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Achyranthes bidentata polysaccharide suppresses osteoclastogenesis and bone resorption via inhibiting RANKL signaling.
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- Journal of Cellular Biochemistry, 2018, v. 119, n. 6, p. 4826, doi. 10.1002/jcb.26682
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- Article
Bafilomycin A1 Attenuates Osteoclast Acidification and Formation, Accompanied by Increased Levels of SQSTM1/p62 Protein.
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- Journal of Cellular Biochemistry, 2016, v. 117, n. 6, p. 1464, doi. 10.1002/jcb.25442
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- Article
A Machine Learning-Based Image Segmentation Method to Quantify In Vitro Osteoclast Culture Endpoints.
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- Calcified Tissue International, 2023, v. 113, n. 4, p. 437, doi. 10.1007/s00223-023-01121-z
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- Article
PKC-δ deficiency in B cells displays osteopenia accompanied with upregulation of RANKL expression and osteoclast–osteoblast uncoupling.
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- Cell Death & Disease, 2020, v. 11, n. 9, p. 1, doi. 10.1038/s41419-020-02947-3
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- Article
HtrA1 is upregulated during RANKL-induced osteoclastogenesis, and negatively regulates osteoblast differentiation and BMP2-induced Smad1/5/8, ERK and p38 phosphorylation.
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- FEBS Letters, 2014, v. 588, n. 1, p. 143, doi. 10.1016/j.febslet.2013.11.022
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Expression Quantitative Trait Locus Study of Bone Mineral Density GWAS Variants in Human Osteoclasts.
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- Journal of Bone & Mineral Research, 2018, v. 33, n. 6, p. 1044, doi. 10.1002/jbmr.3412
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- Article
Dihydroartemisinin, an Anti-Malaria Drug, Suppresses Estrogen Deficiency-Induced Osteoporosis, Osteoclast Formation, and RANKL-Induced Signaling Pathways.
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- Journal of Bone & Mineral Research, 2016, v. 31, n. 5, p. 964, doi. 10.1002/jbmr.2771
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- Article
Cumambrin A prevents OVX-induced osteoporosis via the inhibition of osteoclastogenesis, bone resorption, and RANKL signaling pathways.
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- FASEB Journal, 2019, v. 33, n. 6, p. 6726, doi. 10.1096/fj.201800883RRR
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- Article
Genetic regulatory mechanisms in human osteoclasts suggest a role for the STMP1 and DCSTAMP genes in Paget's disease of bone.
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- Scientific Reports, 2019, v. 9, n. 1, p. 1, doi. 10.1038/s41598-018-37609-0
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Characterisation of genetic regulatory effects for osteoporosis risk variants in human osteoclasts.
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- Genome Biology, 2020, v. 21, n. 1, p. 1, doi. 10.1186/s13059-020-01997-2
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- Article
Bajijiasu Abrogates Osteoclast Differentiation via the Suppression of RANKL Signaling Pathways through NF-κB and NFAT.
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- International Journal of Molecular Sciences, 2017, v. 18, n. 1, p. 203, doi. 10.3390/ijms18010203
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- Article
Berberine Sulfate Attenuates Osteoclast Differentiation through RANKL Induced NF-κB and NFAT Pathways.
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- International Journal of Molecular Sciences, 2015, v. 16, n. 11, p. 27087, doi. 10.3390/ijms161125998
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- Article
Natural Germacrane Sesquiterpenes Inhibit Osteoclast Formation, Bone Resorption, RANKL-Induced NF-κB Activation, and IκBα Degradation.
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- International Journal of Molecular Sciences, 2015, v. 16, n. 11, p. 26599, doi. 10.3390/ijms161125972
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- Article
Asiatic Acid Inhibits OVX-Induced Osteoporosis and Osteoclastogenesis Via Regulating RANKL-Mediated NF-κb and Nfatc1 Signaling Pathways.
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- Frontiers in Pharmacology, 2020, p. N.PAG, doi. 10.3389/fphar.2020.00331
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- Article
Fumitremorgin C Attenuates Osteoclast Formation and Function via Suppressing RANKL-Induced Signaling Pathways.
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- Frontiers in Pharmacology, 2020, p. 1, doi. 10.3389/fphar.2020.00238
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- Article
Age-dependent genetic regulation of osteoarthritis: independent effects of immune system genes.
