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β-Catenin activation synergizes with PTEN loss to cause bladder cancer formation.
Published in:
Oncogene, 2011, v. 30, n. 2, p. 178, doi. 10.1038/onc.2010.399
By:
- Ahmad, I;
- Morton, J P;
- Singh, L B;
- Radulescu, S M;
- Ridgway, R A;
- Patel, S;
- Woodgett, J;
- Winton, D J;
- Taketo, M M;
- Wu, X-R;
- Leung, H Y;
- Sansom, O J
Publication type:
Article
Conditional expression of mutated K-ras accelerates intestinal tumorigenesis in Msh2-deficient mice.
Published in:
Oncogene, 2007, v. 26, n. 30, p. 4415, doi. 10.1038/sj.onc.1210231
By:
- Luo, F.;
- Brooks, D. G.;
- Ye, H.;
- Hamoudi, R.;
- Poulogiannis, G.;
- Patek, C. E.;
- Winton, D. J.;
- Arends, M. J.
Publication type:
Article
A clonal marker induced by mutation in mouse intestinal epithelium.
Published in:
Nature, 1988, v. 333, n. 6172, p. 463, doi. 10.1038/333463a0
By:
- Winton, D. J.;
- Blount, M. A.;
- Ponder, B. A. J.
Publication type:
Article
Stem cells, quiescence and rectal carcinoma: an unexplored relationship and potential therapeutic target.
Published in:
2011
By:
- Buczacki, S;
- Davies, R J;
- Winton, D J
Publication type:
journal article
Wnt ligands influence tumour initiation by controlling the number of intestinal stem cells.
Published in:
Nature Communications, 2018, v. 9, n. 1, p. 1, doi. 10.1038/s41467-018-03426-2
By:
- Huels, D. J.;
- Bruens, L.;
- Hodder, M. C.;
- Cammareri, P.;
- Campbell, A. D.;
- Ridgway, R. A.;
- Gay, D. M.;
- Solar-Abboud, M.;
- Faller, W. J.;
- Nixon, C.;
- Zeiger, L. B.;
- McLaughlin, M. E.;
- Morrissey, E.;
- Winton, D. J.;
- Snippert, H. J.;
- van Rheenen, J.;
- Sansom, O. J.
Publication type:
Article

Found: 5

β-Catenin activation synergizes with PTEN loss to cause bladder cancer formation.

Conditional expression of mutated K-ras accelerates intestinal tumorigenesis in Msh2-deficient mice.

A clonal marker induced by mutation in mouse intestinal epithelium.

Stem cells, quiescence and rectal carcinoma: an unexplored relationship and potential therapeutic target.

Wnt ligands influence tumour initiation by controlling the number of intestinal stem cells.