconditioning in combination with local ischemic postconditioning.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1329, doi. 10.1007/s00395-011-0210-z
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Failure to recapture cardioprotection with high-dose atorvastatin in coronary artery bypass surgery: a randomised controlled trial.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1387, doi. 10.1007/s00395-011-0209-5
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Gene transfer as a strategy to achieve permanent cardioprotection I: rAAV-mediated gene therapy with inducible nitric oxide synthase limits infarct size 1 year later without adverse functional consequences.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1355, doi. 10.1007/s00395-011-0207-7
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Electromechanical effects of the direct renin inhibitor (aliskiren) on the pulmonary vein and atrium.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 979, doi. 10.1007/s00395-011-0206-8
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Class A scavenger receptor attenuates myocardial infarction-induced cardiomyocyte necrosis through suppressing M1 macrophage subset polarization.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1311, doi. 10.1007/s00395-011-0204-x
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Release of atrial natriuretic peptide precedes shedding of the endothelial glycocalyx equally in patients undergoing on- and off-pump coronary artery bypass surgery.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1111, doi. 10.1007/s00395-011-0203-y
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Intracellular [Na<sup>+</sup>] modulates synergy between Na<sup>+</sup>/Ca<sup>2+</sup> exchanger and L-type Ca<sup>2+</sup> current in cardiac excitation–contraction coupling during action potentials.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 967, doi. 10.1007/s00395-011-0202-z
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Coronary arterioles in type 2 diabetic (db/db) mice undergo a distinct pattern of remodeling associated with decreased vessel stiffness.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1123, doi. 10.1007/s00395-011-0201-0
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Therapeutic neovascularization for coronary disease: current state and future prospects.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 897, doi. 10.1007/s00395-011-0200-1
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Burn-induced apoptosis of cardiomyocytes is survivin dependent and regulated by PI3K/Akt, p38 MAPK and ERK pathways.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1207, doi. 10.1007/s00395-011-0199-3
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Circulating microparticles: new insights into the biochemical basis of microparticle release and activity.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 911, doi. 10.1007/s00395-011-0198-4
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Tumor necrosis factor receptor 2 signaling limits β-adrenergic receptor-mediated cardiac hypertrophy in vivo.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1193, doi. 10.1007/s00395-011-0196-6
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Secretome of apoptotic peripheral blood cells (APOSEC) confers cytoprotection to cardiomyocytes and inhibits tissue remodelling after acute myocardial infarction: a preclinical study.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1283, doi. 10.1007/s00395-011-0224-6
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K201 (JTV-519) alters the spatiotemporal properties of diastolic Ca<sup>2+</sup> release and the associated diastolic contraction during β-adrenergic stimulation in rat ventricular cardiomyocytes.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1009, doi. 10.1007/s00395-011-0218-4
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Perfusion defect size predicts engraftment but not early retention of intra-myocardially injected cardiosphere-derived cells after acute myocardial infarction.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1379, doi. 10.1007/s00395-011-0197-5
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Remodeling of inward rectifying K currents in rat atrial myocytes by overexpression of A-adenosine receptors.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 953, doi. 10.1007/s00395-011-0193-9
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Unequal allelic expression of wild-type and mutated β-myosin in familial hypertrophic cardiomyopathy.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1041, doi. 10.1007/s00395-011-0205-9
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Gene transfer as a strategy to achieve permanent cardioprotection II: rAAV-mediated gene therapy with heme oxygenase-1 limits infarct size 1 year later without adverse functional consequences.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1367, doi. 10.1007/s00395-011-0208-6
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Left ventricular remodeling in swine after myocardial infarction: a transcriptional genomics approach.
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- Basic Research in Cardiology, 2011, v. 106, n. 6, p. 1269, doi. 10.1007/s00395-011-0229-1
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