Works matching IS 00778923 AND DT 1999 AND VI 893 AND IP 1
Results: 61
Inhibition of the Neuronal Insulin Receptor Causes Alzheimer-like Disturbances in Oxidative/Energy Brain Metabolism and in Behavior in Adult Rats.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 301, doi. 10.1111/j.1749-6632.1999.tb07842.x
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Regulation of Mitochondrial Gene Expression in Differentiated PC12 Cells.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 341, doi. 10.1111/j.1749-6632.1999.tb07850.x
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Detection of Respiratory Chain Defects in Cultivated Skin Fibroblasts and Skeletal Muscle of Patients with Parkinson's Disease.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 426, doi. 10.1111/j.1749-6632.1999.tb07870.x
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Does Dopamine Contribute to Striatal Damage Caused by Impaired Mitochondrial Function?
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 345, doi. 10.1111/j.1749-6632.1999.tb07851.x
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Calcium Overload Triggers Rod Photoreceptor Apoptotic Cell Death in Chemical-Induced and Inherited Retinal Degenerations.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 282, doi. 10.1111/j.1749-6632.1999.tb07837.x
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Attenuation of Neuronal Death by NMDA and Oxygen-Glucose Deprivation in Cortical Neurons Maintained in High Glucose.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 396, doi. 10.1111/j.1749-6632.1999.tb07864.x
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Maturational Changes in Rabbit Brain Phosphocreatine and Creatine Kinase.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 309, doi. 10.1111/j.1749-6632.1999.tb07844.x
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Molecular Mechanisms of Free Radical Production and Protective Efficacies of Antioxidants in in Vitro Ischemia-Reperfusion.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 286, doi. 10.1111/j.1749-6632.1999.tb07838.x
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Mitochondrial Compartmentation at the Cellular Level: Astrocytes and Neurons.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 421, doi. 10.1111/j.1749-6632.1999.tb07869.x
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Ca<sup>2+</sup>-Mediated Mitochondrial Dysfunction and the Protective Effects of Bcl-2.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 19, doi. 10.1111/j.1749-6632.1999.tb07815.x
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Enhanced Acetate and Glucose Utilization during Graded Photic Stimulation: Neuronal-Glial Interactions in Vivo.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 279, doi. 10.1111/j.1749-6632.1999.tb07836.x
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Interactions of Chloromethyltetramethylrosamine (Mitotracker Orange™) with Isolated Mitochondria and Intact Cells.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 391, doi. 10.1111/j.1749-6632.1999.tb07863.x
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Evidence for Energy Failure following Irreversible Traumatic Brain Injury.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 337, doi. 10.1111/j.1749-6632.1999.tb07849.x
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Cellular and Molecular Mechanisms Underlying Perturbed Energy Metabolism and Neuronal Degeneration in Alzheimer's and Parkinson's Diseases.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 154, doi. 10.1111/j.1749-6632.1999.tb07824.x
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Glutamate Excitotoxicity and Neuronal Energy Metabolism.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 1, doi. 10.1111/j.1749-6632.1999.tb07813.x
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Use of Cytoplasmic Hybrid Cell Lines for Elucidating the Role of Mitochondrial Dysfunction in Alzheimer's Disease and Parkinson's Disease.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 176, doi. 10.1111/j.1749-6632.1999.tb07825.x
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Discordance between Traditional Pathologic and Energy Metabolic Changes in Very Early Alzheimer's Disease: Pathophysiological Implications.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 350, doi. 10.1111/j.1749-6632.1999.tb07852.x
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Preface.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. xi, doi. 10.1111/j.1749-6632.1999.tb07812.x
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Neuronal RNA Oxidation in Alzheimer's Disease and Down's Syndrome.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 362, doi. 10.1111/j.1749-6632.1999.tb07855.x
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Temporary Axonal Conduction Block and Axonal Loss in Inflammatory Neurological Disease: A Potential Role for Nitric Oxide?
