Works matching IS 00778923 AND DT 2006 AND VI 1079 AND IP 1
Results: 61
TEDDY–The Environmental Determinants of Diabetes in the Young: An Observational Clinical Trial.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 320, doi. 10.1196/annals.1375.049
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The Type 1 Diabetes Genetics Consortium.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 1, doi. 10.1196/annals.1375.001
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A Mini Meta-Analysis of Studies on CD4<sup>+</sup>CD25<sup>+</sup> T cells in Human Type 1 Diabetes.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 9, doi. 10.1196/annals.1375.002
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HLA Class I Epitope Discovery in Type 1 Diabetes.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 19, doi. 10.1196/annals.1375.003
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Genetics of Fulminant Type 1 Diabetes.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 24, doi. 10.1196/annals.1375.004
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Why Is Type 1 Diabetes Uncommon in Asia?
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 31, doi. 10.1196/annals.1375.005
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Association of SUMO4, as a Candidate Gene for IDDM5, with Susceptibility to Type 1 Diabetes in Asian Populations.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 41, doi. 10.1196/annals.1375.006
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The Gene for Human IL-21 and Genetic Susceptibility to Type 1 Diabetes in the Japanese.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 47, doi. 10.1196/annals.1375.007
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Genetics of Type 1 Diabetes: Similarities and Differences between Asian and Caucasian Populations.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 51, doi. 10.1196/annals.1375.008
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Immunopathological and Genetic Features in Slowly Progressive Insulin-Dependent Diabetes Mellitus and Latent Autoimmune Diabetes in Adults.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 60, doi. 10.1196/annals.1375.009
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Genes Influencing Innate and Acquired Immunity in Type 1 Diabetes and Latent Autoimmune Diabetes in Adults.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 67, doi. 10.1196/annals.1375.010
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Progression of Autoimmune Diabetes.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 81, doi. 10.1196/annals.1375.011
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Immunomodulation for the Prevention of SPIDDM and LADA.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 90, doi. 10.1196/annals.1375.012
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Heightened Interferon-α/β Response Causes Myeloid Cell Dysfunction and Promotes T1D Pathogenesis in NOD Mice.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 99, doi. 10.1196/annals.1375.013
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Immunolocalization of Monocyte Chemoattractant Protein-1 in Islets of NOD Mice during Cyclophosphamide Administration.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 103, doi. 10.1196/annals.1375.014
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Young NOD Mice Show Increased Diabetes Sensitivity to Low Doses of Streptozotocin.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 109, doi. 10.1196/annals.1375.015
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Contribution of Class III MHC to Susceptibility to Type 1 Diabetes in the NOD Mouse.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 114, doi. 10.1196/annals.1375.016
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MHC-Linked Susceptibility to Type 1 Diabetes in the NOD Mouse.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 118, doi. 10.1196/annals.1375.017
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Long-Term Prevention of Diabetes and Marked Suppression of Insulin Autoantibodies and Insulitis in Mice Lacking Native Insulin B9–23 Sequence.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 122, doi. 10.1196/annals.1375.018
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Eicosanoid Imbalance in the NOD Mouse Is Related to a Dysregulation in Soluble Epoxide Hydrolase and 15-PGDH Expression.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 130, doi. 10.1196/annals.1375.019
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Pancreatic Autoimmunity Induction with Insulin B:9–23 Peptide and Viral Mimics in the NZB Mouse.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 135, doi. 10.1196/annals.1375.020
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Viruses Cause Type 1 Diabetes in Animals.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 138, doi. 10.1196/annals.1375.021
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Characterization of PAF-AH Ib1 in NOD Mice.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 147, doi. 10.1196/annals.1375.022
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Peptide-Pulsed Immature Dendritic Cells Reduce Response to β Cell Target Antigens and Protect NOD Recipients from Type I Diabetes.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 153, doi. 10.1196/annals.1375.023
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New Members of the Interleukin-12 Family of Cytokines: IL-23 and IL-27 Modulate Autoimmune Diabetes.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 157, doi. 10.1196/annals.1375.024
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Glutamic Acid Decarboxylase-Specific CD4<sup>+</sup> Regulatory T Cells.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 161, doi. 10.1196/annals.1375.025
