Works matching IS 00778923 AND DT 1999 AND VI 874 AND IP 1
Results: 39
Nitric Oxide and the Vascular Endothelium in Myocardial Ischemia-Reperfusion Injury<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 354, doi. 10.1111/j.1749-6632.1999.tb09251.x
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Small Heat Shock Proteins and Protection against Injury.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 66, doi. 10.1111/j.1749-6632.1999.tb09225.x
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Interaction of Bradykinin with Angiotensin, Prostacyclin, and Nitric Oxide in Myocardial Preservation<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 286, doi. 10.1111/j.1749-6632.1999.tb09244.x
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Myocardial Response to Stress in Cardiac Hypertrophy and Heart Failure: Effect of Antihypertensive Drugs<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 125, doi. 10.1111/j.1749-6632.1999.tb09230.x
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Physical Exercise and Antioxidant Defenses in the Heart<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 169, doi. 10.1111/j.1749-6632.1999.tb09234.x
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The Ins and Outs of Caveolar Signaling: m2 Muscarinic Cholinergic Receptors and eNOS Activation versus Neuregulin and ErbB4 Signaling in Cardiac Myocytes<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 11, doi. 10.1111/j.1749-6632.1999.tb09220.x
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Cardiac Adaptation to Ischemia-Reperfusion Injury<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 83, doi. 10.1111/j.1749-6632.1999.tb09227.x
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The Role of the Myocardial Sodium-Hydrogen Exchanger in Mediating Ischemic and Reperfusion Injury: From Amiloride to Cariporide<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 326, doi. 10.1111/j.1749-6632.1999.tb09248.x
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Regulation of Cardiovascular Development and Physiology by Hypoxia-Inducible Factor 1<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 262, doi. 10.1111/j.1749-6632.1999.tb09241.x
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Amplification of Angiotensin II Signaling in Cardiac Myocytes by Adenovirus-Mediated Overexpression of the AT<sub>1</sub> Receptor<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 20, doi. 10.1111/j.1749-6632.1999.tb09221.x
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Role of K<sub>ATP</sub> Channel in Heat Shock and Pharmacological Preconditioning<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 211, doi. 10.1111/j.1749-6632.1999.tb09237.x
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Inhibition of Myocardial TNF-α Production by Heat Shock: A Potential Mechanism of Stress-Induced Cardioprotection against Postischemic Dysfunction<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 69, doi. 10.1111/j.1749-6632.1999.tb09226.x
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Pharmacologic Enhancement of Tolerance to Ischemic Cardiac Stress Using Monophosphoryl Lipid A: A Comparison with Antecedent Ischemia.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 222, doi. 10.1111/j.1749-6632.1999.tb09238.x
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Quantitative Analysis of Collagens and Fibronectin Expression in Human Right Ventricular Hypertrophy<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 278, doi. 10.1111/j.1749-6632.1999.tb09243.x
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Alternatives for Myocardial Protection: Adenosine-Enhanced Ischemic Preconditioning<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 295, doi. 10.1111/j.1749-6632.1999.tb09245.x
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Cellular Mechanisms of Infarct Size Reduction with Ischemic Preconditioning: Role of Calcium?
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 192, doi. 10.1111/j.1749-6632.1999.tb09236.x
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Oxidized LDL and Atherogenesis<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 134, doi. 10.1111/j.1749-6632.1999.tb09231.x
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Cardiac Hypertrophy: Signal Transduction, Transcriptional Adaptation, and Altered Growth Control.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 1, doi. 10.1111/j.1749-6632.1999.tb09219.x
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Apoptosis in Isolated Adult Cardiomyocytes Exposed to Adriamycin<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 156, doi. 10.1111/j.1749-6632.1999.tb09233.x
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Effect of Antioxidant Trace Elements on the Response of Cardiac Tissue to Oxidative Stress<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 138, doi. 10.1111/j.1749-6632.1999.tb09232.x
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New Insights into Cardioprotection by Ischemic Preconditioning and Other Forms of Stress<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 178, doi. 10.1111/j.1749-6632.1999.tb09235.x
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Mitochondrial ATP-Dependent Potassium Channels: Viable Candidate Effectors of Ischemic Preconditioning<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 27, doi. 10.1111/j.1749-6632.1999.tb09222.x
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Surgical Stress and the Heat Shock Response: In Vivo Models of Stress Conditioning.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 320, doi. 10.1111/j.1749-6632.1999.tb09247.x
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Stress Signal to Survival and Apoptosis<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 427, doi. 10.1111/j.1749-6632.1999.tb09256.x
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Preface.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. ix, doi. 10.1111/j.1749-6632.1999.tb09218.x
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Differential Regulation of Apoptosis by Ischemia-Reperfusion and Ischemic Adaptation<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 401, doi. 10.1111/j.1749-6632.1999.tb09254.x
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Apoptosis in Myocardial Ischemia-Reperfusion.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 412, doi. 10.1111/j.1749-6632.1999.tb09255.x
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The Molecular Mechanism of Cardiac Hypertrophy and Failure.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 38, doi. 10.1111/j.1749-6632.1999.tb09223.x
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Oxygen Free Radical Signaling in Ischemic Preconditioning<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 49, doi. 10.1111/j.1749-6632.1999.tb09224.x
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Regulation of Cardiac Sarcolemmal Na<sup>+</sup>/H<sup>+</sup> Exchanger Activity by Endogenous Ligands: Relevance to Ischemia<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 335, doi. 10.1111/j.1749-6632.1999.tb09249.x
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Optimal Myocardial Preconditioning in Humans<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 306, doi. 10.1111/j.1749-6632.1999.tb09246.x
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Na<sup>+</sup>/H<sup>+</sup> Exchangers: Molecular Diversity and Relevance to Heart.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 346, doi. 10.1111/j.1749-6632.1999.tb09250.x
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Nitric Oxide Prevents Myoglobin/ tert-Butyl Hydroperoxide-Induced Inhibition of Ca<sup>2+</sup> Transport in Skeletal and Cardiac Sarcoplasmic Reticulum.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 371, doi. 10.1111/j.1749-6632.1999.tb09252.x
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Role for NADPH/NADH Oxidase in the Modulation of Vascular Tone<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 386, doi. 10.1111/j.1749-6632.1999.tb09253.x
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Metabolic Changes in the Normal and Hypoxic Neonatal Myocardium.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 254, doi. 10.1111/j.1749-6632.1999.tb09240.x
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In Vitro Analysis of SERCA2 Gene Regulation in Hypertrophic Cardiomyocytes and Increasing Transfection Efficiency by Gene-Gun Biolistics<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 111, doi. 10.1111/j.1749-6632.1999.tb09229.x
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Adaptation to Chronic Hypoxia Confers Tolerance to Subsequent Myocardial Ischemia by Increased Nitric Oxide Production.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 236, doi. 10.1111/j.1749-6632.1999.tb09239.x
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Activity of Cytochrome c Oxidase in the Right and Left Ventricular Myocardium of Male and Female Rats Exposed To Intermittent High Altitude Hypoxia<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 269, doi. 10.1111/j.1749-6632.1999.tb09242.x
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Subcellular Remodeling and Heart Dysfunction in Cardiac Hypertrophy due to Pressure Overload<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1999, v. 874, n. 1, p. 100, doi. 10.1111/j.1749-6632.1999.tb09228.x
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