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Salmonella manipulates the host to drive pathogenicity via induction of interleukin 1β.
- Published in:
- PLoS Biology, 2024, v. 22, n. 1, p. 1, doi. 10.1371/journal.pbio.3002486
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- Article
Colonocyte-derived lactate promotes E. coli fitness in the context of inflammation-associated gut microbiota dysbiosis.
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- Microbiome, 2022, v. 10, n. 1, p. 1, doi. 10.1186/s40168-022-01389-7
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- Article
Reshaping of bacterial molecular hydrogen metabolism contributes to the outgrowth of commensal E. coli during gut inflammation.
- Published in:
- eLife, 2021, p. 1, doi. 10.7554/eLife.58609
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- Article
Salmonella finds a way: Metabolic versatility of Salmonella enterica serovar Typhimurium in diverse host environments.
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- PLoS Pathogens, 2020, v. 16, n. 6, p. 1, doi. 10.1371/journal.ppat.1008540
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- Article
STAT2 dependent Type I Interferon response promotes dysbiosis and luminal expansion of the enteric pathogen Salmonella Typhimurium.
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- PLoS Pathogens, 2019, v. 15, n. 4, p. 1, doi. 10.1371/journal.ppat.1007745
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- Article
Respiration of Microbiota-Derived 1,2-propanediol Drives Salmonella Expansion during Colitis.
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- PLoS Pathogens, 2017, v. 13, n. 1, p. 1, doi. 10.1371/journal.ppat.1006129
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- Article
The Vi Capsular Polysaccharide Enables <i>Salmonella enterica</i> Serovar Typhi to Evade Microbe-Guided Neutrophil Chemotaxis.
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- PLoS Pathogens, 2014, v. 10, n. 8, p. 1, doi. 10.1371/journal.ppat.1004306
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- Article
<i>Salmonella enterica</i> Serovar Typhi Conceals the Invasion-Associated Type Three Secretion System from the Innate Immune System by Gene Regulation.
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- PLoS Pathogens, 2014, v. 10, n. 7, p. 1, doi. 10.1371/journal.ppat.1004207
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- Article
Why related bacterial species bloom simultaneously in the gut: principles underlying the 'Like will to like' concept.
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- Cellular Microbiology, 2014, v. 16, n. 2, p. 179, doi. 10.1111/cmi.12245
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- Article
Colonization Resistance: Battle of the Bugs or Ménage à Trois with the Host?
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- PLoS Pathogens, 2013, v. 9, n. 11, p. 1, doi. 10.1371/journal.ppat.1003730
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- Article
Manipulation of small Rho GTPases is a pathogen-induced process detected by NOD1.
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- Nature, 2013, v. 496, n. 7444, p. 233, doi. 10.1038/nature12025
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- Article
The dynamics of gut-associated microbial communities during inflammation.
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- EMBO Reports, 2013, v. 14, n. 4, p. 319, doi. 10.1038/embor.2013.27
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- Article
Salmonella Uses Energy Taxis to Benefit from Intestinal Inflammation.
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- PLoS Pathogens, 2013, v. 9, n. 4, p. 1, doi. 10.1371/journal.ppat.1003267
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- Article
Very Long O-antigen Chains Enhance Fitness during Salmonella-induced Colitis by Increasing Bile Resistance.
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- PLoS Pathogens, 2012, v. 8, n. 9, p. 1, doi. 10.1371/journal.ppat.1002918
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- Article
Temporal Expression of Bacterial Proteins Instructs Host CD4 T Cell Expansion and Th17 Development.
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- PLoS Pathogens, 2012, v. 8, n. 1, p. 1, doi. 10.1371/journal.ppat.1002499
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- Article
Intestinal and chronic infections: Salmonella lifestyles in hostile environments.
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- Environmental Microbiology Reports, 2011, v. 3, n. 5, p. 508, doi. 10.1111/j.1758-2229.2011.00242.x
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- Article
Gut inflammation provides a respiratory electron acceptor for Salmonella.
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- Nature, 2010, v. 467, n. 7314, p. 426, doi. 10.1038/nature09415
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- Article
The TviA auxiliary protein renders the Salmonella enterica serotype Typhi RcsB regulon responsive to changes in osmolarity.
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- Molecular Microbiology, 2009, v. 74, n. 1, p. 175, doi. 10.1111/j.1365-2958.2009.06859.x
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- Article
The Vi-capsule prevents Toll-like receptor 4 recognition of Salmonella.
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- Cellular Microbiology, 2008, v. 10, n. 4, p. 876, doi. 10.1111/j.1462-5822.2007.01090.x
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- Article
Simian immunodeficiency virus–induced mucosal interleukin-17 deficiency promotes Salmonella dissemination from the gut.
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- Nature Medicine, 2008, v. 14, n. 4, p. 421, doi. 10.1038/nm1743
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- Article
The Salmonella enterica serotype Typhi regulator TviA reduces interleukin-8 production in intestinal epithelial cells by repressing flagellin secretion.
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- Cellular Microbiology, 2008, v. 10, n. 1, p. 247, doi. 10.1111/j.1462-5822.2007.01037.x
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- Article