Works matching AU Yousif, Mariam H. M.
Results: 18
Chronic Treatment with Ang-(1-7) Reverses Abnormal Reactivity in the Corpus Cavernosum and Normalizes Diabetes-Induced Changes in the Protein Levels of ACE, ACE2, ROCK1, ROCK2 and Omega-Hydroxylase in a Rat Model of Type 1 Diabetes.
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- Journal of Diabetes Research, 2014, p. 1, doi. 10.1155/2014/142154
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- Article
FPTIII Mitigates Peroxisome-Mediated Oxidative Stress in Kidneys of Spontaneously Hypertensive Diabetic Rats.
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- Kidney & Blood Pressure Research, 2010, v. 33, n. 1, p. 60, doi. 10.1159/000289574
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- Article
The dual targeting of EGFR and ErbB2 with the inhibitor Lapatinib corrects high glucose-induced apoptosis and vascular dysfunction by opposing multiple diabetes-induced signaling changes.
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- Journal of Drug Targeting, 2015, v. 23, n. 6, p. 506, doi. 10.3109/1061186X.2015.1057150
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- Article
Role of epidermal growth factor receptor (EGFR) in corneal remodelling in diabetes.
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- Acta Ophthalmologica (1755375X), 2009, v. 87, n. 8, p. 881, doi. 10.1111/j.1755-3768.2008.01434.x
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- Article
Transactivation of ErbB Family of Receptor Tyrosine Kinases Is Inhibited by Angiotensin-(1-7) via Its Mas Receptor.
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- PLoS ONE, 2015, v. 10, n. 10, p. 1, doi. 10.1371/journal.pone.0141657
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- Article
Cationic Polyamidoamine Dendrimers as Modulators of EGFR Signaling In Vitro and In Vivo.
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- PLoS ONE, 2015, v. 10, n. 7, p. 1, doi. 10.1371/journal.pone.0132215
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- Article
Diabetes-Induced Renal Vascular Dysfunction Is Normalized by Inhibition of Epidermal Growth Factor Receptor Tyrosine Kinase.
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- Journal of Vascular Research, 2005, v. 42, n. 4, p. 284, doi. 10.1159/000085904
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- Article
Phosphoinositide 3-kinase contributes to diabetes-induced abnormal vascular reactivity in rat perfused mesenteric bed.
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- Cell Biochemistry & Function, 2008, v. 26, n. 4, p. 451, doi. 10.1002/cbf.1463
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- Article
Role of Ca<sup>2+</sup>/calmodulin-dependent protein kinase II in development of vascular dysfunction in diabetic rats with hypertension.
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- Cell Biochemistry & Function, 2008, v. 26, n. 2, p. 256, doi. 10.1002/cbf.1446
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- Article
Cardioprotection from ischemia-reperfusion injury due to Ras-GTPase inhibition is attenuated by glibenclamide in the globally ischemic heart.
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- Cell Biochemistry & Function, 2007, v. 25, n. 4, p. 455, doi. 10.1002/cbf.1353
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- Article
Role of protein kinases in mediating diabetes-induced augmented vasoconstriction to endothelin-1 in the renal arteries of STZ-diabetic rats.
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- Cell Biochemistry & Function, 2006, v. 24, n. 5, p. 397, doi. 10.1002/cbf.1253
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- Article
Signal transduction through Ras-GTPase and Ca<sup>2+</sup>/ calmodulin-dependent protein kinase II contributes to development of diabetes-induced renal vascular dysfunction.
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- Cell Biochemistry & Function, 2006, v. 24, n. 4, p. 299, doi. 10.1002/cbf.1301
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- Article
Phosphoinositide 3-kinase mediated signalling contributes to development of diabetes-induced abnormal vascular reactivity of rat carotid artery.
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- Cell Biochemistry & Function, 2006, v. 24, n. 1, p. 13, doi. 10.1002/cbf.1278
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- Article
Vasodilator Effect of Trace Amines, 3-Iodothyronamine, and RO5263397 in the Rat Perfused Kidney: Comparison with Tryptamine.
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- Pharmacology, 2023, v. 108, n. 4, p. 368, doi. 10.1159/000530649
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- Article
Role of 20-Hydroxyeicosatetraenoic and Epoxyeicosatrienoic Acids in the Regulation of Vascular Function in a Model of Hypertension and Endothelial Dysfunction.
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- Pharmacology, 2010, v. 86, n. 3, p. 149, doi. 10.1159/000317521
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- Article
Epidermal growth factor receptor tyrosine kinase-mediated signalling contributes to diabetes-induced vascular dysfunction in the mesenteric bed.
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- British Journal of Pharmacology, 2005, v. 145, n. 6, p. 829, doi. 10.1038/sj.bjp.0706238
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- Article
Activation of ErbB2 and Downstream Signalling via Rho Kinases and ERK1/2 Contributes to Diabetes-Induced Vascular Dysfunction.
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- PLoS ONE, 2013, v. 8, n. 6, p. 1, doi. 10.1371/journal.pone.0067813
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- Article
Activation of EGFR/ERBB2 via Pathways Involving ERK1/2, P38 MAPK, AKT and FOXO Enhances Recovery of Diabetic Hearts from Ischemia-Reperfusion Injury.
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- PLoS ONE, 2012, v. 7, n. 6, p. 1, doi. 10.1371/journal.pone.0039066
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- Article