Works matching IS 00086363 AND DT 1999 AND VI 41 AND IP 2
Results: 19
Evidence for 12-lipoxygenase induction in the vessel wall following balloon injury.
- Published in:
- Cardiovascular Research, 1999, v. 41, n. 2, p. 489, doi. 10.1016/S0008-6363(98)00312-5
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- Article
Role of smooth muscle cell death in advanced coronary primary lesions: implications for plaque instability.
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- Cardiovascular Research, 1999, v. 41, n. 2, p. 480, doi. 10.1016/S0008-6363(98)00318-6
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- Article
Biphasic pattern of cell turnover characterizes the progression from fatty streaks to ruptured human atherosclerotic plaques.
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- Cardiovascular Research, 1999, v. 41, n. 2, p. 473, doi. 10.1016/S0008-6363(98)00311-3
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- Article
Expression of inducible nitric oxide synthase in human coronary atherosclerotic plaque.
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- Cardiovascular Research, 1999, v. 41, n. 2, p. 465, doi. 10.1016/S0008-6363(98)00304-6
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Angioscopic complex lesions are predominantly compensatory enlarged: an angioscopy and intracoronary ultrasound study.
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- Cardiovascular Research, 1999, v. 41, n. 2, p. 458, doi. 10.1016/S0008-6363(98)00320-4
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- Article
Collagen–platelet interaction: Gly-Pro-Hyp is uniquely specific for platelet Gp VI and mediates platelet activation by collagen1.
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- Cardiovascular Research, 1999, v. 41, n. 2, p. 450, doi. 10.1016/S0008-6363(98)00306-X
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- Article
Leucocyte recruitment in rupture prone regions of lipid-rich plaques: a prominent role for neovascularization?
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- Cardiovascular Research, 1999, v. 41, n. 2, p. 443, doi. 10.1016/S0008-6363(98)00255-7
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Stents covered by an autologous arterial graft in porcine coronary arteries: feasibility, vascular injury and effect on neointimal hyperplasia.
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- Cardiovascular Research, 1999, v. 41, n. 2, p. 433, doi. 10.1016/S0008-6363(98)00211-9
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Prevention of activation of blood coagulation during acute coronary ischemic syndromes.
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- Cardiovascular Research, 1999, v. 41, n. 2, p. 418, doi. 10.1016/S0008-6363(98)00323-X
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Strategies to achieve coronary arterial plaque stabilization.
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- Cardiovascular Research, 1999, v. 41, n. 2, p. 402, doi. 10.1016/S0008-6363(98)00279-X
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Cellular adhesion molecules on vascular smooth muscle cells.
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- Cardiovascular Research, 1999, v. 41, n. 2, p. 395, doi. 10.1016/S0008-6363(98)00302-2
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Heme oxygenase–carbon monoxide signalling pathway in atherosclerosis: anti-atherogenic actions of bilirubin and carbon monoxide?
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- Cardiovascular Research, 1999, v. 41, n. 2, p. 385, doi. 10.1016/S0008-6363(98)00278-8
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Collagen synthesis in atherosclerosis: too much and not enough.
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- Cardiovascular Research, 1999, v. 41, n. 2, p. 376, doi. 10.1016/S0008-6363(98)00321-6
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Mechanisms of plaque rupture.
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- Cardiovascular Research, 1999, v. 41, n. 2, p. 369, doi. 10.1016/S0008-6363(98)00308-3
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Apoptosis of vascular smooth muscle cells in vascular remodelling and atherosclerotic plaque rupture.
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- Cardiovascular Research, 1999, v. 41, n. 2, p. 361, doi. 10.1016/S0008-6363(98)00212-0
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- Article
Fibrous cap formation or destruction — the critical importance of vascular smooth muscle cell proliferation, migration and matrix formation.
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- Cardiovascular Research, 1999, v. 41, n. 2, p. 345, doi. 10.1016/S0008-6363(98)00286-7
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- Article
Atherosclerotic plaque rupture – pathologic basis of plaque stability and instability.
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- Cardiovascular Research, 1999, v. 41, n. 2, p. 334, doi. 10.1016/S0008-6363(98)00276-4
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Pathophysiology and clinical significance of atherosclerotic plaque rupture.
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- Cardiovascular Research, 1999, v. 41, n. 2, p. 323, doi. 10.1016/S0008-6363(98)00322-8
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- Article
Plaque instability — the real challenge for atherosclerosis research in the next decade?
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- Cardiovascular Research, 1999, v. 41, n. 2, p. 321
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