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Excess Rab4 rescues synaptic and behavioral dysfunction caused by defective HTT-Rab4 axonal transport in Huntington's disease.
- Published in:
- Acta Neuropathologica Communications, 2020, v. 8, n. 1, p. 1, doi. 10.1186/s40478-020-00964-z
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- Article
Axonal transport and neurodegenerative disease: vesicle-motor complex formation and their regulation.
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- Degenerative Neurological & Neuromuscular Disease, 2014, v. 4, p. 29, doi. 10.2147/DNND.S57502
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- Article
Axonal Transport and Neurodegeneration: How Marine Drugs Can Be Used for the Development of Therapeutics.
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- Marine Drugs, 2016, v. 14, n. 5, p. 102, doi. 10.3390/md14050102
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- Article
Amyloid precursor protein–mediated endocytic pathway disruption induces axonal dysfunction and neurodegeneration.
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- Journal of Clinical Investigation, 2016, v. 126, n. 5, p. 1815, doi. 10.1172/JCI82409
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- Article
Huntingtin differentially regulates the axonal transport of a sub-set of Rab-containing vesicles in vivo.
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- Human Molecular Genetics, 2015, v. 24, n. 25, p. 7182, doi. 10.1093/hmg/ddv415
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- Article
Excess active P13K rescues huntingtin-mediated neuronal cell death but has no effect on axonal transport defects.
- Published in:
- Apoptosis, 2019, v. 24, n. 3/4, p. 341, doi. 10.1007/s10495-019-01520-4
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- Article