Found: 5
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Vitiligo pathogenesis: autoimmune disease, genetic defect, excessive reactive oxygen species, calcium imbalance, or what else?
- Published in:
- 2008
- By:
- Publication type:
- Other
Tenascin is overexpressed in vitiligo lesional skin and inhibits melanocyte adhesion.
- Published in:
- British Journal of Dermatology, 1997, v. 137, n. 2, p. 171, doi. 10.1046/j.1365-2133.1997.18011894.x
- By:
- Publication type:
- Article
Cutaneous Biology Expansion of vitiligo lesions is associated with reduced epidermal CDw60 expression and increased expression of HLA-DR in perilesional skin.
- Published in:
- British Journal of Dermatology, 2003, v. 149, n. 4, p. 739, doi. 10.1046/j.1365-2133.2003.05539.x
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- Publication type:
- Article
Revised classification/nomenclature of vitiligo and related issues: the Vitiligo Global Issues Consensus Conference.
- Published in:
- Pigment Cell & Melanoma Research, 2012, v. 25, n. 3, p. E1, doi. 10.1111/j.1755-148X.2012.00997.x
- By:
- Publication type:
- Article
Review of the etiopathomechanism of vitiligo: A convergence theory.
- Published in:
- Experimental Dermatology, 1993, v. 2, n. 4, p. 145, doi. 10.1111/j.1600-0625.1993.tb00023.x
- By:
- Publication type:
- Article