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- Arthritis Research & Therapy, 2023, v. 25, n. 1, p. 1, doi. 10.1186/s13075-023-03216-2
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- Article
Loss of Protein Kinase C-δ Protects against LPS-Induced Osteolysis Owing to an Intrinsic Defect in Osteoclastic Bone Resorption.
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- PLoS ONE, 2013, v. 8, n. 8, p. 1, doi. 10.1371/journal.pone.0070815
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Prolactin Expression in the Cochlea of Aged BALB/c Mice Is Gender Biased and Correlates to Loss of Bone Mineral Density and Hearing Loss
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- PLoS ONE, 2013, v. 8, n. 5, p. 1, doi. 10.1371/journal.pone.0063952
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Calmodulin interacts with Rab3D and modulates osteoclastic bone resorption.
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- Scientific Reports, 2016, p. 37963, doi. 10.1038/srep37963
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Corrigendum: NPNT is Expressed by Osteoblasts and Mediates Angiogenesis via the Activation of Extracellular Signal-regulated Kinase.
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- Scientific Reports, 2016, p. 37482, doi. 10.1038/srep37482
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- Article
Nitidine chloride prevents OVX-induced bone loss via suppressing NFATc1-mediated osteoclast differentiation.
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- Scientific Reports, 2016, p. 36662, doi. 10.1038/srep36662
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- Article
NPNT is Expressed by Osteoblasts and Mediates Angiogenesis via the Activation of Extracellular Signal-regulated Kinase.
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- Scientific Reports, 2016, p. 36210, doi. 10.1038/srep36210
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- Article
Morc3 mutant mice exhibit reduced cortical area and thickness, accompanied by altered haematopoietic stem cells niche and bone cell differentiation.
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- Scientific Reports, 2016, p. 25964, doi. 10.1038/srep25964
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- Article
The bone marrow microenvironment of pre-B acute lymphoblastic leukemia at single-cell resolution.
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- Scientific Reports, 2020, v. 10, n. 1, p. N.PAG, doi. 10.1038/s41598-020-76157-4
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- Article
Protein kinase C delta null mice exhibit structural alterations in articular surface, intra-articular and subchondral compartments.
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- Arthritis Research & Therapy, 2015, v. 17, n. 1, p. 1, doi. 10.1186/s13075-015-0720-4
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- Article
Protein kinase C delta null mice exhibit structural alterations in articular surface, intra-articular and subchondral compartments.
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- Arthritis Research & Therapy, 2015, v. 17, n. 1, p. 210, doi. 10.1186/s13075-015-0720-4
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- Article
Advanced Genetic Approaches in Discovery and Characterization of Genes Involved With Osteoporosis in Mouse and Human.
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- Frontiers in Genetics, 2019, p. N.PAG, doi. 10.3389/fgene.2019.00288
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Osteoblast-derived EGFL6 couples angiogenesis to osteogenesis during bone repair.
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- Theranostics, 2021, v. 11, n. 20, p. 9738, doi. 10.7150/thno.60902
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A missense mutation sheds light on a novel structure–function relationship of RANKL.
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- Journal of Cellular Physiology, 2021, v. 236, n. 4, p. 2800, doi. 10.1002/jcp.30045
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The emerging roles of hnRNPK.
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- Journal of Cellular Physiology, 2020, v. 235, n. 3, p. 1995, doi. 10.1002/jcp.29186
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- Article
Cajaninstilbene acid inhibits osteoporosis through suppressing osteoclast formation and RANKL‐induced signaling pathways.
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- Journal of Cellular Physiology, 2019, v. 234, n. 7, p. 11792, doi. 10.1002/jcp.27868
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- Article
Helvolic acid attenuates osteoclast formation and function via suppressing RANKL‐induced NFATc1 activation.
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- Journal of Cellular Physiology, 2019, v. 234, n. 5, p. 6477, doi. 10.1002/jcp.27385
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- Article
Asiaticoside, a component of Centella asiatica attenuates RANKL‐induced osteoclastogenesis via NFATc1 and NF‐κB signaling pathways.
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- Journal of Cellular Physiology, 2019, v. 234, n. 4, p. 4267, doi. 10.1002/jcp.27195
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Modulating calcium‐mediated NFATc1 and mitogen‐activated protein kinase deactivation underlies the inhibitory effects of kavain on osteoclastogenesis and bone resorption.
