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NF-?B p105 is a target of I?B kinases and controls signal induction of Bcl-3-p50 complexes.
- Published in:
- EMBO Journal, 1999, v. 18, n. 17, p. 4766, doi. 10.1093/emboj/18.17.4766
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- Article
miR-128 regulates neuronal migration, outgrowth and intrinsic excitability via the intellectual disability gene Phf6.
- Published in:
- eLife, 2015, p. 1, doi. 10.7554/eLife.04263.001
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- Article
Candidate proto-oncogene bcl-3 encodes a subunit-specific inhibitor of transcription factor...
- Published in:
- Nature, 1992, v. 358, n. 6387, p. 597, doi. 10.1038/358597a0
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- Article
Expression of Toll-Like Receptors in the Developing Brain.
- Published in:
- PLoS ONE, 2012, v. 7, n. 5, p. 1, doi. 10.1371/journal.pone.0037767
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- Article
The let-7 target gene mouse lin-41 is a stem cell specific E3 ubiquitin ligase for the miRNA pathway protein Ago2.
- Published in:
- Nature Cell Biology, 2009, v. 11, n. 12, p. 1411, doi. 10.1038/ncb1987
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- Article
A feedback loop comprising lin-28 and let-7 controls pre-let-7 maturation during neural stem-cell commitment.
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- Nature Cell Biology, 2008, v. 10, n. 8, p. 987, doi. 10.1038/ncb1759
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- Article
The Bcl-3 oncoprotein acts as a bridging factor between NF-κB/Rel and nuclear co-regulators.
- Published in:
- Oncogene, 1999, v. 18, n. 22, p. 3316, doi. 10.1038/sj.onc.1202717
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- Article
Signal‐dependent degradation of IκBα is mediated by an inducible destruction box that can be transferred to NF‐κB, Bcl‐3 or p53.
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- Nucleic Acids Research, 1998, v. 26, n. 7, p. 1724, doi. 10.1093/nar/26.7.1724
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- Article
Post-transcriptional regulation of the let-7 microRNA during neural cell specification.
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- FASEB Journal, 2007, v. 21, n. 2, p. 415, doi. 10.1096/fj.06-6130com
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- Article
The RNA-binding protein ARPP21 controls dendritic branching by functionally opposing the miRNA it hosts.
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- Nature Communications, 2018, v. 9, n. 1, p. 1, doi. 10.1038/s41467-018-03681-3
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- Article
Regulation of miRNA expression during neural cell specification.
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- European Journal of Neuroscience, 2005, v. 21, n. 6, p. 1469, doi. 10.1111/j.1460-9568.2005.03978.x
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- Article