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A Role for Human DNA Polymerase λ in Alternative Lengthening of Telomeres.
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- International Journal of Molecular Sciences, 2021, v. 22, n. 5, p. 2365, doi. 10.3390/ijms22052365
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- Article
Detection of Telomeric DNA:RNA Hybrids Using TeloDRIP-qPCR.
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- International Journal of Molecular Sciences, 2020, v. 21, n. 24, p. 9774, doi. 10.3390/ijms21249774
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- Article
The Gam protein of bacteriophage Mu is an orthologue of eukaryotic Ku.
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- EMBO Reports, 2003, v. 4, n. 1, p. 47, doi. 10.1038/sj.embor.embor709
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- Article
Human replication protein Cdc6 is selectively cleaved by caspase 3 during apoptosis.
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- EMBO Reports, 2002, v. 3, n. 8, p. 780, doi. 10.1093/embo-reports/kvf161
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- Article
Telomere damage promotes vascular smooth muscle cell senescence and immune cell recruitment after vessel injury.
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- Communications Biology, 2021, v. 4, n. 1, p. 1, doi. 10.1038/s42003-021-02123-z
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- Article
Resection is responsible for loss of transcription around a double-strand break in Saccharomyces cerevisiae.
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- eLife, 2015, p. 1, doi. 10.7554/eLife.08942
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- Article
Neural stem cells exposed to BrdU lose their global DNA methylation and undergo astrocytic differentiation.
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- Nucleic Acids Research, 2012, v. 40, n. 13, p. 5332, doi. 10.1093/nar/gks207
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- Article
Neural stem cells exposed to BrdU lose their global DNA methylation and undergo astrocytic differentiation.
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- Nucleic Acids Research, 2012, v. 40, n. 12, p. 5332, doi. 10.1093/nar/gks207
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- Article
TGS1 mediates 2,2,7-trimethyl guanosine capping of the human telomerase RNA to direct telomerase dependent telomere maintenance.
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- Nature Communications, 2022, v. 13, n. 1, p. 1, doi. 10.1038/s41467-022-29907-z
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- Article
BRCA1 deficiency specific base substitution mutagenesis is dependent on translesion synthesis and regulated by 53BP1.
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- Nature Communications, 2022, v. 13, n. 1, p. 1, doi. 10.1038/s41467-021-27872-7
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- Article
Functions of poly(ADP-ribose) polymerase in controlling telomere length and chromosomal stability.
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- Nature Genetics, 1999, v. 23, n. 1, p. 76, doi. 10.1038/12680
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- Article
Notch is a direct negative regulator of the DNA-damage response.
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- Nature Structural & Molecular Biology, 2015, v. 22, n. 5, p. 417, doi. 10.1038/nsmb.3013
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- Article
Cellular senescence: when bad things happen to good cells.
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- Nature Reviews Molecular Cell Biology, 2007, v. 8, n. 9, p. 729, doi. 10.1038/nrm2233
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Transcriptional and post-transcriptional regulation of the ionizing radiation response by ATM and p53.
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- Scientific Reports, 2017, p. 43598, doi. 10.1038/srep43598
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- Article
Alternative lengthening of telomeres (ALT) cells viability is dependent on C-rich telomeric RNAs.
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- Nature Communications, 2023, v. 14, n. 1, p. 1, doi. 10.1038/s41467-023-42831-0
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- Article
Stable Cellular Senescence Is Associated with Persistent DDR Activation.
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- PLoS ONE, 2014, v. 9, n. 10, p. 1, doi. 10.1371/journal.pone.0110969
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DNA damage response at telomeres boosts the transcription of SARS‐CoV‐2 receptor ACE2 during aging.
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- EMBO Reports, 2022, v. 23, n. 2, p. 1, doi. 10.15252/embr.202153658
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- Article
RNA-processing proteins regulate Mec1/ ATR activation by promoting generation of RPA-coated ss DNA.
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- EMBO Reports, 2015, v. 16, n. 2, p. 221, doi. 10.15252/embr.201439458
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- Article
Inhibition of DNA damage response at telomeres improves the detrimental phenotypes of Hutchinson–Gilford Progeria Syndrome.
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- Nature Communications, 2019, v. 10, n. 1, p. 1, doi. 10.1038/s41467-019-13018-3
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- Article
BRCA2 controls DNA:RNA hybrid level at DSBs by mediating RNase H2 recruitment.
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- Nature Communications, 2018, v. 9, n. 1, p. 1, doi. 10.1038/s41467-018-07799-2
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- Article
Telomerase abrogates aneuploidy-induced telomere replication stress, senescence and cell depletion.
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- EMBO Journal, 2017, v. 36, n. 19, p. 2922, doi. 10.15252/embj.201797470
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- Article
Telomerase abrogates aneuploidy-induced telomere replication stress, senescence and cell depletion.
