Works matching IS 02646021 AND DT 2015 AND VI 471 AND IP 1
Results: 11
Differential reduction in cardiac and liver monolysocardiolipin acyltransferase-1 and reduction in cardiac and liver tetralinoleoyl-cardiolipin in the α-subunit of trifunctional protein heterozygous knockout mice.
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- Biochemical Journal, 2015, v. 471, n. 1, p. 123, doi. 10.1042/BJ20150648
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- Article
Calcium modulation of exocytosis-linked plasma membrane potential oscillations in INS-1 832/13 cells.
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- Biochemical Journal, 2015, v. 471, n. 1, p. 111, doi. 10.1042/BJ20150616
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- Article
Unliganded EphA3 dimerization promoted by the SAM domain.
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- Biochemical Journal, 2015, v. 471, n. 1, p. 101, doi. 10.1042/BJ20150433
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- Article
Dengue NS3, an RNAi suppressor, modulates the human miRNA pathways through its interacting partner.
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- Biochemical Journal, 2015, v. 471, n. 1, p. 89, doi. 10.1042/BJ20150445
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- Article
A non-canonical ESCRT pathway, including histidine domain phosphotyrosine phosphatase (HD-PTP), is used for down-regulation of virally ubiquitinated MHC class I.
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- Biochemical Journal, 2015, v. 471, n. 1, p. 79, doi. 10.1042/BJ20150336
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- Article
c-Abl-mediated tyrosine phosphorylation of JunB is required for Adriamycin-induced expression of p21.
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- Biochemical Journal, 2015, v. 471, n. 1, p. 67, doi. 10.1042/BJ20150372
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- Article
Inhibition of HIV-1 gp41 expression with hammerhead ribozymes.
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- Biochemical Journal, 2015, v. 471, n. 1, p. 53, doi. 10.1042/BJ20150398
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- Article
Akt kinase C-terminal modifications control activation loop dephosphorylation and enhance insulin response.
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- Biochemical Journal, 2015, v. 471, n. 1, p. 37, doi. 10.1042/BJ20150325
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- Article
DNA cytosine and methylcytosine deamination by APOBEC3B: enhancing methylcytosine deamination by engineering APOBEC3B.
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- Biochemical Journal, 2015, v. 471, n. 1, p. 25, doi. 10.1042/BJ20150382
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- Article
The nucleoid-associated protein HU enhances 8-oxoguanine base excision by the formamidopyrimidine-DNA glycosylase.
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- Biochemical Journal, 2015, v. 471, n. 1, p. 13, doi. 10.1042/BJ20150387
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- Article
How cancer cells hijack DNA double-strand break repair pathways to gain genomic instability.
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- Biochemical Journal, 2015, v. 471, n. 1, p. 1, doi. 10.1042/BJ20150582
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- Article