Works matching IS 00778923 AND DT 2000 AND VI 908 AND IP 1
Results: 42
Fibroblast Responses to Exogenous and Autocrine Growth Factors Relevant to Tissue Repair: The Effect of Aging.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 155, doi. 10.1111/j.1749-6632.2000.tb06644.x
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Poly(ADP-Ribosyl)ation, Genomic Instability, and Longevity.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 126, doi. 10.1111/j.1749-6632.2000.tb06641.x
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Role of Oxidative Stress in Telomere Length Regulation and Replicative Senescence.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 99, doi. 10.1111/j.1749-6632.2000.tb06639.x
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Role of Mitochondrial DNA Mutations in Disease and Aging.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 199, doi. 10.1111/j.1749-6632.2000.tb06647.x
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Aging-Related Muscle Dysfunction: Failure of Adaptation to Oxidative Stress?
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 341, doi. 10.1111/j.1749-6632.2000.tb06669.x
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Molecular Gerontology: Bridging the Simple and the Complex.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 14, doi. 10.1111/j.1749-6632.2000.tb06631.x
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Biogerontology: The Next Step.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 282, doi. 10.1111/j.1749-6632.2000.tb06655.x
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Genetics of Human Aging: The Search for Genes Contributing to Human Longevity and Diseases of the Old.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 50, doi. 10.1111/j.1749-6632.2000.tb06635.x
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Telomere Length As a Marker of Oxidative Stress in Primary Human Fibroblast Cultures.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 327, doi. 10.1111/j.1749-6632.2000.tb06666.x
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Metabolic Control and Gene Dysregulation in Yeast Aging.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 21, doi. 10.1111/j.1749-6632.2000.tb06632.x
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Transgenic Mouse Models of Alzheimer's Disease.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 260, doi. 10.1111/j.1749-6632.2000.tb06653.x
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Protein Degradation by the Proteasome and Its Implications in Aging.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 143, doi. 10.1111/j.1749-6632.2000.tb06643.x
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A Theoretical Model for 'in machina' Experiments on Immunosenescence.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 344, doi. 10.1111/j.1749-6632.2000.tb06670.x
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Age-Related Changes in Irradiation-Induced Apoptosis and Expression of p21 and p53 in Crypt Stem Cells of Murine Intestine.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 315, doi. 10.1111/j.1749-6632.2000.tb06662.x
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A Novel in Vitro Model of Conditionally Immortalized Human Vascular Smooth Muscle Cells: A Tool for Aging Studies.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 321, doi. 10.1111/j.1749-6632.2000.tb06664.x
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Mitochondrial Oxidative Stress and Aging in the Filamentous Fungus Podospora anserina.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 31, doi. 10.1111/j.1749-6632.2000.tb06633.x
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Testing Evolutionary Theories of Aging.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 319, doi. 10.1111/j.1749-6632.2000.tb06663.x
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Genetics of Survival.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 64, doi. 10.1111/j.1749-6632.2000.tb06636.x
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Molecular Misreading: A New Type of Transcript Mutation in Gerontology.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 267, doi. 10.1111/j.1749-6632.2000.tb06654.x
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Replicative Senescence of Interleukin-2-Dependent Human T Lymphocytes: Flow Cytometric Characteristics of Phenotype Changes.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 310, doi. 10.1111/j.1749-6632.2000.tb06661.x
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Melanin Accumulation Accelerates Melanocyte Senescence by a Mechanism Involving p16<sup>INK4a</sup>/CDK4/pRB and E2F1.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 71, doi. 10.1111/j.1749-6632.2000.tb06637.x
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Nuclear-Mitochondrial Interactions Involved in Aging in Podospora anserina.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 291, doi. 10.1111/j.1749-6632.2000.tb06656.x
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Study of the H1 Linker Histone Variant, H1o, during the in Vitro Aging of Human Diploid Fibroblasts.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 336, doi. 10.1111/j.1749-6632.2000.tb06668.x
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Inflamm-aging: An Evolutionary Perspective on Immunosenescence.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 244, doi. 10.1111/j.1749-6632.2000.tb06651.x
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The CA1 Region of the Human Hippocampus Is a Hot Spot in Alzheimer's Disease.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 255, doi. 10.1111/j.1749-6632.2000.tb06652.x
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Inherited Frailty: ApoE Alleles Determine Survival after a Diagnosis of Heart Disease or Stroke at Ages 85+.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 295, doi. 10.1111/j.1749-6632.2000.tb06657.x
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Tissue Mitochondrial DNA Changes: A Stochastic System.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 226, doi. 10.1111/j.1749-6632.2000.tb06650.x
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Inherited Variability of the Mitochondrial Genome and Successful Aging in Humans.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 208, doi. 10.1111/j.1749-6632.2000.tb06648.x
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Stress-Induced Premature Senescence: Essence of Life, Evolution, Stress, and Aging.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 85, doi. 10.1111/j.1749-6632.2000.tb06638.x
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Mitochondrial Oxidative Stress: Physiologic Consequences and Potential for a Role in Aging.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 219, doi. 10.1111/j.1749-6632.2000.tb06649.x
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MtDNA Deletions in Aging and in Nonmitochondrial Pathologies.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 299, doi. 10.1111/j.1749-6632.2000.tb06658.x
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Preface.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. ix, doi. 10.1111/j.1749-6632.2000.tb06629.x
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The Werner Syndrome: A Model for the Study of Human Aging.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 167, doi. 10.1111/j.1749-6632.2000.tb06645.x
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Programmed Cell Death and Senescence in Skeletal Muscle Stem Cells.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 331, doi. 10.1111/j.1749-6632.2000.tb06667.x
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Transcriptome and Proteome Analysis in Human Senescent Fibroblasts and Fibroblasts Undergoing Premature Senescence Induced by Repeated Sublethal Stresses.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 302, doi. 10.1111/j.1749-6632.2000.tb06659.x
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Using Stress Resistance to Isolate Novel Longevity Mutations in Caenorhabditis elegans.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 324, doi. 10.1111/j.1749-6632.2000.tb06665.x
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Human Diploid Fibroblasts Display a Decreased Level of c-fos mRNA at 72 Hours after Exposure to Sublethal H<sub>2</sub>O<sub>2</sub> Stress.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 306, doi. 10.1111/j.1749-6632.2000.tb06660.x
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Molecular Genetic Mechanisms of Life Span Manipulation in Caenorhabditis elegans.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 40, doi. 10.1111/j.1749-6632.2000.tb06634.x
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Calorie Restriction and Age-Related Oxidative Stress.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 180, doi. 10.1111/j.1749-6632.2000.tb06646.x
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Replicative Senescence and Oxidant-Induced Premature Senescence: Beyond the Control of Cell Cycle Checkpoints.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 111, doi. 10.1111/j.1749-6632.2000.tb06640.x
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Aging and Longevity: A Paradigm of Complementation between Homeostatic Mechanisms and Genetic Control?
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 133, doi. 10.1111/j.1749-6632.2000.tb06642.x
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Some New Directions for Research on the Biology of Aging.
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- Annals of the New York Academy of Sciences, 2000, v. 908, n. 1, p. 1, doi. 10.1111/j.1749-6632.2000.tb06630.x
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