Found: 21
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Annexin A1 Deficiency does not Affect Myofiber Repair but Delays Regeneration of Injured Muscles.
- Published in:
- Scientific Reports, 2015, p. 18246, doi. 10.1038/srep18246
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- Article
Effects of Dantrolene Therapy on Disease Phenotype in Dystrophin Deficient mdx Mice.
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- PLoS Currents, 2013, p. 218, doi. 10.1371/currents.md.e246cf493a7edb1669f42fb735936b46
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- Article
Termino-lateral neurorrhaphy: The functional axonal anatomy.
- Published in:
- Microsurgery, 2000, v. 20, n. 1, p. 6, doi. 10.1002/(SICI)1098-2752(2000)20:1<6::AID-MICR2>3.0.CO;2-5
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- Article
Long-term treatment with naproxcinod significantly improves skeletal and cardiac disease phenotype in the mdx mouse model of dystrophy.
- Published in:
- Human Molecular Genetics, 2014, v. 23, n. 12, p. 3239, doi. 10.1093/hmg/ddu033
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- Article
Mitochondrial redox signaling enables repair of injured skeletal muscle cells.
- Published in:
- Science Signaling, 2017, v. 10, n. 495, p. 1, doi. 10.1126/scisignal.aaj1978
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- Article
Mitochondrial dysfunction and consequences in calpain-3-deficient muscle.
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- Skeletal Muscle, 2020, v. 10, n. 1, p. 1, doi. 10.1186/s13395-020-00254-1
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- Article
Motor Unit Changes Seen With Skeletal Muscle Sarcopenia in Oldest Old Rats.
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- Journals of Gerontology Series A: Biological Sciences & Medical Sciences, 2014, v. 69, n. 6, p. 657, doi. 10.1093/gerona/glt135
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- Article
Lateral transmission of force is impaired in skeletal muscles of dystrophic mice and very old rats.
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- Journal of Physiology, 2011, v. 589, n. 5, p. 1195, doi. 10.1113/jphysiol.2010.201921
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- Article
VBP15, a novel anti-inflammatory and membrane-stabilizer, improves muscular dystrophy without side effects.
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- EMBO Molecular Medicine, 2013, v. 5, n. 10, p. 1569, doi. 10.1002/emmm.201302621
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- Article
The Proton Pump Inhibitor Lansoprazole Improves the Skeletal Phenotype in Dystrophin Deficient <i>mdx</i> Mice.
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- PLoS ONE, 2013, v. 8, n. 7, p. 1, doi. 10.1371/journal.pone.0066617
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- Article
Daily Supplementation of D-ribose Shows No Therapeutic Benefits in the MHC-I Transgenic Mouse Model of Inflammatory Myositis.
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- PLoS ONE, 2013, v. 8, n. 6, p. 1, doi. 10.1371/journal.pone.0065970
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- Article
Characterization of Dysferlin Deficient SJL/J Mice to Assess Preclinical Drug Efficacy: Fasudil Exacerbates Muscle Disease Phenotype.
- Published in:
- PLoS ONE, 2010, v. 5, n. 9, p. 1, doi. 10.1371/journal.pone.0012981
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- Article
Selective modulation through the glucocorticoid receptor ameliorates muscle pathology in mdx mice.
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- Journal of Pathology, 2013, v. 231, n. 2, p. 223, doi. 10.1002/path.4231
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- Article
Eps homology domain endosomal transport proteins differentially localize to the neuromuscular junction.
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- Skeletal Muscle, 2012, v. 2, n. 1, p. 19, doi. 10.1186/2044-5040-2-19
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- Article
Metabolic remodeling agents show beneficial effects in the dystrophin-deficient mdx mouse model.
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- Skeletal Muscle, 2012, v. 2, n. 1, p. 16, doi. 10.1186/2044-5040-2-16
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- Article
Annexin A2 links poor myofiber repair with inflammation and adipogenic replacement of the injured muscle.
- Published in:
- Human Molecular Genetics, 2017, v. 26, n. 11, p. 1979, doi. 10.1093/hmg/ddx065
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- Article
Effect of genetic background on the dystrophic phenotype in mdx mice.
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- Human Molecular Genetics, 2016, v. 25, n. 1, p. 130, doi. 10.1093/hmg/ddv460
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- Article
Preclinical drug trials in the mdx mouse: Assessment of reliable and sensitive outcome measures.
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- Muscle & Nerve, 2009, v. 39, n. 5, p. 591, doi. 10.1002/mus.21211
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- Article
The age-related failure of adaptive responses to contractile activity in skeletal muscle is mimicked in young mice by deletion of Cu,Zn superoxide dismutase A. Vasilaki et al. Age-related failure of adaptive responses.
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- Aging Cell, 2010, v. 9, n. 6, p. 979, doi. 10.1111/j.1474-9726.2010.00635.x
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- Article
Effect of the IL-1 Receptor Antagonist Kineret<sup>®</sup> on Disease Phenotype in mdx Mice.
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- PLoS ONE, 2016, v. 11, n. 5, p. 1, doi. 10.1371/journal.pone.0155944
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- Article
Membrane Sealant Poloxamer P188 Protects Against Isoproterenol Induced Cardiomyopathy in Dystrophin Deficient Mice.
- Published in:
- BMC Cardiovascular Disorders, 2011, v. 11, n. 1, p. 20, doi. 10.1186/1471-2261-11-20
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- Article