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- Journal of Cellular Physiology, 2019, v. 234, n. 1, p. 789, doi. 10.1002/jcp.26893
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MiR‐214 is an important regulator of the musculoskeletal metabolism and disease.
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- Journal of Cellular Physiology, 2019, v. 234, n. 1, p. 231, doi. 10.1002/jcp.26856
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Cistanche deserticola polysaccharide attenuates osteoclastogenesis and bone resorption via inhibiting RANKL signaling and reactive oxygen species production.
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- Journal of Cellular Physiology, 2018, v. 233, n. 12, p. 9674, doi. 10.1002/jcp.26882
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Lumichrome inhibits osteoclastogenesis and bone resorption through suppressing RANKL‐induced NFAT activation and calcium signaling.
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- Journal of Cellular Physiology, 2018, v. 233, n. 11, p. 8971, doi. 10.1002/jcp.26841
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- Article
EGFL7: Master regulator of cancer pathogenesis, angiogenesis and an emerging mediator of bone homeostasis.
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- Journal of Cellular Physiology, 2018, v. 233, n. 11, p. 8526, doi. 10.1002/jcp.26792
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- Article
The emerging role of NPNT in tissue injury repair and bone homeostasis.
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- Journal of Cellular Physiology, 2018, v. 233, n. 3, p. 1887, doi. 10.1002/jcp.26013
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- Article
Luteoloside prevents lipopolysaccharide-induced osteolysis and suppresses RANKL-induced osteoclastogenesis through attenuating RANKL signaling cascades.
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- Journal of Cellular Physiology, 2018, v. 233, n. 2, p. 1723, doi. 10.1002/jcp.26084
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- Article
Cyanidin Chloride inhibits ovariectomy-induced osteoporosis by suppressing RANKL-mediated osteoclastogenesis and associated signaling pathways.
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- Journal of Cellular Physiology, 2018, v. 233, n. 3, p. 2502, doi. 10.1002/jcp.26126
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- Article
Mechanical Stress Regulates Bone Metabolism Through MicroRNAs.
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- Journal of Cellular Physiology, 2017, v. 232, n. 6, p. 1239, doi. 10.1002/jcp.25688
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The Emerging Role of MORC Family Proteins in Cancer Development and Bone Homeostasis.
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- Journal of Cellular Physiology, 2017, v. 232, n. 5, p. 928, doi. 10.1002/jcp.25665
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- Article
Eriodictyol Inhibits RANKL-Induced Osteoclast Formation and Function Via Inhibition of NFATc1 Activity.
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- Journal of Cellular Physiology, 2016, v. 231, n. 9, p. 1983, doi. 10.1002/jcp.25304
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- Article
Protein Kinase C Inhibitor, GF109203X Attenuates Osteoclastogenesis, Bone Resorption and RANKL-Induced NF-κB and NFAT Activity.
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- Journal of Cellular Physiology, 2015, v. 230, n. 6, p. 1235, doi. 10.1002/jcp.24858
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EGFL7 Is Expressed in Bone Microenvironment and Promotes Angiogenesis via ERK, STAT3, and Integrin Signaling Cascades.
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- Journal of Cellular Physiology, 2015, v. 230, n. 1, p. 82, doi. 10.1002/jcp.24684
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- Article
Astilbin prevents bone loss in ovariectomized mice through the inhibition of RANKL‐induced osteoclastogenesis.
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- Journal of Cellular & Molecular Medicine, 2019, v. 23, n. 12, p. 8355, doi. 10.1111/jcmm.14713
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- Article
Asperpyrone A attenuates RANKL‐induced osteoclast formation through inhibiting NFATc1, Ca<sup>2+</sup> signalling and oxidative stress.
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- Journal of Cellular & Molecular Medicine, 2019, v. 23, n. 12, p. 8269, doi. 10.1111/jcmm.14700
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Evodiamine inhibits RANKL‐induced osteoclastogenesis and prevents ovariectomy‐induced bone loss in mice.
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- Journal of Cellular & Molecular Medicine, 2019, v. 23, n. 1, p. 522, doi. 10.1111/jcmm.13955
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- Article
Madecassoside inhibits estrogen deficiency‐induced osteoporosis by suppressing RANKL‐induced osteoclastogenesis.
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- Journal of Cellular & Molecular Medicine, 2019, v. 23, n. 1, p. 380, doi. 10.1111/jcmm.13942
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- Article