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 304, doi. 10.1111/j.1749-6632.1999.tb07843.x
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Ganglioside GD3, the Mitochondrial Permeability Transition, and Apoptosis.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 321, doi. 10.1111/j.1749-6632.1999.tb07846.x
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Mitochondrial Membrane Potential and the Permeability Transition in Excitotoxicity.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 33, doi. 10.1111/j.1749-6632.1999.tb07816.x
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The Blood-Brain Barrier and Cerebrovascular Pathology in Alzheimer's Disease.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 113, doi. 10.1111/j.1749-6632.1999.tb07821.x
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The Use of Transgenic and Mutant Mice to Study Oxygen Free Radical Metabolism.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 95, doi. 10.1111/j.1749-6632.1999.tb07820.x
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Bcl-2 and p53: Role in Dopamine-Induced Apoptosis and Differentiation.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 372, doi. 10.1111/j.1749-6632.1999.tb07858.x
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ATPases of Synaptic Plasma Membranes and Vesicles from Rat Cerebral Cortex during Aging and Hypoxia.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 417, doi. 10.1111/j.1749-6632.1999.tb07868.x
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Neurotoxicity and Oxidative Damage of Beta Amyloid 1-42 versus Beta Amyloid 1-40 in the Mouse Cerebral Cortex.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 314, doi. 10.1111/j.1749-6632.1999.tb07845.x
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Interactions between Melatonin, Reactive Oxygen Species, and Nitric Oxide.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 325, doi. 10.1111/j.1749-6632.1999.tb07847.x
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Ca<sup>2+</sup>-Dependent Permeability Transition and Complex I Activity in Lymphoblast Mitochondria from Normal Individuals and Patients with Huntington's or Alzheimer's Disease.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 365, doi. 10.1111/j.1749-6632.1999.tb07856.x
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Mitochondria Mediate Nitric Oxide-Induced Cell Death.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 376, doi. 10.1111/j.1749-6632.1999.tb07859.x
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Monoamine Oxidase Inhibits Mitochondrial Respiration.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 273, doi. 10.1111/j.1749-6632.1999.tb07835.x
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Prevention of Neurodegeneration by a Neuroprotective Radical Scavenger.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 430, doi. 10.1111/j.1749-6632.1999.tb07871.x
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Effects of the Solvent 1,2,4-Trimethylcyclohexane on Respiratory Burst in Human Neutrophil Granulocytes: A Chemiluminescence and Electron Paramagnetic Resonance Spectrometry Study.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 358, doi. 10.1111/j.1749-6632.1999.tb07854.x
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Mitochondrial Porin, a Novel Target to Prevent Ischemia-Induced Neurodegeneration?
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 369, doi. 10.1111/j.1749-6632.1999.tb07857.x
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Polyglutamine Domain Proteins with Expanded Repeats Bind Neurofilament, Altering the Neurofilament Network.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 192, doi. 10.1111/j.1749-6632.1999.tb07826.x
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Functional Brain Imaging in the Resting State and during Activation in Alzheimer's Disease: Implications for Disease Mechanisms Involving Oxidative Phosphorylation.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 138, doi. 10.1111/j.1749-6632.1999.tb07823.x
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The α-Ketoglutarate Dehydrogenase Complex.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 61, doi. 10.1111/j.1749-6632.1999.tb07818.x
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Inhibition of α-Ketoglutarate Dehydrogenase due to H<sub>2</sub>O<sub>2</sub>-Induced Oxidative Stress in Nerve Terminals.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 412, doi. 10.1111/j.1749-6632.1999.tb07867.x
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Conference Summary: Mitochondria, Neurodegenerative Diseases, and Selective Neuronal Vulnerability.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 434, doi. 10.1111/j.1749-6632.1999.tb07872.x
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Effect of Oxidative Stress on DNA Damage and β-Amyloid Precursor Proteins in Lymphoblastoid Cell Lines from a Nigerian Population.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 331, doi. 10.1111/j.1749-6632.1999.tb07848.x
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Measurement of Oxidative DNA Damage in the Human p53 and pgk1 Gene at Nucleotide Resolution<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 382, doi. 10.1111/j.1749-6632.1999.tb07861.x
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Low Frequencies of Mitochondrial DNA Mutations Cause Cardiac Disease in the Mouse.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 353, doi. 10.1111/j.1749-6632.1999.tb07853.x
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A Disturbance in the Neuronal Insulin Receptor Signal Transduction in Sporadic Alzheimer's Disease.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 290, doi. 10.1111/j.1749-6632.1999.tb07839.x
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Signaling Events in NMDA Receptor-Induced Apoptosis in Cerebrocortical Cultures.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 261, doi. 10.1111/j.1749-6632.1999.tb07832.x
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The Glucose Paradox in Cerebral Ischemia: New Insights.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 386, doi. 10.1111/j.1749-6632.1999.tb07862.x
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Bioenergetics in Huntington's Disease.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 203, doi. 10.1111/j.1749-6632.1999.tb07827.x
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Induction of Apoptosis and Necrosis in Human Neuroblastoma Cells by Cholesterol Oxides.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 379, doi. 10.1111/j.1749-6632.1999.tb07860.x
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Apoptosis and Necrosis in Cerebrovascular Disease.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 243, doi. 10.1111/j.1749-6632.1999.tb07829.x
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Effects of Ebselen, a Glutathione Peroxidase Mimic, in Several Models of Mitochondrial Dysfunction.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 254, doi. 10.1111/j.1749-6632.1999.tb07830.x
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Modulation of Presenilin-1 Processing by Nitric Oxide during Apoptosis Induced by Serum Withdrawal and Glucose Deprivation.
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- Annals of the New York Academy of Sciences, 1999, v. 893, n. 1, p. 294, doi. 10.1111/j.1749-6632.1999.tb07840.x
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