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Metabolism Genes Are among the Differentially Expressed Ones Observed in Lymphomononuclear Cells of Recently Diagnosed Type 1 Diabetes Mellitus Patients.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 171, doi. 10.1196/annals.1375.026
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In Vitro TNF-α and IL-6 Production by Adherent Peripheral Blood Mononuclear Cells Obtained from Type 1 and Type 2 Diabetic Patients Evaluated according to the Metabolic Control.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 177, doi. 10.1196/annals.1375.027
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T Cell Immunity to Glutamic Acid Decarboxylase in Fulminant Type 1 Diabetes without Significant Elevation of Serum Amylase.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 181, doi. 10.1196/annals.1375.028
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Expression Levels of CXC Chemokine Receptors 3 Are Associated with Clinical Phenotype of Type 1 Diabetes.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 186, doi. 10.1196/annals.1375.029
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HLA Class I Epitope Discovery in Type 1 Diabetes.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 190, doi. 10.1196/annals.1375.030
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Upregulation of Foxp3 Expression in Mouse and Human Treg Is IL-2/STAT5 Dependent.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 198, doi. 10.1196/annals.1375.031
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Normal T Cell Development in the Absence of Thymic Insulin Expression.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 205, doi. 10.1196/annals.1375.032
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Antigenic Determinants to GAD Autoantibodies in Patients With Type 1 Diabetes With and Without Autoimmune Thyroid Disease.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 213, doi. 10.1196/annals.1375.033
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TSH Receptor Antibodies in Subjects with Type 1 Diabetes Mellitus.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 220, doi. 10.1196/annals.1375.034
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Time-Resolved Fluorescence Imaging of Islet Cell Autoantibodies.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 226, doi. 10.1196/annals.1375.035
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MHC Class I Chain-Related Gene-A Is Associated with IA2 and IAA but Not GAD in Swedish Type 1 Diabetes Mellitus.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 229, doi. 10.1196/annals.1375.036
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Predominance of the Group A Killer Ig-Like Receptor Haplotypes in Korean Patients With T1D.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 240, doi. 10.1196/annals.1375.037
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Frequency of CTLA-4 Gene CT60 Polymorphism May Not Be Affected by Vitamin D Receptor Gene Bsm I Polymorphism or HLA DR9 in Autoimmune-Related Type 1 Diabetes in the Japanese.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 251, doi. 10.1196/annals.1375.038
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Genetic and Functional Evidence Supporting SUMO4 as a Type 1 Diabetes Susceptibility Gene.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 257, doi. 10.1196/annals.1375.039
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No Association of TLR2 and TLR4 Polymorphisms with Type I Diabetes Mellitus in the Basque Population.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 268, doi. 10.1196/annals.1375.040
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Association of SUMO4 M55V Polymorphism with Autoimmune Diabetes in Latvian Patients.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 273, doi. 10.1196/annals.1375.041
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A Second Component of HLA-Linked Susceptibility to Type 1 Diabetes Maps to Class I Region.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 278, doi. 10.1196/annals.1375.042
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Molecular Scanning of the Gene for Programmed Cell Death-1 (PDCD-1) as a Candidate for Type 1 Diabetes Susceptibility.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 285, doi. 10.1196/annals.1375.043
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Genetic Determinants of Type 1 Diabetes Across Populations.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 289, doi. 10.1196/annals.1375.044
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TNFa-e Microsatellite, HLA-DRB1 and -DQB1 Alleles and Haplotypes in Brazilian Patients Presenting Recently Diagnosed Type 1 Diabetes Mellitus.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 300, doi. 10.1196/annals.1375.045
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Is HLA Class II Profile Relevant for the Study of Large-Scale Differentially Expressed Genes in Type 1 Diabetes Mellitus Patients?
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 305, doi. 10.1196/annals.1375.046
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Feasibility of a Type 1 Diabetes Primary Prevention Trial Using 2000 IU Vitamin D3 in Infants from the General Population with Increased HLA-Associated Risk.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 310, doi. 10.1196/annals.1375.047
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Interleukin-10 Plasmid Construction and Delivery for the Prevention of Type 1 Diabetes.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 313, doi. 10.1196/annals.1375.048
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Protection From Type 1 Diabetes by Vitamin D Receptor Haplotypes.
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- Annals of the New York Academy of Sciences, 2006, v. 1079, n. 1, p. 327, doi. 10.1196/annals.1375.050
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