Works matching IS 03008428 AND DT 2006 AND VI 101 AND IP 2
Results: 11
Post–conditioning reduces infarct size in the isolated rat heart: Role of coronary flow and pressure and the nitric oxide/cGMP pathway.
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- Basic Research in Cardiology, 2006, v. 101, n. 2, p. 168, doi. 10.1007/s00395-005-0543-6
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- Article
Glycoxidation of low–density lipoprotein promotes multiple apoptotic pathways and NF kB activation in human coronary cells.
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- Basic Research in Cardiology, 2006, v. 101, n. 2, p. 101, doi. 10.1007/s00395-005-0560-5
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- Article
Left ventricular remodeling after myocardial infarction in mice with targeted deletion of the NADPH oxidase subunit gp91<sup>PHOX</sup>.
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- Basic Research in Cardiology, 2006, v. 101, n. 2, p. 127, doi. 10.1007/s00395-005-0568-x
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- Article
Adenylyl cyclase type V deletion increases basal left ventricular function and reduces left ventricular contractile responsiveness to β–adrenergic stimulation.
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- Basic Research in Cardiology, 2006, v. 101, n. 2, p. 117, doi. 10.1007/s00395-005-0559-y
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- Article
Myocardial preconditioning and remote renal preconditioning.
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- Basic Research in Cardiology, 2006, v. 101, n. 2, p. 149, doi. 10.1007/s00395-005-0565-0
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- Article
Localizing extracellular signal–regulated kinase (ERK) in pharmacological preconditioning's trigger pathway.
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- Basic Research in Cardiology, 2006, v. 101, n. 2, p. 159, doi. 10.1007/s00395-005-0566-z
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- Article
Effect of sternotomy and extracorporeal circulation on pulmonary neutrophil kinetics in pigs.
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- Basic Research in Cardiology, 2006, v. 101, n. 2, p. 133, doi. 10.1007/s00395-005-0579-7
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- Article
A phase of increased ST elevation during coronary occlusion following ischemic preconditioning.
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- Basic Research in Cardiology, 2006, v. 101, n. 2, p. 140, doi. 10.1007/s00395-005-0574-z
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- Article
The antiatherogenic and antiinflammatory effect of HDL–associated lysosphingolipids operates via Akt ➞NF–kappaB signalling pathways in human vascular endothelial cells.
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- Basic Research in Cardiology, 2006, v. 101, n. 2, p. 109, doi. 10.1007/s00395-005-0582-z
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- Article
Post–conditioning induced cardioprotection requires signaling through a redox–sensitive mechanism, mitochondrial ATP–sensitive K<sup>+</sup> channel and protein kinase C activation.
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- Basic Research in Cardiology, 2006, v. 101, n. 2, p. 180, doi. 10.1007/s00395-006-0584-5
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- Article
A really radical observation.
- Published in:
- Basic Research in Cardiology, 2006, v. 101, n. 2, p. 190, doi. 10.1007/s00395-006-0586-3
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- Article