Decreased SAPK/ JNK signalling affects cytokine release and STAT3 activation in psoriatic fibroblasts.
- Published in:
- Experimental Dermatology, 2015, v. 24, n. 10, p. 800, doi. 10.1111/exd.12787
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- Publication type:
- Article
Works matching DE "C-Jun N-terminal kinases regulation"
Results: 13
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Inhibition of c-Jun N-terminal Kinase Ameliorates Early Brain Injury After Subarachnoid Hemorrhage Through Inhibition of a Nur77 Dependent Apoptosis Pathway.
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- Neurochemical Research, 2014, v. 39, n. 8, p. 1603, doi. 10.1007/s11064-014-1355-6
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- Publication type:
- Article
3
The oncogene c-Jun impedes somatic cell reprogramming.
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- Nature Cell Biology, 2015, v. 17, n. 7, p. 856, doi. 10.1038/ncb3193
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- Publication type:
- Article
4
Melanoma cell adhesion molecule stimulates yes-associated protein transcription by enhancing CREB activity via c-Jun/c-Fos in hepatocellular carcinoma cells.
- Published in:
- Oncology Letters, 2016, v. 11, n. 6, p. 3702, doi. 10.3892/ol.2016.4442
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- Publication type:
- Article
5
Interleukin-33 induces interleukin-8 expression via JNK/c-Jun/AP-1 pathway in human umbilical vein endothelial cells.
- Published in:
- PLoS ONE, 2018, v. 13, n. 1, p. 1, doi. 10.1371/journal.pone.0191659
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- Publication type:
- Article
6
Power Frequency Magnetic Fields Affect the p38 MAPK-Mediated Regulation of NB69 Cell Proliferation Implication of Free Radicals.
- Published in:
- International Journal of Molecular Sciences, 2016, v. 17, n. 4, p. 510, doi. 10.3390/ijms17040510
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- Publication type:
- Article
7
c-Jun N-terminal kinase regulates mGluR-dependent expression of post-synaptic FMRP target proteins.
- Published in:
- Journal of Neurochemistry, 2013, v. 127, n. 6, p. 772, doi. 10.1111/jnc.12453
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- Publication type:
- Article
8
ERK1/2, p38, and JNK regulate the expression and the activity of the three isoforms of the Na<sup>+</sup>/Ca<sup>2+</sup>exchanger, NCX1, NCX2, and NCX3, in neuronal PC12 cells.
- Published in:
- Journal of Neurochemistry, 2012, v. 122, n. 5, p. 911, doi. 10.1111/j.1471-4159.2012.07838.x
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- Publication type:
- Article
9
De-Regulation of JNK and JAK/STAT Signaling in <i>ESCRT-II</i> Mutant Tissues Cooperatively Contributes to Neoplastic Tumorigenesis.
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- PLoS ONE, 2013, v. 8, n. 2, p. 1, doi. 10.1371/journal.pone.0056021
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- Publication type:
- Article
10
JNK3 Maintains Expression of the Insulin Receptor Substrate 2 (IRS2) in Insulin-Secreting Cells: Functional Consequences for Insulin Signaling.
- Published in:
- PLoS ONE, 2012, v. 7, n. 5, p. 1, doi. 10.1371/journal.pone.0035997
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- Publication type:
- Article
11
Endoplasmic Reticulum Stress-Induced JNK Activation Is a Critical Event Leading to Mitochondria-Mediated Cell Death Caused by β-Lapachone Treatment.
- Published in:
- PLoS ONE, 2011, v. 6, n. 6, p. 1, doi. 10.1371/journal.pone.0021533
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- Publication type:
- Article
12
Effects of the JWA gene in the regulation of human breast cancer cells.
- Published in:
- Molecular Medicine Reports, 2015, v. 11, n. 5, p. 3848, doi. 10.3892/mmr.2015.3188
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- Publication type:
- Article
13
Glabridin induces apoptosis and cell cycle arrest in oral cancer cells through the JNK1/2 signaling pathway.
- Published in:
- Environmental Toxicology, 2018, v. 33, n. 6, p. 679, doi. 10.1002/tox.22555
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- Publication type:
- Article