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Animal and Cellular Models of Alzheimer's Disease: Progress, Promise, and Future Approaches.
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- Neuroscientist, 2022, v. 28, n. 6, p. 572, doi. 10.1177/10738584211001753
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- Article
Calretinin Interneurons are Early Targets of Extracellular Amyloid-β Pathology in PS1/AβPP Alzheimer Mice Hippocampus.
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- Journal of Alzheimer's Disease, 2010, v. 21, n. 1, p. 119, doi. 10.3233/JAD-2010-100066
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- Article
Extracellular Amyloid-β and Cytotoxic Glial Activation Induce Significant Entorhinal Neuron Loss in Young PS1^{M146L}/APP^{751SL} Mice.
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- Journal of Alzheimer's Disease, 2009, v. 18, n. 4, p. 755, doi. 10.3233/JAD-2009-1192
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- Article
Transgenic Mouse Models of Alzheimer's Disease: An Integrative Analysis.
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- International Journal of Molecular Sciences, 2022, v. 23, n. 10, p. 5404, doi. 10.3390/ijms23105404
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- Article
Soluble phospho-tau from Alzheimer's disease hippocampus drives microglial degeneration.
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- Acta Neuropathologica, 2016, v. 132, n. 6, p. 897, doi. 10.1007/s00401-016-1630-5
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- Article
Abnormal accumulation of autophagic vesicles correlates with axonal and synaptic pathology in young Alzheimer's mice hippocampus.
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- Acta Neuropathologica, 2012, v. 123, n. 1, p. 53, doi. 10.1007/s00401-011-0896-x
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- Article
Defective lysosomal proteolysis and axonal transport are early pathogenic events that worsen with age leading to increased APP metabolism and synaptic Abeta in transgenic APP/PS1 hippocampus.
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- Molecular Neurodegeneration, 2012, v. 7, n. 1, p. 1, doi. 10.1186/1750-1326-7-59
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- Article
Amyloid propagation in a sporadic model of Alzheimer's disease: Development of new models and analysis methods/seeding and spreading of proteinopathies.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2020, v. 16, n. 11, p. 1, doi. 10.1002/alz.045657
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- Article
P4‐522: TYPE 2 DIABETES MELLITUS INDUCES TAU‐INDEPENDENT COGNITIVE AND SYNAPTIC DEFICITS IN A MOUSE MODEL.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2019, v. 15, p. P1514, doi. 10.1016/j.jalz.2019.08.069
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- Article
P3‐069: HUMAN WILD TYPE Aβ KNOCK‐IN MICE AS A BASIS TO STUDY SPORADIC ALZHEIMER'S DISEASE.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2019, v. 15, p. P953, doi. 10.1016/j.jalz.2019.06.3096
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- Article
P3‐173: IMPACT OF SYNAPTIC REGULATORS' LOSS ON ALZHEIMER'S DISEASE.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2018, v. 14, p. P1134, doi. 10.1016/j.jalz.2018.06.1531
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- Article
P2‐172: THE DYSREGULATION OF TOM1 IN ALZHEIMER'S DISEASE.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2018, v. 14, p. P734, doi. 10.1016/j.jalz.2018.06.859
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- Article
O1‐01‐04: HAβ‐KI: A KNOCK‐IN MOUSE MODEL FOR SPORADIC ALZHEIMER'S DISEASE.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2018, v. 14, p. P213, doi. 10.1016/j.jalz.2018.06.2333
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- Article
APPKI-HaβWT: A NOVEL TRANSGENIC MOUSE TO MODEL SPORADIC ALZHEIMER’S DISEASE.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2017, v. 13, p. P281, doi. 10.1016/j.jalz.2017.06.174
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- Article
Different activated microglial response in human hippocampus during Alzheimer's disease progression
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- 2012
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- Abstract
Cholinergic degenerative pathology in ps1m146l/APP751sl alzheimer transgenic model
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- 2011
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- Abstract
Degeneration of calretinin interneurons is associated to the early onset of extracellular amyloid pathology in Alzheimer mice hippocampus
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- 2010
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- Abstract
Early synaptic degenerative changes in the hippocampus of PS1(M146L)xAPP(751SL) mouse model of Alzheimer's disease
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- 2009
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- Abstract
Extracellular amyloid-beta pathology induces entorhinal neurodegeneration in PS1(M146L)xAPP(751SL) mouse model of Alzheimer's disease
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- 2009
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- Abstract
P3-375: Phenotypic and functional switch in microglial cells correlates with neurodegeneration in the hippocampus of aged PS1xAPP transgenic model of Alzheimer's disease
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- 2008
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- Abstract
Monocyte-derived cells invade brain parenchyma and amyloid plaques in human Alzheimer's disease hippocampus.
