Works matching IS 00219541 AND DT 2000 AND VI 183 AND IP 3
Results: 16
Thrombin-stimulated phosphatidylinositol 3-kinase activity in platelets is associated with activation of PYK2 tyrosine kinase: activation of both enzymes is aggregation independent.
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- Journal of Cellular Physiology, 2000, v. 183, n. 3, p. 314, doi. 10.1002/(SICI)1097-4652(200006)183:3<314::AID-JCP3>3.0.CO;2-J
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Tumor necrosis factor-α-induced apoptosis is associated with suppression of insulin-like growth factor binding protein-5 secretion in differentiating murine skeletal myoblasts.
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- Journal of Cellular Physiology, 2000, v. 183, n. 3, p. 330, doi. 10.1002/(SICI)1097-4652(200006)183:3<330::AID-JCP5>3.0.CO;2-N
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Rapid lamellipodia formation in nerve growth factor-stimulated PC12 cells is dependent on Rac and PI3K activity.
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- Journal of Cellular Physiology, 2000, v. 183, n. 3, p. 416, doi. 10.1002/(SICI)1097-4652(200006)183:3<416::AID-JCP15>3.0.CO;2-R
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Stretch-activated signaling of nerve growth factor secretion in bladder and vascular smooth muscle cells from hypertensive and hyperactive rats.
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- Journal of Cellular Physiology, 2000, v. 183, n. 3, p. 289, doi. 10.1002/(SICI)1097-4652(200006)183:3<289::AID-JCP1>3.0.CO;2-6
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Expression of functional mGlu5 metabotropic glutamate receptors in human melanocytes.
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- Journal of Cellular Physiology, 2000, v. 183, n. 3, p. 364, doi. 10.1002/(SICI)1097-4652(200006)183:3<364::AID-JCP9>3.0.CO;2-X
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Interaction of Smad3 with a proximal smad-binding element of the human α2(I) procollagen gene promoter required for transcriptional activation by TGF-β.
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- Journal of Cellular Physiology, 2000, v. 183, n. 3, p. 381, doi. 10.1002/(SICI)1097-4652(200006)183:3<381::AID-JCP11>3.0.CO;2-O
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Divergent phenotypic patterns and commitment to apoptosis of Caco-2 cells during spontaneous and butyrate-induced differentiation.
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- Journal of Cellular Physiology, 2000, v. 183, n. 3, p. 347, doi. 10.1002/(SICI)1097-4652(200006)183:3<347::AID-JCP7>3.0.CO;2-W
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Analysis of Myc/Max/Mad network members in adipogenesis: Inhibition of the proliferative burst and differentiation by ectopically expressed Mad1.
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- Journal of Cellular Physiology, 2000, v. 183, n. 3, p. 399, doi. 10.1002/(SICI)1097-4652(200006)183:3<399::AID-JCP13>3.0.CO;2-7
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Ectopic expression of the ErbB-3 binding protein Ebp1 inhibits growth and induces differentiation of human breast cancer cell lines.
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- Journal of Cellular Physiology, 2000, v. 183, n. 3, p. 321, doi. 10.1002/(SICI)1097-4652(200006)183:3<321::AID-JCP4>3.0.CO;2-O
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High glucose-induced inhibition of α-methyl- D-glucopyranoside uptake is mediated by protein kinase C-dependent activation of arachidonic acid release in primary cultured rabbit renal proximal tubule cells.
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- Journal of Cellular Physiology, 2000, v. 183, n. 3, p. 355, doi. 10.1002/(SICI)1097-4652(200006)183:3<355::AID-JCP8>3.0.CO;2-Y
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Intracellular pH regulation in a nonmalignant and a derived malignant human breast cell line.
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- Journal of Cellular Physiology, 2000, v. 183, n. 3, p. 373, doi. 10.1002/(SICI)1097-4652(200006)183:3<373::AID-JCP10>3.0.CO;2-S
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Grb7 signal transduction protein mediates metastatic progression of esophageal carcinoma.
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- Journal of Cellular Physiology, 2000, v. 183, n. 3, p. 411, doi. 10.1002/(SICI)1097-4652(200006)183:3<411::AID-JCP14>3.0.CO;2-Z
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Heregulin-β is especially potent in activating phosphatidylinositol 3-kinase in nontransformed human mammary epithelial cells.
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- Journal of Cellular Physiology, 2000, v. 183, n. 3, p. 301, doi. 10.1002/(SICI)1097-4652(200006)183:3<301::AID-JCP2>3.0.CO;2-W
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Involvement of hepatocyte epidermal growth factor receptor mediated activation of mitogen-activated protein kinase signaling pathways in response to growth inhibition by a novel K vitamin.
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- Journal of Cellular Physiology, 2000, v. 183, n. 3, p. 338, doi. 10.1002/(SICI)1097-4652(200006)183:3<338::AID-JCP6>3.0.CO;2-X
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Adenosine acts as a chemoprotective agent by stimulating G-CSF production: A role for A1 and A3 adenosine receptors.
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- Journal of Cellular Physiology, 2000, v. 183, n. 3, p. 393, doi. 10.1002/(SICI)1097-4652(200006)183:3<393::AID-JCP12>3.0.CO;2-G
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Sensitization of tumor cells to Fas killing through overexpression of heat-shock transcription factor 1.
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- Journal of Cellular Physiology, 2000, v. 183, n. 3, p. 425, doi. 10.1002/(SICI)1097-4652(200006)183:3<425::AID-JCP16>3.0.CO;2-M
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