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Globally elevating the AGE clearance receptor, OST48, does not protect against the development of diabetic kidney disease, despite improving insulin secretion.
- Published in:
- Scientific Reports, 2019, v. 9, n. 1, p. N.PAG, doi. 10.1038/s41598-019-50221-0
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- Publication type:
- Article
Modulation of the Cellular Expression of Circulating Advanced Glycation End-Product Receptors in Type 2 Diabetic Nephropathy.
- Published in:
- Experimental Diabetes Research, 2010, p. 1, doi. 10.1155/2010/974681
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- Publication type:
- Article
The AGE receptor, OST48 drives podocyte foot process effacement and basement membrane expansion (alters structural composition).
- Published in:
- Endocrinology, Diabetes & Metabolism, 2021, v. 4, n. 3, p. 1, doi. 10.1002/edm2.278
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- Publication type:
- Article
Advanced glycation end products are direct modulators of β-cell function.
- Published in:
- 2011
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- Publication type:
- journal article
Advanced Glycation End Products Are Direct Modulators of β-Cell Function.
- Published in:
- Diabetes, 2011, v. 60, n. 10, p. 2523, doi. 10.2337/db10-1033
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- Publication type:
- Article
Inhibition of NADPH Oxidase Prevents Advanced Glycation End Product--Mediated Damage in Diabetic Nephropathy Through a Protein Kinase C-α--Dependent Pathway.
- Published in:
- Diabetes, 2008, v. 57, n. 2, p. 460, doi. 10.2337/db07-1119
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- Publication type:
- Article