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CD36/Fatty Acid Translocase, An Inflammatory Mediator, Is Involved in Hyperlipidemia-Induced Exacerbation in Ischemic Brain Injury.
- Published in:
- Journal of Neuroscience, 2008, v. 28, n. 18, p. 4661, doi. 10.1523/JNEUROSCI.0982-08.2008
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- Publication type:
- Article
Pulse Inhibition of Histone Deacetylases Induces Complete Resistance to Oxidative Death in Cortical Neurons without Toxicity and Reveals a Role for Cytoplasmic p21<sup>waf1/cip1</sup> in Cell Cycle-Independent Neuroprotection.
- Published in:
- Journal of Neuroscience, 2008, v. 27, n. 53, p. 163, doi. 10.1523/JNEUROSCI.3200-07.2008
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- Article
Deregulation of inflammatory response in the diabetic condition is associated with increased ischemic brain injury.
- Published in:
- Journal of Neuroinflammation, 2014, v. 11, n. 1, p. 1, doi. 10.1186/1742-2094-11-83
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- Article
Deregulation of inflammatory response in the diabetic condition is associated with increased ischemic brain injury.
- Published in:
- 2014
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- Publication type:
- journal article
Targeting CD36-Mediated Inflammation Reduces Acute Brain Injury in Transient, but not Permanent, Ischemic Stroke.
- Published in:
- CNS Neuroscience & Therapeutics, 2015, v. 21, n. 4, p. 385, doi. 10.1111/cns.12326
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- Publication type:
- Article
CD36 in the periphery and brain synergizes in stroke injury in hyperlipidemia.
- Published in:
- Annals of Neurology, 2012, v. 71, n. 6, p. 753, doi. 10.1002/ana.23569
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- Publication type:
- Article