Found: 13
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β-Amyloid Oligomers Induce Phosphorylation of Tau and Inactivation of Insulin Receptor Substrate via c-Jun N-Terminal Kinase Signaling: Suppression by Omega-3 Fatty Acids and Curcumin.
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- Journal of Neuroscience, 2009, v. 29, n. 28, p. 9078, doi. 10.1523/JNEUROSCI.1071-09.2009
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- Article
Omega-3 Fatty Acid Docosahexaenoic Acid Increases SorLA/LR11, a Sorting Protein with Reduced Expression in Sporadic Alzheimer's Disease (AD): Relevance to AD Prevention.
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- Journal of Neuroscience, 2007, v. 27, n. 52, p. 14299, doi. 10.1523/JNEUROSCI.3593-07.2007
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- Article
Evidence of Aβ- and transgene-dependent defects in ERK-CREB signaling in Alzheimer’s models.
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- Journal of Neurochemistry, 2007, v. 103, n. 4, p. 1594, doi. 10.1111/j.1471-4159.2007.04869.x
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- Article
Antibodies against β‐amyloid reduce aβ oligomers, glycogen synthase kinase‐3β activation and τ phosphorylation in vivo and in vitro.
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- Journal of Neuroscience Research, 2006, v. 83, n. 3, p. 374
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- Article
Loss of MAP Function Leads to Hippocampal Synapse Loss and Deficits in the Morris Water Maze with Aging.
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- Journal of Neuroscience, 2014, v. 34, n. 21, p. 7124, doi. 10.1523/JNEUROSCI.3439-13.2014
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- Article
Protection of excitatory synapses by DHA through preserving function of NMDA-Tiam1-PAK signal pathway in Alzheimer models
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- 2010
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- Abstract
P4-272: P21-activated kinase aberrant activation and translocation in Alzheimer's disease pathogenesis
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- 2008
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- Abstract
P3-064: Can SorLA/LR11, a sorting protein limiting Abeta production, serve as both therapeutic and diagnostic target in Alzheimer's disease?
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- 2008
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- Abstract
O3-02-05: p21-activated kinase pathway defects and synaptic function in Alzheimer’s disease
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- 2006
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- Abstract
O3-02-05: p21-activated kinase pathway defects and synaptic function in Alzheimer’s disease
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- 2006
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- Abstract
Insulin-Degrading Enzyme as a Downstream Target of Insulin Receptor Signaling Cascade: Implications for Alzheimer's Disease Intervention.
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- Journal of Neuroscience, 2004, v. 24, n. 49, p. 11120, doi. 10.1523/JNEUROSCI.2860-04.2004
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- Article
Use of Copper and Insulin-Resistance to Accelerate Cognitive Deficits and Synaptic Protein Loss in a Rat Aβ-Infusion Alzheimer's Disease Model.
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- Journal of Alzheimer's Disease, 2008, v. 15, n. 4, p. 625, doi. 10.3233/JAD-2008-15409
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- Article
Role of p21-activated kinase pathway defects in the cognitive deficits of Alzheimer disease.
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- Nature Neuroscience, 2006, v. 9, n. 2, p. 234, doi. 10.1038/nn1630
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- Article