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In Utero Exposure of Female CD-1 Mice to AZT and/or 3TC: II. Persistence of Functional Alterations in Cardiac Tissue.
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- Cardiovascular Toxicology, 2010, v. 10, n. 2, p. 87, doi. 10.1007/s12012-010-9065-z
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- Article
In Utero Exposure of Female CD-1 Mice to AZT and/or 3TC: I. Persistence of Microscopic Lesions in Cardiac Tissue.
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- Cardiovascular Toxicology, 2010, v. 10, n. 1, p. 37, doi. 10.1007/s12012-010-9061-3
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- Article
WR1065 conjugated to thiol-PEG polymers as novel anticancer prodrugs: broad spectrum efficacy, synergism, and drug resistance reversal.
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- Frontiers in Oncology, 2023, p. 1, doi. 10.3389/fonc.2023.1212604
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Clonality, trafficking, and molecular alterations among Hprt mutant T lymphocytes isolated from control mice versus mice treated with N‐ethyl‐N‐nitrosourea.
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- Environmental & Molecular Mutagenesis, 2023, v. 64, n. 8/9, p. 432, doi. 10.1002/em.22579
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Human blood PIG‐A mutation and micronucleated reticulocyte flow cytometric assays: Method optimization and evaluation of intra‐ and inter‐subject variation.
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- Environmental & Molecular Mutagenesis, 2020, v. 61, n. 8, p. 807, doi. 10.1002/em.22393
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Biomarkers of exposure and effect as indicators of potential carcinogenic risk arising from in vivo metabolism of ethylene to ethylene oxide.
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- Carcinogenesis, 2000, v. 21, n. 9, p. 1661, doi. 10.1093/carcin/21.9.1661
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- Article
Molecular dosimetry of endogenous and ethylene oxide-induced N7-(2-hydroxyethyl) guanine formation in tissues of rodents.
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- Carcinogenesis, 1999, v. 20, n. 9, p. 1787, doi. 10.1093/carcin/20.9.1787
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The stress response resolution assay. I. Quantitative assessment of environmental agent/condition effects on cellular stress resolution outcomes in epithelium.
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- Environmental & Molecular Mutagenesis, 2013, v. 54, n. 4, p. 268, doi. 10.1002/em.21772
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- Article
The stress response resolution assay. II. Quantitative assessment of environmental agent/condition effects on cellular stress resolution outcomes in epithelium.
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- Environmental & Molecular Mutagenesis, 2013, v. 54, n. 4, p. 281, doi. 10.1002/em.21771
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- Article
WR1065 Mitigates AZT-ddI-Induced Mutagenesis and Inhibits Viral Replication.
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- Environmental & Molecular Mutagenesis, 2009, v. 50, n. 6, p. 460, doi. 10.1002/em.20482
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Special issue on health risks of perinatal exposure to nucleoside reverse transcriptase inhibitorsInvited article on the genotoxicity of perinatal NRTI therapy.
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- Environmental & Molecular Mutagenesis, 2007, v. 48, n. 3/4, p. 159, doi. 10.1002/em.20296
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Plasma and cellular markers of 3′‐azido‐3′‐dideoxythymidine (AZT) metabolism as indicators of DNA damage in cord blood mononuclear cells from infants receiving prepartum NRTIsInvited article on the genotoxicity of perinatal NRTI therapy.Contributing investigators at clinical sites for the protocol entitled “A study of the potential mutagenicity of ZDV/nucleoside analogues in perinatal HIV prophylaxis” were Alan Bateman (Harlem Hospital Center, New York, NY), Eleanor Jimenez and Jorge Gandia (Centro Medico, San Juan, Puerto Rico), Renee Samelson (Albany Medical Center, Albany, NY), Sohail Rana (Howard University Hospital, Washington, DC), Andrea Kovacs and Alice Stek (University of Southern California Medical Center, Los Angeles, CA), and Mobeen H. Rathore and Isaac Delke (University of Florida Health Science Center, Jacksonville, FL).
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- Environmental & Molecular Mutagenesis, 2007, v. 48, n. 3/4, p. 307
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- Article
Transplacental carcinogenicity of 3′‐azido‐3′‐deoxythymidine in B6C3F1 mice and F344 ratsInvited article on the genotoxicity of perinatal NTRI therapy.
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- Environmental & Molecular Mutagenesis, 2007, v. 48, n. 3/4, p. 283, doi. 10.1002/em.20297
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Genotoxicity assessed by the comet and GPA assays following in vitro exposure of human lymphoblastoid cells (H9) or perinatal exposure of mother–child pairs to AZT or AZT‐3TCInvited article on the genotoxicity of perinatal NRTI therapy.
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- Environmental & Molecular Mutagenesis, 2007, v. 48, n. 3/4, p. 330
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Relative mutagenic potencies of several nucleoside analogs, alone or in drug pairs, at the HPRT and TK loci of human TK6 lymphoblastoid cellsInvited article on the genotoxicity of perinatal NRTI therapy.The contents of this communication are the sole responsibility of the authors and do not necessarily represent the official views of the NCI, NHLBI, or NIH.