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- EMBO Journal, 2015, v. 34, n. 10, p. 1371, doi. 10.15252/embj.201490070
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- Article
Oncogene-induced telomere dysfunction enforces cellular senescence in human cancer precursor lesions.
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- EMBO Journal, 2012, v. 31, n. 13, p. 2839, doi. 10.1038/emboj.2012.132
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- Article
Oncogene-induced senescence is a DNA damage response triggered by DNA hyper-replication.
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- Nature, 2006, v. 444, n. 7119, p. 638, doi. 10.1038/nature05327
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- Article
Telomeric DNA damage is irreparable and causes persistent DNA-damage-response activation.
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- Nature Cell Biology, 2012, v. 14, n. 5, p. 555, doi. 10.1038/ncb0512-555b
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- Article
Telomeric DNA damage is irreparable and causes persistent DNA-damage-response activation.
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- Nature Cell Biology, 2012, v. 14, n. 4, p. 355, doi. 10.1038/ncb2466
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Interplay between oncogene-induced DNA damage response and heterochromatin in senescence and cancer.
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- Nature Cell Biology, 2011, v. 13, n. 3, p. 292, doi. 10.1038/ncb2170
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SASPense and DDRama in cancer and ageing.
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- Nature Cell Biology, 2009, v. 11, n. 8, p. 921, doi. 10.1038/ncb0809-921
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- Article
Cleavage of the Bloom’s syndrome gene product during apoptosis by caspase-3 results in an impaired interaction with topoisomerase IIIα.
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- Nucleic Acids Research, 2001, v. 29, n. 15, p. 3172, doi. 10.1093/nar/29.15.3172
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- Article
Express or repress? The transcriptional dilemma of damaged chromatin.
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- FEBS Journal, 2017, v. 284, n. 14, p. 2133, doi. 10.1111/febs.14048
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- Article
A damaged genome's transcriptional landscape through multilayered expression profiling around in situ-mapped DNA double-strand breaks.
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- Nature Communications, 2017, v. 8, n. 5, p. 15656, doi. 10.1038/ncomms15656
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Corrigendum: DNA damage response inhibition at dysfunctional telomeres by modulation of telomeric DNA damage response RNAs.
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- Nature Communications, 2017, v. 8, n. 4, p. 15344, doi. 10.1038/ncomms15344
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- Article
DNA damage response inhibition at dysfunctional telomeres by modulation of telomeric DNA damage response RNAs.
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- Nature Communications, 2017, v. 8, n. 2, p. 13980, doi. 10.1038/ncomms13980
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- Article
Polycomb proteins control proliferation and transformation independently of cell cycle checkpoints by regulating DNA replication.
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- Nature Communications, 2014, v. 5, n. 4, p. 3649, doi. 10.1038/ncomms4649
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Need telomere maintenance? Call 911.
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- Cell Division, 2007, v. 2, p. 3, doi. 10.1186/1747-1028-2-3
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A DNA damage checkpoint response in telomere-initiated senescence.
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- Nature, 2003, v. 426, n. 6963, p. 194, doi. 10.1038/nature02118
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- Article
Colorectal cancer patient-derived organoids and cell lines harboring ATRX and/or DAXX mutations lack Alternative Lengthening of Telomeres (ALT).
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- Cell Death & Disease, 2023, v. 14, n. 2, p. 1, doi. 10.1038/s41419-023-05640-3
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- Article
Site-specific DICER and DROSHA RNA products control the DNA-damage response.
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- Nature, 2012, v. 488, n. 7410, p. 231, doi. 10.1038/nature11179
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RNase A treatment and reconstitution with DNA damage response RNA in living cells as a tool to study the role of non-coding RNA in the formation of DNA damage response foci.
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- Nature Protocols, 2019, v. 14, n. 5, p. 1489, doi. 10.1038/s41596-019-0147-5
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- Article
Crosstalk between chromatin state and DNA damage response in cellular senescence and cancer.
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- Nature Reviews Cancer, 2012, v. 12, n. 10, p. 709, doi. 10.1038/nrc3344
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- Article
Living on a break: cellular senescence as a DNA-damage response.
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- Nature Reviews Cancer, 2008, v. 8, n. 7, p. 512, doi. 10.1038/nrc2440
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- Article
A novel single-cell method provides direct evidence of persistent DNA damage in senescent cells and aged mammalian tissues.
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- Aging Cell, 2017, v. 16, n. 2, p. 422, doi. 10.1111/acel.12573
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Is cellular senescence an example of antagonistic pleiotropy?
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- Aging Cell, 2012, v. 11, n. 3, p. 378, doi. 10.1111/j.1474-9726.2012.00807.x
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- Article