Works matching IS 00223751 AND DT 2015 AND VI 593 AND IP 6

Results: 16

Maximal acceleration of Ca2+ release refractoriness by β-adrenergic stimulation requires dual activation of kinases PKA and CaMKII in mouse ventricular myocytes.

Published in:

Journal of Physiology, 2015, v. 593, n. 6, p. 1495, doi. 10.1113/jphysiol.2014.278051

By:

Poláková, Eva;
Illaste, Ardo;
Niggli, Ernst;
Sobie, Eric A.

Publication type:

Article

Decreased inward rectifying K+ current and increased ryanodine receptor sensitivity synergistically contribute to sustained focal arrhythmia in the intact rabbit heart.

Published in:

Journal of Physiology, 2015, v. 593, n. 6, p. 1479, doi. 10.1113/jphysiol.2014.279638

By:

Myles, Rachel C.;
Wang, Lianguo;
Bers, Donald M.;
Ripplinger, Crystal M.

Publication type:

Article

Sodium and calcium regulation in cardiac myocytes: from molecules to heart failure and arrhythmia.

Published in:

2015

By:

Bers, Donald M.;
Chen‐Izu, Ye

Publication type:

Editorial

Na+ channel function, regulation, structure, trafficking and sequestration.

Published in:

Journal of Physiology, 2015, v. 593, n. 6, p. 1347, doi. 10.1113/jphysiol.2014.281428

By:

Chen‐Izu, Ye;
Shaw, Robin M.;
Pitt, Geoffrey S.;
Yarov‐Yarovoy, Vladimir;
Sack, Jon T.;
Abriel, Hugues;
Aldrich, Richard W.;
Belardinelli, Luiz;
Cannell, Mark B.;
Catterall, William A.;
Chazin, Walter J.;
Chiamvimonvat, Nipavan;
Deschenes, Isabelle;
Grandi, Eleonora;
Hund, Thomas J.;
Izu, Leighton T.;
Maier, Lars S.;
Maltsev, Victor A.;
Marionneau, Celine;
Mohler, Peter J.

Publication type:

Article

Palpitations, potassium and the pump.

Published in:

Journal of Physiology, 2015, v. 593, n. 6, p. 1387, doi. 10.1113/jphysiol.2014.285924

By:

Weiss, James N.

Publication type:

Article

Scn1b deletion leads to increased tetrodotoxin-sensitive sodium current, altered intracellular calcium homeostasis and arrhythmias in murine hearts.

Published in:

Journal of Physiology, 2015, v. 593, n. 6, p. 1389, doi. 10.1113/jphysiol.2014.277699

By:

Lin, Xianming;
O'Malley, Heather;
Chen, Chunling;
Auerbach, David;
Foster, Monique;
Shekhar, Akshay;
Zhang, Mingliang;
Coetzee, William;
Jalife, José;
Fishman, Glenn I.;
Isom, Lori;
Delmar, Mario

Publication type:

Article

A computational modelling approach combined with cellular electrophysiology data provides insights into the therapeutic benefit of targeting the late Na+ current.

Published in:

Journal of Physiology, 2015, v. 593, n. 6, p. 1429, doi. 10.1113/jphysiol.2014.279554

By:

Yang, Pei‐Chi;
Song, Yejia;
Giles, Wayne R.;
Horvath, Balazs;
Chen‐Izu, Ye;
Belardinelli, Luiz;
Rajamani, Sridharan;
Clancy, Colleen E.

Publication type:

Article

Contribution of sodium channel neuronal isoform Nav1.1 to late sodium current in ventricular myocytes from failing hearts.

Published in:

Journal of Physiology, 2015, v. 593, n. 6, p. 1409, doi. 10.1113/jphysiol.2014.278259

By:

Mishra, Sudhish;
Reznikov, Vitaliy;
Maltsev, Victor A.;
Undrovinas, Nidas A.;
Sabbah, Hani N.;
Undrovinas, Albertas

Publication type:

Article

Hypokalaemia induces Ca2+ overload and Ca2+ waves in ventricular myocytes by reducing Na+,K+-ATPase α2 activity.

Published in:

Journal of Physiology, 2015, v. 593, n. 6, p. 1509, doi. 10.1113/jphysiol.2014.279893

By:

Aronsen, J. M.;
Skogestad, J.;
Lewalle, A.;
Louch, W. E.;
Hougen, K.;
Stokke, M. K.;
Swift, F.;
Niederer, S.;
Smith, N. P.;
Sejersted, O. M.;
Sjaastad, I.

Publication type:

Article

David Yue (1957-2014).

Published in:

Journal of Physiology, 2015, v. 593, n. 6, p. 1325, doi. 10.1113/JP270290

By:

Giles, Wayne;
Zamponi, Gerald

Publication type:

Article

Inositol-1,4,5-trisphosphate induced Ca2+ release and excitation-contraction coupling in atrial myocytes from normal and failing hearts.