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- Environmental & Molecular Mutagenesis, 2007, v. 48, n. 3/4, p. 239, doi. 10.1002/em.20282
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Mutagenicity of zidovudine, lamivudine, and abacavir following in vitro exposure of human lymphoblastoid cells or in utero exposure of CD‐1 mice to single agents or drug combinationsThe contents of this communication are the sole responsibility of the authors and do not necessarily represent official views of the NCI or the NIH.Invited article on the genotoxicity of perinatal NRTI therapy.
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- Environmental & Molecular Mutagenesis, 2007, v. 48, n. 3/4, p. 224, doi. 10.1002/em.20264
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Transplacentally exposed human and monkey newborn infants show similar evidence of nucleoside reverse transcriptase inhibitor‐induced mitochondrial toxicityInvited article on the genotoxicity of perinatal NRTI therapy.
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- Environmental & Molecular Mutagenesis, 2007, v. 48, n. 3/4, p. 201, doi. 10.1002/em.20201
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Mitochondrial toxicity in hearts of CD‐1 mice following perinatal exposure to AZT, 3TC, or AZT/3TC in combinationThis article is a US Government work and, as such, is in the public domain in the United States of America.Invited article on the genotoxicity of perinatal NRTI therapy.
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- Environmental & Molecular Mutagenesis, 2007, v. 48, n. 3/4, p. 190, doi. 10.1002/em.20191
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Hprt mutant frequencies in splenic T-cells of male F344 rats exposed by inhalation to propylene.
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- Environmental & Molecular Mutagenesis, 2004, v. 43, n. 4, p. 265
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- Article
Characterization of Hprt mutations in cDNA and genomic DNA of T-cell mutants from control and 1,3-butadiene-exposed male B6C3F1 mice and F344 rats.
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- Environmental & Molecular Mutagenesis, 2004, v. 43, n. 2, p. 75
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Molecular analysis of mutations at the HPRT and TK loci of human lymphoblastoid cells after combined treatments with 3′-azido-3′-deoxythymidine and 2′,3′-dideoxyinosine†.
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- Environmental & Molecular Mutagenesis, 2002, v. 39, n. 4, p. 282, doi. 10.1002/em.10073
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Transplacental mutagenicity of N-ethyl- N-nitrosourea at the hprt locus in T-lymphocytes of exposed B6C3F1 mice.
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- Environmental & Molecular Mutagenesis, 2001, v. 38, n. 1, p. 30, doi. 10.1002/em.1047
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Detection of cyclophosphamide-induced mutations at the Hprt but not the lacI locus in splenic lymphocytes of exposed mice.
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- Environmental & Molecular Mutagenesis, 1999, v. 34, n. 2/3, p. 167, doi. 10.1002/(SICI)1098-2280(1999)34:2/3<167::AID-EM16>3.0.CO;2-O
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Tissue-specific mutant frequencies and mutational spectra in cyclophosphamide-treated lacI transgenic mice.
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- Environmental & Molecular Mutagenesis, 1999, v. 34, n. 2/3, p. 154, doi. 10.1002/(SICI)1098-2280(1999)34:2/3<154::AID-EM15>3.0.CO;2-0
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Culture and propagation of Hprt mutant T-lymphocytes isolated from mouse spleen.
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- Environmental & Molecular Mutagenesis, 1998, v. 32, n. 3, p. 236, doi. 10.1002/(SICI)1098-2280(1998)32:3<236::AID-EM6>3.0.CO;2-Y
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Bis-butanediol-mercapturic acid (bis-BDMA) as a urinary biomarker of metabolic activation of butadiene to its ultimate carcinogenic species.
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- Carcinogenesis, 2014, v. 35, n. 6, p. 1, doi. 10.1093/carcin/bgu047
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Evaluation of Effects from Repeated Inhalation Exposure of F344 Rats to High Concentrations of Propylene.
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- Toxicological Sciences, 2007, v. 97, n. 2, p. 336, doi. 10.1093/toxsci/kfm038
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Carcinogenicity. Relationships between DNA Incorporation, Mutant Frequency, and Loss of Heterozygosity at the TK Locus in Human Lymphoblastoid Cells Exposed to 3′-Azido-3′-deoxythymidine.
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- Toxicological Sciences, 2000, v. 54, n. 2, p. 322, doi. 10.1093/toxsci/54.2.322
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Mutagenicity and loss of heterozygosity at the APRT locus in human lymphoblastoid cells exposed to 3′-azido-3′-deoxythymidine.
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- Mutagenesis, 2000, v. 15, n. 5, p. 405, doi. 10.1093/mutage/15.5.405
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Exposure-Response of 1,2:3,4-Diepoxybutane–Specific N-Terminal Valine Adducts in Mice and Rats after Inhalation Exposure to 1,3-Butadiene.
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- Toxicological Sciences, 2010, v. 115, n. 2, p. 322, doi. 10.1093/toxsci/kfq060
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- Article