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- Acta Neuropathologica Communications, 2023, v. 11, n. 1, p. 1, doi. 10.1186/s40478-023-01530-z
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- Article
Intra- and extracellular β-amyloid overexpression via adeno-associated virus-mediated gene transfer impairs memory and synaptic plasticity in the hippocampus.
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- Scientific Reports, 2019, v. 9, n. 1, p. N.PAG, doi. 10.1038/s41598-019-52324-0
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- Article
Generation of a humanized Aβ expressing mouse demonstrating aspects of Alzheimer's disease-like pathology.
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- Nature Communications, 2021, v. 12, n. 1, p. 1, doi. 10.1038/s41467-021-22624-z
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- Article
Plaque-Associated Oligomeric Amyloid-Beta Drives Early Synaptotoxicity in APP/PS1 Mice Hippocampus: Ultrastructural Pathology Analysis.
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- Frontiers in Neuroscience, 2021, v. 15, p. 1, doi. 10.3389/fnins.2021.752594
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- Article
Phagocytic clearance of presynaptic dystrophies by reactive astrocytes in Alzheimer's disease.
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- Glia, 2018, v. 66, n. 3, p. 637, doi. 10.1002/glia.23270
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- Article
Distinct disease‐sensitive GABAergic neurons in the perirhinal cortex of Alzheimer's mice and patients.
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- Brain Pathology, 2020, v. 30, n. 2, p. 345, doi. 10.1111/bpa.12785
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- Article
Adipose tissue as a therapeutic target for vascular damage in Alzheimer's disease.
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- British Journal of Pharmacology, 2024, v. 181, n. 6, p. 840, doi. 10.1111/bph.16243
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- Article
miR‐181a negatively modulates synaptic plasticity in hippocampal cultures and its inhibition rescues memory deficits in a mouse model of Alzheimer's disease.
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- Aging Cell, 2020, v. 19, n. 3, p. 1, doi. 10.1111/acel.13118
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- Article
Tau underlies synaptic and cognitive deficits for type 1, but not type 2 diabetes mouse models.
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- Aging Cell, 2019, v. 18, n. 3, p. 1, doi. 10.1111/acel.12919
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- Article
Impaired AMPA signaling and cytoskeletal alterations induce early synaptic dysfunction in a mouse model of Alzheimer's disease.
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- Aging Cell, 2018, v. 17, n. 4, p. 1, doi. 10.1111/acel.12791
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- Article
Visceral adipose tissue triggers tau pathogenesis in transgenic mice.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2023, v. 19, p. 1, doi. 10.1002/alz.076287
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- Article
Neurons and astrocytes derived from human iPSCs to model Alzheimer´s disease.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2023, v. 19, p. 1, doi. 10.1002/alz.074740
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- Article
Human and mouse seeds differentially affect Aβ aggregation by modulating the inflammatory response.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2023, v. 19, p. 1, doi. 10.1002/alz.074787
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- Article
MONOCYTE‐DERIVED CELLS INVADE AMYLOID PLAQUES IN HUMAN ALZHEIMER'S DISEASE HIPPOCAMPUS.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2023, v. 19, p. 1, doi. 10.1002/alz.074574
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- Article
Human and mouse seeds differentially affect Aβ aggregation by modulating the inflammatory response.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2023, v. 19, p. 1, doi. 10.1002/alz.074787
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- Publication type:
- Article
MONOCYTE‐DERIVED CELLS INVADE AMYLOID PLAQUES IN HUMAN ALZHEIMER'S DISEASE HIPPOCAMPUS.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2023, v. 19, p. 1, doi. 10.1002/alz.074574
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- Article
Fat and the brain: its connection and implication in AD pathogenesis.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2023, v. 19, p. 1, doi. 10.1002/alz.062317
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- Article
Human amyloid seeds induce a more profound pathology than seeds from old 3x‐Tg‐AD mice.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2023, v. 19, p. 1, doi. 10.1002/alz.062277
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- Article
Peroxisome Proliferator‐Activated Receptor γ is involved in the neuroprotection exerted by 2‐hydroxydocosahexaenoic acid against Alzheimer's disease.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2021, v. 17, p. 1, doi. 10.1002/alz.054514
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- Article
Amyloid propagation in a sporadic model of Alzheimer disease.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2021, v. 17, n. 3, p. 1, doi. 10.1002/alz.051813
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- Article