Found: 17
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Transformation of renal tubule epithelial cells by simian virus-40 is associated with emergence of Ca<sup>2+</sup>-insensitive K<sup>+</sup> channels and altered mitogenic sensitivity to K<sup>+</sup> channel blockers.
- Published in:
- Journal of Cellular Physiology, 1992, v. 151, n. 1, p. 113, doi. 10.1002/jcp.1041510116
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- Publication type:
- Article
Vasopressin-stimulated CFTR Cl<sup>−</sup> currents are increased in the renal collecting duct cells of a mouse model of Liddle's syndrome.
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- Journal of Physiology, 2005, v. 562, n. 1, p. 271, doi. 10.1113/jphysiol.2004.077933
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- Publication type:
- Article
An inward rectifier K<sup>+</sup> channel at the basolateral membrane of the mouse distal convoluted tubule: similarities with Kir4-Kir5.1 heteromeric channels.
- Published in:
- Journal of Physiology, 2002, v. 538, n. 2, p. 391, doi. 10.1113/jphysiol.2001.012961
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- Publication type:
- Article
A new regulator of the vacuolar H(+)-ATPase in the kidney.
- Published in:
- 2011
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- Publication type:
- journal article
A new regulator of the vacuolar H<sup>+</sup>-ATPase in the kidney.
- Published in:
- 2011
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- Publication type:
- Opinion
Novel CLCN5 mutations in patients with Dent’s disease result in altered ion currents or impaired exchanger processing.
- Published in:
- Kidney International, 2009, v. 76, n. 9, p. 999, doi. 10.1038/ki.2009.305
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- Publication type:
- Article
Phosphatidylinositol (4,5)-bisphosphate dynamically regulates the K<sub>2P</sub> background K<sup>+</sup> channel TASK-2.
- Published in:
- Scientific Reports, 2017, p. 45407, doi. 10.1038/srep45407
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- Publication type:
- Article
Piezo1-dependent stretch-activated channels are inhibited by Polycystin-2 in renal tubular epithelial cells.
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- EMBO Reports, 2013, v. 14, n. 12, p. 1143, doi. 10.1038/embor.2013.170
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- Publication type:
- Article
CLCNKB mutations causing mild Bartter syndrome profoundly alter the pH and Ca dependence of ClC-Kb channels.
- Published in:
- Pflügers Archiv: European Journal of Physiology, 2014, v. 466, n. 9, p. 1713, doi. 10.1007/s00424-013-1401-2
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- Publication type:
- Article
ClC-5 mutations associated with Dent's disease: a major role of the dimer interface.
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- Pflügers Archiv: European Journal of Physiology, 2012, v. 463, n. 2, p. 247, doi. 10.1007/s00424-011-1052-0
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- Publication type:
- Article
Identification and Functional Expression of a Glutamate- and Avermectin-Gated Chloride Channel from Caligus rogercresseyi, a Southern Hemisphere Sea Louse Affecting Farmed Fish.
- Published in:
- PLoS Pathogens, 2014, v. 10, n. 9, p. 1, doi. 10.1371/journal.ppat.1004402
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- Publication type:
- Article
Vasopressin‐stimulated chloride transport in transimmortalized mouse cell lines derived from the distal convoluted tubule and cortical and inner medullary collecting ducts.
- Published in:
- Nephrology Dialysis Transplantation, 2001, v. 16, n. 2, p. 238, doi. 10.1093/ndt/16.2.238
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- Publication type:
- Article
Analysis of CLCNKB mutations at dimer‐interface, calcium‐binding site, and pore reveals a variety of functional alterations in ClC‐Kb channel leading to Bartter syndrome.
- Published in:
- Human Mutation, 2020, v. 41, n. 4, p. 774, doi. 10.1002/humu.23962
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- Publication type:
- Article
Novel CLCNKB Mutations Causing Bartter Syndrome Affect Channel Surface Expression.
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- Human Mutation, 2013, v. 34, n. 9, p. 1269, doi. 10.1002/humu.22361
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- Publication type:
- Article
Heterogeneity in the processing of CLCN5 mutants related to Dent disease.
- Published in:
- Human Mutation, 2011, v. 32, n. 4, p. 476, doi. 10.1002/humu.21467
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- Publication type:
- Article
Misprocessing of the CFTR protein leads to mild cystic fibrosis phenotype.
- Published in:
- Human Mutation, 2005, v. 25, n. 4, p. 360, doi. 10.1002/humu.20156
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- Publication type:
- Article
Acute genetic ablation of pendrin lowers blood pressure in mice.
- Published in:
- Nephrology Dialysis Transplantation, 2017, v. 32, n. 7, p. 1137, doi. 10.1093/ndt/gfw393
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- Publication type:
- Article