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The Mitochondrial Ca<sup>2+</sup> Overload via Voltage-Gated Ca<sup>2+</sup> Entry Contributes to an Anti-Melanoma Effect of Diallyl Trisulfide.
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- International Journal of Molecular Sciences, 2020, v. 21, n. 2, p. 1, doi. 10.3390/ijms21020491
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- Article
Disrupting mitochondrial Ca<sup>2+</sup> homeostasis causes tumor-selective TRAIL sensitization through mitochondrial network abnormalities.
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- International Journal of Oncology, 2017, v. 51, n. 4, p. 1146, doi. 10.3892/ijo.2017.4096
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- Article
Mitochondrial Ca<sup>2+</sup> removal amplifies TRAIL cytotoxicity toward apoptosis-resistant tumor cells via promotion of multiple cell death modalities.
- Published in:
- International Journal of Oncology, 2017, v. 51, n. 1, p. 193, doi. 10.3892/ijo.2017.4020
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- Article
Mitochondrial division inhibitor-1 induces mitochondrial hyperfusion and sensitizes human cancer cells to TRAIL-induced apoptosis.
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- International Journal of Oncology, 2014, v. 45, n. 5, p. 1901, doi. 10.3892/ijo.2014.2608
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- Article
Mitochondria-associated endoplasmic reticulum membranes microenvironment: targeting autophagic and apoptotic pathways in cancer therapy.
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- Frontiers in Oncology, 2015, p. 1, doi. 10.3389/fonc.2015.00173
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- Article
The human cathelicidin antimicrobial peptide LL-37 and mimics are potential anticancer drugs.
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- Frontiers in Oncology, 2015, p. 1, doi. 10.3389/fonc.2015.00144
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- Article
Depolarization controls TRAIL-sensitization and tumor-selective killing of cancer cells: crosstalk with ROS.
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- Frontiers in Oncology, 2014, v. 4, p. 1, doi. 10.3389/fonc.2014.00128
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- Article