Independent roles of the priming and the triggering of the NLRP3 inflammasome in the heart.
- Published in:
- Cardiovascular Research, 2015, v. 105, n. 2, p. 203, doi. 10.1093/cvr/cvu259
- By:
- Publication type:
- Article
Works matching IS 00086363 AND DT 2015 AND VI 105 AND IP 2
Results: 15
1
2
Na<sup>+</sup> ions as spatial intracellular messengers for co-ordinating Ca<sup>2+</sup> signals during pH heterogeneity in cardiomyocytes.
- Published in:
- Cardiovascular Research, 2015, v. 105, n. 2, p. 171, doi. 10.1093/cvr/cvu251
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- Publication type:
- Article
3
Optogenetic release of norepinephrine from cardiac sympathetic neurons alters mechanical and electrical function.
- Published in:
- Cardiovascular Research, 2015, v. 105, n. 2, p. 143, doi. 10.1093/cvr/cvu258
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- Publication type:
- Article
4
Dissecting abdominal aortic aneurysm in Ang II-infused mice: suprarenal branch ruptures and apparent luminal dilatation.
- Published in:
- Cardiovascular Research, 2015, v. 105, n. 2, p. 213, doi. 10.1093/cvr/cvu257
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- Publication type:
- Article
5
Impaired P2X signalling pathways in renal microvascularmyocytes in genetic hypertension.
- Published in:
- Cardiovascular Research, 2015, v. 105, n. 2, p. 131, doi. 10.1093/cvr/cvu249
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- Publication type:
- Article
6
Marine n-3 PUFAs modulate I<sub>Ks</sub> gating, channel expression, and location in membrane microdomains.
- Published in:
- Cardiovascular Research, 2015, v. 105, n. 2, p. 223, doi. 10.1093/cvr/cvu250
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- Publication type:
- Article
7
Renalase is a novel target gene of hypoxia-inducible factor-1 in protection against cardiac ischaemia–reperfusion injury.
- Published in:
- Cardiovascular Research, 2015, v. 105, n. 2, p. 182, doi. 10.1093/cvr/cvu255
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- Publication type:
- Article
8
Uncoupling protein 3 mediates H<sub>2</sub>O<sub>2</sub> preconditioning-afforded cardioprotection through the inhibition of MPTP opening.
- Published in:
- Cardiovascular Research, 2015, v. 105, n. 2, p. 192, doi. 10.1093/cvr/cvu256
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- Publication type:
- Article
9
Akt activation by PHLPP1 ablation prevents pathological hypertrophy by promoting angiogenesis.
- Published in:
- Cardiovascular Research, 2015, v. 105, n. 2, p. 129, doi. 10.1093/cvr/cvu261
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- Publication type:
- Article
10
Physiological activation of Akt by PHLPP1 deletion protects against pathological hypertrophy.
- Published in:
- Cardiovascular Research, 2015, v. 105, n. 2, p. 160, doi. 10.1093/cvr/cvu243
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- Publication type:
- Article
11
Aims and Scope.
- Published in:
- Cardiovascular Research, 2015, v. 105, n. 2, p. NP, doi. 10.1093/cvr/cvv011
- Publication type:
- Article
12
Cover Page.
- Published in:
- Cardiovascular Research, 2015, v. 105, n. 2, p. NP, doi. 10.1093/cvr/cvv012
- Publication type:
- Article
13
Contents Page.
- Published in:
- Cardiovascular Research, 2015, v. 105, n. 2, p. NP, doi. 10.1093/cvr/cvv014
- Publication type:
- Article
14
Editorial Board.
- Published in:
- Cardiovascular Research, 2015, v. 105, n. 2, p. NP, doi. 10.1093/cvr/cvv013
- Publication type:
- Article
15
Deficiency of filamin A in endothelial cells impairs left ventricular remodelling after myocardial infarction.
- Published in:
- Cardiovascular Research, 2015, v. 105, n. 2, p. 151, doi. 10.1093/cvr/cvu226
- By:
- Publication type:
- Article