Published in:

Journal of Physiology, 2015, v. 593, n. 6, p. 1459, doi. 10.1113/jphysiol.2014.283226

By:

Hohendanner, Felix;
Walther, Stefanie;
Maxwell, Joshua T.;
Kettlewell, Sarah;
Awad, Sawsan;
Smith, Godfrey L.;
Lonchyna, Vassyl A.;
Blatter, Lothar A.

Publication type:

Article

Alternating membrane potential/calcium interplay underlies repetitive focal activity in a genetic model of calcium-dependent atrial arrhythmias.

Published in:

Journal of Physiology, 2015, v. 593, n. 6, p. 1443, doi. 10.1113/jphysiol.2014.280784

By:

Lou, Qing;
Belevych, Andriy E.;
Radwański, Przemysław B.;
Liu, Bin;
Kalyanasundaram, Anuradha;
Knollmann, Bjorn C.;
Fedorov, Vadim V.;
Györke, Sándor

Publication type:

Article

Na+/Ca2+ exchange and Na+/K+-ATPase in the heart.

Published in:

Journal of Physiology, 2015, v. 593, n. 6, p. 1361, doi. 10.1113/jphysiol.2014.282319

By:

Shattock, Michael J.;
Ottolia, Michela;
Bers, Donald M.;
Blaustein, Mordecai P.;
Boguslavskyi, Andrii;
Bossuyt, Julie;
Bridge, John H. B.;
Chen‐Izu, Ye;
Clancy, Colleen E.;
Edwards, Andrew;
Goldhaber, Joshua;
Kaplan, Jack;
Lingrel, Jerry B.;
Pavlovic, Davor;
Philipson, Kenneth;
Sipido, Karin R.;
Xie, Zi‐Jian

Publication type:

Article

A leap(frog) forward in understanding focal arrhythmia.

Published in:

Journal of Physiology, 2015, v. 593, n. 6, p. 1383, doi. 10.1113/jphysiol.2014.287128

By:

Ripplinger, Crystal M.

Publication type:

Article

IP3 and Ca2+ signals in the heart: boost them or bust them?

Published in:

Journal of Physiology, 2015, v. 593, n. 6, p. 1385, doi. 10.1113/jphysiol.2014.287987

By:

Niggli, Ernst

Publication type:

Article

Deranged sodium to sudden death.

Published in:

Journal of Physiology, 2015, v. 593, n. 6, p. 1331, doi. 10.1113/jphysiol.2014.281204

By:

Clancy, Colleen E.;
Chen‐Izu, Ye;
Bers, Donald M.;
Belardinelli, Luiz;
Boyden, Penelope A.;
Csernoch, Laszlo;
Despa, Sanda;
Fermini, Bernard;
Hool, Livia C.;
Izu, Leighton;
Kass, Robert S.;
Lederer, W. Jonathan;
Louch, William E.;
Maack, Christoph;
Matiazzi, Alicia;
Qu, Zhilin;
Rajamani, Sridharan;
Rippinger, Crystal M.;
Sejersted, Ole M.;
O'Rourke, Brian

Publication type:

Article

Works matching IS 00223751 AND DT 2015 AND VI 593 AND IP 6

Maximal acceleration of Ca<sup>2+</sup> release refractoriness by β-adrenergic stimulation requires dual activation of kinases PKA and CaMKII in mouse ventricular myocytes.

Decreased inward rectifying K<sup>+</sup> current and increased ryanodine receptor sensitivity synergistically contribute to sustained focal arrhythmia in the intact rabbit heart.

Sodium and calcium regulation in cardiac myocytes: from molecules to heart failure and arrhythmia.

Na<sup>+</sup> channel function, regulation, structure, trafficking and sequestration.

Palpitations, potassium and the pump.

Scn1b deletion leads to increased tetrodotoxin-sensitive sodium current, altered intracellular calcium homeostasis and arrhythmias in murine hearts.

A computational modelling approach combined with cellular electrophysiology data provides insights into the therapeutic benefit of targeting the late Na<sup>+</sup> current.

Contribution of sodium channel neuronal isoform Na<sub>v</sub>1.1 to late sodium current in ventricular myocytes from failing hearts.

Hypokalaemia induces Ca<sup>2+</sup> overload and Ca<sup>2+</sup> waves in ventricular myocytes by reducing Na<sup>+</sup>,K<sup>+</sup>-ATPase α<sub>2</sub> activity.

David Yue (1957-2014).

Inositol-1,4,5-trisphosphate induced Ca<sup>2+</sup> release and excitation-contraction coupling in atrial myocytes from normal and failing hearts.

Alternating membrane potential/calcium interplay underlies repetitive focal activity in a genetic model of calcium-dependent atrial arrhythmias.

Na<sup>+</sup>/Ca<sup>2+</sup> exchange and Na<sup>+</sup>/K<sup>+</sup>-ATPase in the heart.

A leap(frog) forward in understanding focal arrhythmia.

IP<sub>3</sub> and Ca<sup>2+</sup> signals in the heart: boost them or bust them?

Deranged sodium to sudden death.