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Hypokalaemia induces Ca<sup>2+</sup> overload and Ca<sup>2+</sup> waves in ventricular myocytes by reducing Na<sup>+</sup>,K<sup>+</sup>-ATPase α<sub>2</sub> activity.
- Published in:
- Journal of Physiology, 2015, v. 593, n. 6, p. 1509, doi. 10.1113/jphysiol.2014.279893
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- Publication type:
- Article
David Yue (1957-2014).
- Published in:
- Journal of Physiology, 2015, v. 593, n. 6, p. 1325, doi. 10.1113/JP270290
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- Publication type:
- Article
Inositol-1,4,5-trisphosphate induced Ca<sup>2+</sup> release and excitation-contraction coupling in atrial myocytes from normal and failing hearts.
- Published in:
- Journal of Physiology, 2015, v. 593, n. 6, p. 1459, doi. 10.1113/jphysiol.2014.283226
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- Publication type:
- Article
Alternating membrane potential/calcium interplay underlies repetitive focal activity in a genetic model of calcium-dependent atrial arrhythmias.
- Published in:
- Journal of Physiology, 2015, v. 593, n. 6, p. 1443, doi. 10.1113/jphysiol.2014.280784
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- Publication type:
- Article
Maximal acceleration of Ca<sup>2+</sup> release refractoriness by β-adrenergic stimulation requires dual activation of kinases PKA and CaMKII in mouse ventricular myocytes.
- Published in:
- Journal of Physiology, 2015, v. 593, n. 6, p. 1495, doi. 10.1113/jphysiol.2014.278051
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- Publication type:
- Article
Na<sup>+</sup> channel function, regulation, structure, trafficking and sequestration.
- Published in:
- Journal of Physiology, 2015, v. 593, n. 6, p. 1347, doi. 10.1113/jphysiol.2014.281428
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- Publication type:
- Article
Decreased inward rectifying K<sup>+</sup> current and increased ryanodine receptor sensitivity synergistically contribute to sustained focal arrhythmia in the intact rabbit heart.
- Published in:
- Journal of Physiology, 2015, v. 593, n. 6, p. 1479, doi. 10.1113/jphysiol.2014.279638
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- Publication type:
- Article
Sodium and calcium regulation in cardiac myocytes: from molecules to heart failure and arrhythmia.
- Published in:
- 2015
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- Publication type:
- Editorial
Palpitations, potassium and the pump.
- Published in:
- Journal of Physiology, 2015, v. 593, n. 6, p. 1387, doi. 10.1113/jphysiol.2014.285924
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- Publication type:
- Article
Contribution of sodium channel neuronal isoform Na<sub>v</sub>1.1 to late sodium current in ventricular myocytes from failing hearts.
- Published in:
- Journal of Physiology, 2015, v. 593, n. 6, p. 1409, doi. 10.1113/jphysiol.2014.278259
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- Publication type:
- Article
Scn1b deletion leads to increased tetrodotoxin-sensitive sodium current, altered intracellular calcium homeostasis and arrhythmias in murine hearts.
- Published in:
- Journal of Physiology, 2015, v. 593, n. 6, p. 1389, doi. 10.1113/jphysiol.2014.277699
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- Publication type:
- Article
A computational modelling approach combined with cellular electrophysiology data provides insights into the therapeutic benefit of targeting the late Na<sup>+</sup> current.
- Published in:
- Journal of Physiology, 2015, v. 593, n. 6, p. 1429, doi. 10.1113/jphysiol.2014.279554
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- Publication type:
- Article
Na<sup>+</sup>/Ca<sup>2+</sup> exchange and Na<sup>+</sup>/K<sup>+</sup>-ATPase in the heart.
- Published in:
- Journal of Physiology, 2015, v. 593, n. 6, p. 1361, doi. 10.1113/jphysiol.2014.282319
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- Publication type:
- Article
A leap(frog) forward in understanding focal arrhythmia.
- Published in:
- Journal of Physiology, 2015, v. 593, n. 6, p. 1383, doi. 10.1113/jphysiol.2014.287128
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- Publication type:
- Article
IP<sub>3</sub> and Ca<sup>2+</sup> signals in the heart: boost them or bust them?
- Published in:
- Journal of Physiology, 2015, v. 593, n. 6, p. 1385, doi. 10.1113/jphysiol.2014.287987
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- Publication type:
- Article
Deranged sodium to sudden death.
- Published in:
- Journal of Physiology, 2015, v. 593, n. 6, p. 1331, doi. 10.1113/jphysiol.2014.281204
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- Publication type:
- Article